Polyunsaturated fatty acid status at birth, childhood growth, and cardiometabolic risk: a pooled analysis of the MEFAB and RHEA cohorts.


Journal

European journal of clinical nutrition
ISSN: 1476-5640
Titre abrégé: Eur J Clin Nutr
Pays: England
ID NLM: 8804070

Informations de publication

Date de publication:
04 2019
Historique:
received: 26 06 2017
accepted: 09 04 2018
revised: 28 03 2018
pubmed: 17 5 2018
medline: 14 7 2020
entrez: 17 5 2018
Statut: ppublish

Résumé

Polyunsaturated fatty acid (PUFA) status during pregnancy has been suggested to influence offspring obesity and cardiometabolic health. We assessed whether prenatal PUFA exposure is associated with rapid infant growth, childhood BMI, and cardiometabolic profile. In the Dutch MEFAB (n = 266) and Greek RHEA (n = 263) cohorts, we measured n-3 and n-6 PUFA concentrations in cord blood phospholipids, which reflect fetal exposure in late pregnancy. We defined rapid infant growth from birth to 6 months of age as an increase in weight z-score >0.67. We analyzed body mass index (BMI) as continuous and in categories of overweight/obesity at 4 and 6 years. We computed a cardiometabolic risk score at 6-7 years as the sum of waist circumference, non-high-density lipoprotein cholesterol and blood pressure z-scores. Associations of PUFAs with child health outcomes were assessed using generalized linear models for binary outcomes and linear regression models for continuous ones after adjusting for important covariates, and for the pooled estimates, a cohort indicator. In pooled analyses, we found no association of PUFA levels with rapid infant growth, childhood BMI (β per SD increase in the total n-3:n-6 PUFA ratio = -0.04 SD; 99% CI: -0.15, 0.06; P = 0.65 at 4 years, and -0.05 SD; 99% CI: -0.18, 0.08; P = 0.78 at 6 years), and overweight/obesity. We also found no associations for clustered cardiometabolic risk and its individual components. The results were similar across cohorts. Our findings suggest that PUFA concentrations at birth are not associated with later obesity development and cardiometabolic risk in childhood.

Sections du résumé

BACKGROUND/OBJECTIVES
Polyunsaturated fatty acid (PUFA) status during pregnancy has been suggested to influence offspring obesity and cardiometabolic health. We assessed whether prenatal PUFA exposure is associated with rapid infant growth, childhood BMI, and cardiometabolic profile.
SUBJECTS/METHODS
In the Dutch MEFAB (n = 266) and Greek RHEA (n = 263) cohorts, we measured n-3 and n-6 PUFA concentrations in cord blood phospholipids, which reflect fetal exposure in late pregnancy. We defined rapid infant growth from birth to 6 months of age as an increase in weight z-score >0.67. We analyzed body mass index (BMI) as continuous and in categories of overweight/obesity at 4 and 6 years. We computed a cardiometabolic risk score at 6-7 years as the sum of waist circumference, non-high-density lipoprotein cholesterol and blood pressure z-scores. Associations of PUFAs with child health outcomes were assessed using generalized linear models for binary outcomes and linear regression models for continuous ones after adjusting for important covariates, and for the pooled estimates, a cohort indicator.
RESULTS
In pooled analyses, we found no association of PUFA levels with rapid infant growth, childhood BMI (β per SD increase in the total n-3:n-6 PUFA ratio = -0.04 SD; 99% CI: -0.15, 0.06; P = 0.65 at 4 years, and -0.05 SD; 99% CI: -0.18, 0.08; P = 0.78 at 6 years), and overweight/obesity. We also found no associations for clustered cardiometabolic risk and its individual components. The results were similar across cohorts.
CONCLUSIONS
Our findings suggest that PUFA concentrations at birth are not associated with later obesity development and cardiometabolic risk in childhood.

Identifiants

pubmed: 29765163
doi: 10.1038/s41430-018-0175-1
pii: 10.1038/s41430-018-0175-1
doi:

Substances chimiques

Fatty Acids, Unsaturated 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

566-576

Auteurs

Nikos Stratakis (N)

Section of Complex Genetics and Epidemiology, Departments of Genetics and Cell Biology, School for Nutrition and Translational Research in Metabolism (NUTRIM), Faculty of Health, Medicine and Life Sciences, Maastricht University, Maastricht, The Netherlands. n.stratakis@maastrichtuniversity.nl.
Department of Social Medicine, Faculty of Medicine, University of Crete, Heraklion, Greece. n.stratakis@maastrichtuniversity.nl.

Marij Gielen (M)

Section of Complex Genetics and Epidemiology, Departments of Genetics and Cell Biology, School for Nutrition and Translational Research in Metabolism (NUTRIM), Faculty of Health, Medicine and Life Sciences, Maastricht University, Maastricht, The Netherlands.

Katerina Margetaki (K)

Department of Social Medicine, Faculty of Medicine, University of Crete, Heraklion, Greece.

Renate H M de Groot (RHM)

Section of Complex Genetics and Epidemiology, Departments of Genetics and Cell Biology, School for Nutrition and Translational Research in Metabolism (NUTRIM), Faculty of Health, Medicine and Life Sciences, Maastricht University, Maastricht, The Netherlands.
Welten Institute-Research Centre for Learning, Teaching and Technology, Open University of the Netherlands, Heerlen, The Netherlands.

Maria Apostolaki (M)

Environmental Chemical Processes Laboratory (ECPL), Department of Chemistry, University of Crete, Heraklion, Greece.

Georgia Chalkiadaki (G)

Department of Social Medicine, Faculty of Medicine, University of Crete, Heraklion, Greece.

Marina Vafeiadi (M)

Department of Social Medicine, Faculty of Medicine, University of Crete, Heraklion, Greece.

Vasiliki Leventakou (V)

Department of Social Medicine, Faculty of Medicine, University of Crete, Heraklion, Greece.

Marianna Karachaliou (M)

Department of Social Medicine, Faculty of Medicine, University of Crete, Heraklion, Greece.

Roger W Godschalk (RW)

Department of Pharmacology and Toxicology, School for Nutrition and Translational Research in Metabolism (NUTRIM), Faculty of Health, Medicine and Life Sciences, Maastricht University, Maastricht, The Netherlands.

Manolis Kogevinas (M)

ISGlobal, Centre for Research in Environmental Epidemiology (CREAL), Barcelona, Spain.
IMIM Hospital del Mar Medicine Research Institute, Barcelona, Spain.
Spanish Consortium for Research in epidemiology and Public Health (CIBERESP), Madrid, Spain.

Euripides G Stephanou (EG)

Environmental Chemical Processes Laboratory (ECPL), Department of Chemistry, University of Crete, Heraklion, Greece.
The Cyprus Institute, Nicosia, Cyprus.

Maurice P Zeegers (MP)

Section of Complex Genetics and Epidemiology, Departments of Genetics and Cell Biology, School for Nutrition and Translational Research in Metabolism (NUTRIM), Faculty of Health, Medicine and Life Sciences, Maastricht University, Maastricht, The Netherlands.
School CAPHRI: Care and Public Health Research Institute, Maastricht University, Maastricht, The Netherlands.

Leda Chatzi (L)

Section of Complex Genetics and Epidemiology, Departments of Genetics and Cell Biology, School for Nutrition and Translational Research in Metabolism (NUTRIM), Faculty of Health, Medicine and Life Sciences, Maastricht University, Maastricht, The Netherlands.
Department of Social Medicine, Faculty of Medicine, University of Crete, Heraklion, Greece.
Department of Preventive Medicine, Keck School of Medicine, University of Southern California, Los Angeles, USA.

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