Biomarkers for Antidepressant Efficacy of Electroconvulsive Therapy: An Exploratory Cerebrospinal Fluid Study.


Journal

Neuropsychobiology
ISSN: 1423-0224
Titre abrégé: Neuropsychobiology
Pays: Switzerland
ID NLM: 7512895

Informations de publication

Date de publication:
2019
Historique:
received: 18 05 2018
accepted: 21 06 2018
pubmed: 20 8 2018
medline: 12 12 2018
entrez: 20 8 2018
Statut: ppublish

Résumé

No candidate biomarkers based on cerebrospinal fluid (CSF) have been identified as prognostic factors in patients with major depression treated with electroconvulsive therapy (ECT), yet. Following different underlying hypotheses, we analysed baseline CSF levels of markers of neurodegeneration (tau proteins, β-amyloids and neurogranin), elements of the innate immune system (interleukin [IL]-6, neopterin, soluble CD14, soluble CD163, migration inhibitory factor and monocyte chemotactic protein 1), endocannabinoids, sphingolipids and Klotho before ECT in patients with depression (n = 12) to identify possible correlations with the clinical antidepressant response to ECT. Correlation with the reduction of the depressive symptoms could be observed especially for markers of neurodegeneration and elements of the innate immune system. Differences for CSF levels of several markers were found between the groups of responders and non-responders at the trend level. The sample size is small and the -distribution of responders and non-responders is uneven. It is this first study on CSF biomarkers for antidepressant efficacy of ECT warrants further research regarding the mechanism of ECT and personalized antidepressant therapy.

Sections du résumé

BACKGROUND BACKGROUND
No candidate biomarkers based on cerebrospinal fluid (CSF) have been identified as prognostic factors in patients with major depression treated with electroconvulsive therapy (ECT), yet.
METHOD METHODS
Following different underlying hypotheses, we analysed baseline CSF levels of markers of neurodegeneration (tau proteins, β-amyloids and neurogranin), elements of the innate immune system (interleukin [IL]-6, neopterin, soluble CD14, soluble CD163, migration inhibitory factor and monocyte chemotactic protein 1), endocannabinoids, sphingolipids and Klotho before ECT in patients with depression (n = 12) to identify possible correlations with the clinical antidepressant response to ECT.
RESULTS RESULTS
Correlation with the reduction of the depressive symptoms could be observed especially for markers of neurodegeneration and elements of the innate immune system. Differences for CSF levels of several markers were found between the groups of responders and non-responders at the trend level.
LIMITATIONS CONCLUSIONS
The sample size is small and the -distribution of responders and non-responders is uneven.
CONCLUSIONS CONCLUSIONS
It is this first study on CSF biomarkers for antidepressant efficacy of ECT warrants further research regarding the mechanism of ECT and personalized antidepressant therapy.

Identifiants

pubmed: 30121652
pii: 000491401
doi: 10.1159/000491401
doi:

Substances chimiques

Biomarkers 0
Endocannabinoids 0
Sphingolipids 0
Glucuronidase EC 3.2.1.31
Klotho Proteins EC 3.2.1.31

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

13-22

Informations de copyright

© 2018 S. Karger AG, Basel.

Auteurs

Laura Kranaster (L)

Department of Psychiatry and Psychotherapy, Central Institute of Mental Health, Medical Faculty Mannheim/Heidelberg University, Mannheim, Germanylaura.kranaster@zi-mannheim.de.

Carolin Hoyer (C)

Department of Neurology, University Medical Centre Mannheim, Mannheim, Germany.

Suna S Aksay (SS)

Department of Psychiatry and Psychotherapy, Central Institute of Mental Health, Medical Faculty Mannheim/Heidelberg University, Mannheim, Germany.

J Malte Bumb (JM)

Department of Addictive Behavior and Addiction Medicine, Central Institute of Mental Health, Medical Faculty Mannheim/Heidelberg University, Mannheim, Germany.

Norbert Müller (N)

Department of Psychiatry and Psychotherapy, Ludwig Maximilian University Munich, Munich, Germany.
Marion von Tessin Memory-Zentrum, Munich, Germany.

Peter Zill (P)

Department of Psychiatry and Psychotherapy, Ludwig Maximilian University Munich, Munich, Germany.

Markus J Schwarz (MJ)

Institute of Laboratory Medicine, University Hospital LMU Munich, Munich, Germany.

Natalie Moll (N)

Institute of Laboratory Medicine, University Hospital LMU Munich, Munich, Germany.

Beat Lutz (B)

Institute for Physiological Chemistry, University Medical Center Mainz of the Johannes Gutenberg University Mainz, Mainz, Germany.

Laura Bindila (L)

Institute for Physiological Chemistry, University Medical Center Mainz of the Johannes Gutenberg University Mainz, Mainz, Germany.

Inga Zerr (I)

Department of Neurology, University Medical Center Göttingen and the German Center for Neurodegenerative Diseases (DZNE), Göttingen, Germany.

Matthias Schmitz (M)

Department of Neurology, University Medical Center Göttingen and the German Center for Neurodegenerative Diseases (DZNE), Göttingen, Germany.

Kaj Blennow (K)

Department of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, the Sahlgrenska Academy at the University of Gothenburg, Mölndal, Sweden.
Clinical Neurochemistry Laboratory, Sahlgrenska University Hospital, Mölndal, Sweden.

Henrik Zetterberg (H)

Department of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology, the Sahlgrenska Academy at the University of Gothenburg, Mölndal, Sweden.
Clinical Neurochemistry Laboratory, Sahlgrenska University Hospital, Mölndal, Sweden.
Department of Molecular Neuroscience, UCL Institute of Neurology, London, United Kingdom.
UK Dementia Research Institute at UCL, London, United Kingdom.

Dieter Haffner (D)

Department of Pediatric Kidney, Liver and Metabolic Diseases, Hannover Medical School, Hannover, Germany.

Maren Leifheit-Nestler (M)

Department of Pediatric Kidney, Liver and Metabolic Diseases, Hannover Medical School, Hannover, Germany.

Cagakan Ozbalci (C)

Randall Centre for Cell and Molecular Biophysics, King's College London, London, United Kingdom.
Heidelberg University Biochemistry Center, Heidelberg, Germany.

Christoph Janke (C)

Department of Anesthesiology and Critical Care Medicine, University Medical Centre Mannheim, Mannheim, Germany.

Manfred Thiel (M)

Department of Anesthesiology and Critical Care Medicine, University Medical Centre Mannheim, Mannheim, Germany.

Alexander Sartorius (A)

Department of Psychiatry and Psychotherapy, Central Institute of Mental Health, Medical Faculty Mannheim/Heidelberg University, Mannheim, Germany.

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Classifications MeSH