The Effects of Different mTOR Inhibitors in EGFR Inhibitor Resistant Colon Carcinoma Cells.
Apoptosis
/ drug effects
Cell Proliferation
/ drug effects
Colonic Neoplasms
/ drug therapy
Drug Resistance, Neoplasm
/ drug effects
ErbB Receptors
/ antagonists & inhibitors
Humans
Protein Kinase Inhibitors
/ pharmacology
TOR Serine-Threonine Kinases
/ antagonists & inhibitors
Tumor Cells, Cultured
Colon carcinoma
EGFR inhibitor
Resistance
mTOR inhibitor
Journal
Pathology oncology research : POR
ISSN: 1532-2807
Titre abrégé: Pathol Oncol Res
Pays: Switzerland
ID NLM: 9706087
Informations de publication
Date de publication:
Oct 2019
Oct 2019
Historique:
received:
07
02
2018
accepted:
29
05
2018
pubmed:
9
6
2018
medline:
31
3
2020
entrez:
9
6
2018
Statut:
ppublish
Résumé
Several monoclonal antibodies and inhibitors targeting signalling pathways are being used in personalised medicine. Anti-EGFR antibodies seem to be effective, however, therapy resistance often occurs in colon carcinoma cases. mTOR inhibitors (mTORIs) could have a potential role in the breakthrough of therapy resistance. The mTOR activity related protein expression patterns and the in vitro effects of EGFR inhibitors (EGFRIs), mTORIs and their combinations were studied in different colon carcinoma cell lines (with different genetic backgrounds). Alamar Blue test and flow cytometry were used to analyse the in vitro proliferation and apoptotic effects of cetuximab, gefitinib, cisplatin, rapamycin, PP242 and NVP-BEZ235. The expressions of mTOR activity related proteins (p-70S6K, p-S6, Rictor, p-mTOR, Raptor) were studied by Western blot, immunocytochemistry and Duolink staining. The EGFRI resistance of the studied colon carcinoma cell lines related to their known mutations were confirmed, neither gefitinib nor cetuximab inhibited the proliferation or induced apoptosis in vitro. Individual differences in Rictor and Raptor expressions were detected by Western blot and immunocytochemistry beside elevated mTOR activity of these different colon carcinoma cell lines. These expression patterns correlated to the mTORIs sensitivity differences, moreover, mTORIs could enhance the effects of EGFRIs and other in vitro treatments. Our results suggest that mTORI combinations could be helpful in both EGFRI and platinum-based therapy of colon carcinomas. Moreover, we suggest determining both mTOR complex activity and mutations in Akt/mTOR signalling pathways for selecting the appropriate mTORIs and patients in potential future combination treatments.
Identifiants
pubmed: 29882195
doi: 10.1007/s12253-018-0434-4
pii: 10.1007/s12253-018-0434-4
doi:
Substances chimiques
Protein Kinase Inhibitors
0
MTOR protein, human
EC 2.7.1.1
EGFR protein, human
EC 2.7.10.1
ErbB Receptors
EC 2.7.10.1
TOR Serine-Threonine Kinases
EC 2.7.11.1
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Pagination
1379-1386Subventions
Organisme : OTKA
ID : K84262
Organisme : Semmelweis University Scientific and Innovation Founds
ID : STIA-KF-17
Organisme : Bolyai fellowship
ID : 590/2015
Organisme : New National Excellence Programs
ID : ÚNKP-17-3
Organisme : New National Excellence Programs
ID : ÚNKP-17-2
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