Effects of fenoldopam on renal blood flow in hypertensive chronic kidney disease.


Journal

Journal of nephrology
ISSN: 1724-6059
Titre abrégé: J Nephrol
Pays: Italy
ID NLM: 9012268

Informations de publication

Date de publication:
Feb 2019
Historique:
received: 30 01 2018
accepted: 06 04 2018
pubmed: 17 5 2018
medline: 6 5 2020
entrez: 17 5 2018
Statut: ppublish

Résumé

The synthetic drug fenoldopam mesylate (FM) may have a renoprotective role, and a "renal dose" of 0.1 µg/kg/min intravenous (IV) infusion of FM has been reported as able to increase renal blood flow without affecting systemic blood pressure. But conclusive data are still lacking. We aimed to investigate by color-Doppler ultrasonography the effects of IV administration of FM at this dosage in hypertensive chronic kidney disease (CKD) patients, and verify whether it may induce any systemic hemodynamic alteration. In 60 hypertensive CKD patients, we measured by duplex Doppler ultrasonography, at baseline and during infusion of 0.1 µg/kg/min of FM, the systolic and diastolic flow velocity (sampled at the renal hilum, intermediate section and origin of both renal arteries) and the intra-parenchymal renal resistive index (RRI) sampled on interlobular arteries of both kidneys. Patients were divided into four subgroups (I-IV) according to classification of National Kidney Foundation Kidney Disease Outcomes Quality Initiative (NKF-DOQI). Infusion of 0.1 µg/kg/min FM significantly decreased the RRI (0.73 ± 0.05 vs. 0.65 ± 0.06; p < 0.0001) and increased the systolic and diastolic flow velocities in all renal artery tracts examined. No single episode of systemic hypotension was observed. Very low-dose FM may significantly increase renal blood flow and exert a renal protective effect in hypertensive CKD patients. Infusion of FM at such low dosage appears also to be quite safe, even in CKD and hypertensive patients.

Sections du résumé

BACKGROUND AND AIM OBJECTIVE
The synthetic drug fenoldopam mesylate (FM) may have a renoprotective role, and a "renal dose" of 0.1 µg/kg/min intravenous (IV) infusion of FM has been reported as able to increase renal blood flow without affecting systemic blood pressure. But conclusive data are still lacking. We aimed to investigate by color-Doppler ultrasonography the effects of IV administration of FM at this dosage in hypertensive chronic kidney disease (CKD) patients, and verify whether it may induce any systemic hemodynamic alteration.
METHODS METHODS
In 60 hypertensive CKD patients, we measured by duplex Doppler ultrasonography, at baseline and during infusion of 0.1 µg/kg/min of FM, the systolic and diastolic flow velocity (sampled at the renal hilum, intermediate section and origin of both renal arteries) and the intra-parenchymal renal resistive index (RRI) sampled on interlobular arteries of both kidneys. Patients were divided into four subgroups (I-IV) according to classification of National Kidney Foundation Kidney Disease Outcomes Quality Initiative (NKF-DOQI).
RESULTS RESULTS
Infusion of 0.1 µg/kg/min FM significantly decreased the RRI (0.73 ± 0.05 vs. 0.65 ± 0.06; p < 0.0001) and increased the systolic and diastolic flow velocities in all renal artery tracts examined. No single episode of systemic hypotension was observed.
CONCLUSIONS CONCLUSIONS
Very low-dose FM may significantly increase renal blood flow and exert a renal protective effect in hypertensive CKD patients. Infusion of FM at such low dosage appears also to be quite safe, even in CKD and hypertensive patients.

Identifiants

pubmed: 29766465
doi: 10.1007/s40620-018-0496-0
pii: 10.1007/s40620-018-0496-0
doi:

Substances chimiques

Antihypertensive Agents 0
Vasodilator Agents 0
Fenoldopam INU8H2KAWG

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

75-81

Références

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Auteurs

Valentina Rovella (V)

Division of Hypertension and Nephrology, Department of Systems Medicine, University of Rome "Tor Vergata", Viale Oxford 81, 00133, Rome, Italy.

Michele Ferrannini (M)

Division of Hypertension and Nephrology, Department of Systems Medicine, University of Rome "Tor Vergata", Viale Oxford 81, 00133, Rome, Italy.

Manfredi Tesauro (M)

Division of Hypertension and Nephrology, Department of Systems Medicine, University of Rome "Tor Vergata", Viale Oxford 81, 00133, Rome, Italy.

Giulia Marrone (G)

Division of Hypertension and Nephrology, Department of Systems Medicine, University of Rome "Tor Vergata", Viale Oxford 81, 00133, Rome, Italy.
PhD School of Applied Medical-Surgical Sciences, University of Rome "Tor Vergata", Via Montpellier 1, 00133, Rome, Italy.

Andrea Busca (A)

Division of Hypertension and Nephrology, Department of Systems Medicine, University of Rome "Tor Vergata", Viale Oxford 81, 00133, Rome, Italy.

Roberto Sorge (R)

Laboratory of Biometry, Department of Systems Medicine, University of Rome "Tor Vergata", Via Montpellier 1, 00133, Rome, Italy.

Simone Manca di Villahermosa (S)

Division of Hypertension and Nephrology, Department of Systems Medicine, University of Rome "Tor Vergata", Viale Oxford 81, 00133, Rome, Italy.

Maurizio Casasco (M)

Federazione Medico Sportiva Italiana, 00196, Rome, Italy.

Nicola Di Daniele (N)

Division of Hypertension and Nephrology, Department of Systems Medicine, University of Rome "Tor Vergata", Viale Oxford 81, 00133, Rome, Italy. didaniele@med.uniroma2.it.

Annalisa Noce (A)

Division of Hypertension and Nephrology, Department of Systems Medicine, University of Rome "Tor Vergata", Viale Oxford 81, 00133, Rome, Italy. annalisa.noce@uniroma2.it.

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