Predictors of angiographically visible distal embolization in STEMI.

Prädiktoren der angiographisch sichtbaren distalen Embolie bei STEMI.

Journal

Herz
ISSN: 1615-6692
Titre abrégé: Herz
Pays: Germany
ID NLM: 7801231

Informations de publication

Date de publication:
May 2020
Historique:
received: 30 03 2018
accepted: 03 06 2018
revised: 06 05 2018
pubmed: 22 6 2018
medline: 1 7 2020
entrez: 22 6 2018
Statut: ppublish

Résumé

Distal embolization during primary percutaneous coronary intervention (p-PCI) in the treatment of ST-segment elevation myocardial infarction (STEMI) is associated with a poor prognosis. In this situation, thrombectomy is performed to prevent distal embolization and to restore myocardial reperfusion. The aim of our study was to determine angiographic predictors of angiographically visible distal embolization (AVDE) in patients with STEMI treated by p‑PCI with thrombectomy. This prospective study included all consecutive patients who underwent p‑PCI with thrombectomy for STEMI at our institution between October 2011 and December 2014 AVDE was defined as a distal filling defect with an abrupt cut-off in one of the peripheral coronary branches of the infarct-related artery, distal to the angioplasty site. Thrombectomy was considered positive when it removed thrombi, and successful when it improved coronary flow. Among the 346 patients included, 59 (17%) developed AVDE during p‑PCI. In multivariate analysis, the infarct-related right coronary artery (OR: 2.48, 95% CI: 1.36-4.52; p = 0.003) and a culprit lesion diameter of >3 mm (OR : 1.90, 95% CI: 1.01-3.56; p = 0.048) were identified as independent factors associated with AVDE during p‑PCI with thrombectomy for STEMI. The success of thrombectomy and the Syntax score were not associated with AVDE. AVDE complicating p‑PCI with thrombectomy in STEMI is frequent (17%) and a successful thrombectomy does not rule out AVDE.

Sections du résumé

BACKGROUND BACKGROUND
Distal embolization during primary percutaneous coronary intervention (p-PCI) in the treatment of ST-segment elevation myocardial infarction (STEMI) is associated with a poor prognosis. In this situation, thrombectomy is performed to prevent distal embolization and to restore myocardial reperfusion. The aim of our study was to determine angiographic predictors of angiographically visible distal embolization (AVDE) in patients with STEMI treated by p‑PCI with thrombectomy.
PATIENTS AND METHODS METHODS
This prospective study included all consecutive patients who underwent p‑PCI with thrombectomy for STEMI at our institution between October 2011 and December 2014 AVDE was defined as a distal filling defect with an abrupt cut-off in one of the peripheral coronary branches of the infarct-related artery, distal to the angioplasty site. Thrombectomy was considered positive when it removed thrombi, and successful when it improved coronary flow.
RESULTS RESULTS
Among the 346 patients included, 59 (17%) developed AVDE during p‑PCI. In multivariate analysis, the infarct-related right coronary artery (OR: 2.48, 95% CI: 1.36-4.52; p = 0.003) and a culprit lesion diameter of >3 mm (OR : 1.90, 95% CI: 1.01-3.56; p = 0.048) were identified as independent factors associated with AVDE during p‑PCI with thrombectomy for STEMI. The success of thrombectomy and the Syntax score were not associated with AVDE.
CONCLUSION CONCLUSIONS
AVDE complicating p‑PCI with thrombectomy in STEMI is frequent (17%) and a successful thrombectomy does not rule out AVDE.

Identifiants

pubmed: 29926119
doi: 10.1007/s00059-018-4723-1
pii: 10.1007/s00059-018-4723-1
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

288-292

Auteurs

N V Yaméogo (NV)

Cardiology Department, University Hospital, 14 rue Paul Gaffarel, 21079, Dijon CEDEX, France.

C Guenancia (C)

Cardiology Department, University Hospital, 14 rue Paul Gaffarel, 21079, Dijon CEDEX, France. charles.guenancia@gmail.com.
PEC2, UFR Sciences de Santé, Univ. Bourgogne Franche-Comté, Dijon, France. charles.guenancia@gmail.com.

G Porot (G)

Cardiology Department, University Hospital, 14 rue Paul Gaffarel, 21079, Dijon CEDEX, France.

K Stamboul (K)

Cardiology Department, University Hospital, 14 rue Paul Gaffarel, 21079, Dijon CEDEX, France.
PEC2, UFR Sciences de Santé, Univ. Bourgogne Franche-Comté, Dijon, France.

C Richard (C)

Cardiology Department, University Hospital, 14 rue Paul Gaffarel, 21079, Dijon CEDEX, France.
PEC2, UFR Sciences de Santé, Univ. Bourgogne Franche-Comté, Dijon, France.

A Gudjoncik (A)

Cardiology Department, University Hospital, 14 rue Paul Gaffarel, 21079, Dijon CEDEX, France.
PEC2, UFR Sciences de Santé, Univ. Bourgogne Franche-Comté, Dijon, France.

J Hamblin (J)

Cardiology Department, University Hospital, 14 rue Paul Gaffarel, 21079, Dijon CEDEX, France.

P Buffet (P)

Cardiology Department, University Hospital, 14 rue Paul Gaffarel, 21079, Dijon CEDEX, France.

L Lorgis (L)

Cardiology Department, University Hospital, 14 rue Paul Gaffarel, 21079, Dijon CEDEX, France.
PEC2, UFR Sciences de Santé, Univ. Bourgogne Franche-Comté, Dijon, France.

Y Cottin (Y)

Cardiology Department, University Hospital, 14 rue Paul Gaffarel, 21079, Dijon CEDEX, France.
PEC2, UFR Sciences de Santé, Univ. Bourgogne Franche-Comté, Dijon, France.

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