Association between dopamine receptor gene polymorphisms and effects of risperidone treatment: A systematic review and meta-analysis.


Journal

Basic & clinical pharmacology & toxicology
ISSN: 1742-7843
Titre abrégé: Basic Clin Pharmacol Toxicol
Pays: England
ID NLM: 101208422

Informations de publication

Date de publication:
Jan 2019
Historique:
received: 19 03 2018
accepted: 17 07 2018
pubmed: 14 8 2018
medline: 17 4 2019
entrez: 14 8 2018
Statut: ppublish

Résumé

The effect of risperidone treatment in patients with schizophrenia varies according to the dopamine receptor genes. This study aimed to evaluate the relationship between genes of the dopamine receptors (D1, D2, and D3) and the effect of risperidone treatment. Three electronic databases (PubMed, Embase, and Cochrane Library) were searched for relevant cohort or case-control studies published before 9 May 2018. A systematic review and meta-analysis was performed for qualitative and quantitative assessment of the relationship between the dopamine receptors D1, D2, and D3 (DRD1, 2, and 3) and the effect of risperidone treatment. The summary odds ratio (OR) and weighted mean difference (WMD) in a random-effects model were used to measure these relationships. Twelve studies involving 24 SNPs were included. DRD2 (Ser311Cys, rs1801028 Ser/Ser) significantly lowered the improvement rate (determined by the PANSS score) unlike Ser/Cys (WMD: -11.58, 95% CI: -17.35 to -5.18). For Asian patients, A241G (rs1799978) AA carriers showed greater improvement after risperidone therapy (P < 0.05). The polymorphisms of 141C Ins/Del (rs1799732), T939C (rs6275), rs6277, and TaqID (rs1800498) may also influence the treatment effect. TaqIA (rs1800497) and TaqIB (rs17294542) were not associated with the rate of response to risperidone. DRD3 was not associated with an improvement in the PANSS total score; however, Ser9Gly might be related to a change in negative symptoms. No significant effect of DRD1 (rs5326, rs4867798, rs4532, and rs11749676) was found. Our result supported the hypothesis that DRD2 affected risperidone treatment. DRD1 had no significant effect on the response to risperidone, whereas DRD3 might be associated with an improvement in negative symptoms. Larger observational studies are warranted to verify these findings and identify other genetic factors involved.

Sections du résumé

BACKGROUND BACKGROUND
The effect of risperidone treatment in patients with schizophrenia varies according to the dopamine receptor genes. This study aimed to evaluate the relationship between genes of the dopamine receptors (D1, D2, and D3) and the effect of risperidone treatment.
METHODS METHODS
Three electronic databases (PubMed, Embase, and Cochrane Library) were searched for relevant cohort or case-control studies published before 9 May 2018. A systematic review and meta-analysis was performed for qualitative and quantitative assessment of the relationship between the dopamine receptors D1, D2, and D3 (DRD1, 2, and 3) and the effect of risperidone treatment. The summary odds ratio (OR) and weighted mean difference (WMD) in a random-effects model were used to measure these relationships.
RESULTS RESULTS
Twelve studies involving 24 SNPs were included. DRD2 (Ser311Cys, rs1801028 Ser/Ser) significantly lowered the improvement rate (determined by the PANSS score) unlike Ser/Cys (WMD: -11.58, 95% CI: -17.35 to -5.18). For Asian patients, A241G (rs1799978) AA carriers showed greater improvement after risperidone therapy (P < 0.05). The polymorphisms of 141C Ins/Del (rs1799732), T939C (rs6275), rs6277, and TaqID (rs1800498) may also influence the treatment effect. TaqIA (rs1800497) and TaqIB (rs17294542) were not associated with the rate of response to risperidone. DRD3 was not associated with an improvement in the PANSS total score; however, Ser9Gly might be related to a change in negative symptoms. No significant effect of DRD1 (rs5326, rs4867798, rs4532, and rs11749676) was found.
CONCLUSIONS CONCLUSIONS
Our result supported the hypothesis that DRD2 affected risperidone treatment. DRD1 had no significant effect on the response to risperidone, whereas DRD3 might be associated with an improvement in negative symptoms. Larger observational studies are warranted to verify these findings and identify other genetic factors involved.

Identifiants

pubmed: 30103286
doi: 10.1111/bcpt.13111
doi:

Substances chimiques

Antipsychotic Agents 0
DRD1 protein, human 0
DRD2 protein, human 0
DRD3 protein, human 0
Receptors, Dopamine D1 0
Receptors, Dopamine D2 0
Receptors, Dopamine D3 0
Risperidone L6UH7ZF8HC

Types de publication

Journal Article Meta-Analysis Systematic Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

94-104

Subventions

Organisme : National Key R&D Program of China
ID : 2016YFC0904900
Organisme : National Natural Science Foundation
ID : 81673509
Organisme : National Natural Science Foundation
ID : 81573504
Organisme : Beijing Municipal Natural Science Foundation
ID : 7171012
Organisme : National Science and Technology Major Projects for "Major New Drugs Innovation and Development" of China
ID : 2017ZX09304028
Organisme : National Science and Technology Major Projects for "Major New Drugs Innovation and Development" of China
ID : 2017ZX09101001

Informations de copyright

© 2018 Nordic Association for the Publication of BCPT (former Nordic Pharmacological Society).

Auteurs

Lingyue Ma (L)

Department of Pharmacy, Peking University First Hospital, Beijing, China.

Xiaodan Zhang (X)

Department of Pharmacy, Peking University First Hospital, Beijing, China.

Qian Xiang (Q)

Department of Pharmacy, Peking University First Hospital, Beijing, China.

Shuang Zhou (S)

Department of Pharmacy, Peking University First Hospital, Beijing, China.

Nan Zhao (N)

Department of Pharmacy, Peking University First Hospital, Beijing, China.

Qiufen Xie (Q)

Department of Pharmacy, Peking University First Hospital, Beijing, China.

Xia Zhao (X)

Department of Pharmacy, Peking University First Hospital, Beijing, China.

Ying Zhou (Y)

Department of Pharmacy, Peking University First Hospital, Beijing, China.

Yimin Cui (Y)

Department of Pharmacy, Peking University First Hospital, Beijing, China.

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Classifications MeSH