Linking migraine frequency with family history of migraine.


Journal

Cephalalgia : an international journal of headache
ISSN: 1468-2982
Titre abrégé: Cephalalgia
Pays: England
ID NLM: 8200710

Informations de publication

Date de publication:
02 2019
Historique:
pubmed: 19 6 2018
medline: 14 4 2020
entrez: 19 6 2018
Statut: ppublish

Résumé

Migraine is a complex genetic disorder that is brought about by multiple genetic and environmental factors. We aimed to assess whether migraine frequency is associated with genetic susceptibility. We investigated in 2829 migraine patients (14% males) whether 'migraine frequency' (measured as the number of migraine days per month) was related to 'genetic load' (measured as the number of parents affected with migraine) using a validated web-based questionnaire. In addition, we investigated associations with age-at-onset, migraine subtype, use of acute headache medication, and comorbid depression. We found an association between the number of migraine days per month and family history of migraine for males ( p = 0.03), but not for females ( p = 0.97). This association was confirmed in a linear regression analysis. Also, a lower age-at-onset ( p < 0.001), having migraine with aura ( p = 0.03), and a high number of medication days ( p = 0.006) were associated with a stronger family history of migraine, whereas lifetime depression ( p = 0.13) was not. Migraine frequency, as measured by the number of migraine days per month, seems associated with a genetic predisposition only in males. A stronger family history of migraine was also associated with a lower age-at-onset, a higher number of medication days, and migraine with aura. Our findings suggest that specific clinical features of migraine seem more determined by genetic factors.

Sections du résumé

BACKGROUND
Migraine is a complex genetic disorder that is brought about by multiple genetic and environmental factors. We aimed to assess whether migraine frequency is associated with genetic susceptibility.
METHODS
We investigated in 2829 migraine patients (14% males) whether 'migraine frequency' (measured as the number of migraine days per month) was related to 'genetic load' (measured as the number of parents affected with migraine) using a validated web-based questionnaire. In addition, we investigated associations with age-at-onset, migraine subtype, use of acute headache medication, and comorbid depression.
RESULTS
We found an association between the number of migraine days per month and family history of migraine for males ( p = 0.03), but not for females ( p = 0.97). This association was confirmed in a linear regression analysis. Also, a lower age-at-onset ( p < 0.001), having migraine with aura ( p = 0.03), and a high number of medication days ( p = 0.006) were associated with a stronger family history of migraine, whereas lifetime depression ( p = 0.13) was not.
DISCUSSION
Migraine frequency, as measured by the number of migraine days per month, seems associated with a genetic predisposition only in males. A stronger family history of migraine was also associated with a lower age-at-onset, a higher number of medication days, and migraine with aura. Our findings suggest that specific clinical features of migraine seem more determined by genetic factors.

Identifiants

pubmed: 29911421
doi: 10.1177/0333102418783295
pmc: PMC6376592
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

229-236

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Auteurs

Nadine Pelzer (N)

1 Department of Neurology, Leiden University Medical Centre, Leiden, the Netherlands.

Mark A Louter (MA)

1 Department of Neurology, Leiden University Medical Centre, Leiden, the Netherlands.
2 Department of Psychiatry, Leiden University Medical Centre, Leiden, the Netherlands.
3 Viersprong Institute for Studies on Personality Disorders, De Viersprong, Halsteren, the Netherlands.

Erik W van Zwet (EW)

4 Department of Biostatistics, Leiden University Medical Centre, Leiden, the Netherlands.

Dale R Nyholt (DR)

5 Institute of Health and Biomedical Innovation and School of Biomedical Science, Queensland University of Technology, Brisbane, Queensland, Australia.

Michel D Ferrari (MD)

1 Department of Neurology, Leiden University Medical Centre, Leiden, the Netherlands.

Arn Mjm van den Maagdenberg (AM)

1 Department of Neurology, Leiden University Medical Centre, Leiden, the Netherlands.
6 Department of Human Genetics, Leiden University Medical Centre, Leiden, the Netherlands.

Joost Haan (J)

1 Department of Neurology, Leiden University Medical Centre, Leiden, the Netherlands.
7 Department of Neurology, Alrijne Hospital, Leiderdorp, the Netherlands.

Gisela M Terwindt (GM)

1 Department of Neurology, Leiden University Medical Centre, Leiden, the Netherlands.

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