Variation at Interleukin-10 Locus Represents Susceptibility to Psoriasis in North Indian Population.


Journal

Endocrine, metabolic & immune disorders drug targets
ISSN: 2212-3873
Titre abrégé: Endocr Metab Immune Disord Drug Targets
Pays: United Arab Emirates
ID NLM: 101269157

Informations de publication

Date de publication:
2019
Historique:
received: 08 01 2018
revised: 31 05 2018
accepted: 05 07 2018
pubmed: 28 8 2018
medline: 18 5 2019
entrez: 28 8 2018
Statut: ppublish

Résumé

IL-10 is an important pleiotropic, immunoregulatory and anti-inflammatory cytokine which plays a significant role in the pathogenesis of psoriasis. The aim of the present study was to determine whether the three polymorphic sites of the IL-10 gene, haplotype and serum level confer susceptibility to psoriasis. 200 psoriatic patients and 200 controls were genotyped for three IL-10 polymorphic sites by ARMS polymerase chain reaction. Serum levels of IL -10 were measured by ELISA. Our results demonstrated that polymorphism of IL-10 -592 C/A (adjusted* OR = 9.25; 95% CI =3.16- 27.06) and IL-10 1082 A/G (adjusted* OR = 4.28; (95% CI =1.46- 12.56) was found to be in association with increased risk of psoriasis while as IL- 10 819 C/T (adjusted* OR= 1.60; (95% CI = 0.65-3.95) polymorphism does not show any significant association with the risk of psoriasis. HT7 GTC haplotype is associated with increased risk of psoriasis. Serum levels of IL-10 were found to be significantly low in patients, as compared to controls with a non-significant correlation between serum IL-10 level and psoriasis severity. IL-10 polymorphism imparted significant risk towards the development of psoriasis in North Indian population. Highlighting the role of IL-10 cytokine in the pathogenesis of psoriasis will help in the development of psoriasis management.

Sections du résumé

BACKGROUND BACKGROUND
IL-10 is an important pleiotropic, immunoregulatory and anti-inflammatory cytokine which plays a significant role in the pathogenesis of psoriasis.
OBJECTIVE OBJECTIVE
The aim of the present study was to determine whether the three polymorphic sites of the IL-10 gene, haplotype and serum level confer susceptibility to psoriasis.
METHOD METHODS
200 psoriatic patients and 200 controls were genotyped for three IL-10 polymorphic sites by ARMS polymerase chain reaction. Serum levels of IL -10 were measured by ELISA.
RESULTS RESULTS
Our results demonstrated that polymorphism of IL-10 -592 C/A (adjusted* OR = 9.25; 95% CI =3.16- 27.06) and IL-10 1082 A/G (adjusted* OR = 4.28; (95% CI =1.46- 12.56) was found to be in association with increased risk of psoriasis while as IL- 10 819 C/T (adjusted* OR= 1.60; (95% CI = 0.65-3.95) polymorphism does not show any significant association with the risk of psoriasis. HT7 GTC haplotype is associated with increased risk of psoriasis. Serum levels of IL-10 were found to be significantly low in patients, as compared to controls with a non-significant correlation between serum IL-10 level and psoriasis severity.
CONCLUSION CONCLUSIONS
IL-10 polymorphism imparted significant risk towards the development of psoriasis in North Indian population. Highlighting the role of IL-10 cytokine in the pathogenesis of psoriasis will help in the development of psoriasis management.

Identifiants

pubmed: 30147018
pii: EMIDDT-EPUB-92583
doi: 10.2174/1871530318666180821161629
doi:

Substances chimiques

IL10 protein, human 0
Interleukin-10 130068-27-8
DNA 9007-49-2

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

53-58

Informations de copyright

Copyright© Bentham Science Publishers; For any queries, please email at epub@benthamscience.net.

Auteurs

Wani Aadil (W)

Department of Human Genetics, Punjabi University, Patiala, Punjab, India.
Department of Biochemistry, University of Kashmir Srinagar, Srinagar, Jammu and Kashmir, India.

Rajinder Kaur (R)

Department of Human Genetics, Punjabi University, Patiala, Punjab, India.

Bashir Ahmad Ganai (BA)

Centre of Research and Development (CORD) University of Kashmir Srinagar, Jammu and Kashmir, India.

Tahseena Akhtar (T)

Department of Human Genetics, Punjabi University, Patiala, Punjab, India.

Tarun Narang (T)

Department of Dermatology, Venerology and Leprology Post Graduate Institute of Medical Education and Research Chandigarh, Chandigarh, India.

Iffat Hassan (I)

Department of Dermatology, Venerology and Leprology SMHS Srinagar, Srinagar, Jammu and Kashmir, India.

Sunil Kumar (S)

Department of Human Genetics, Punjabi University, Patiala, Punjab, India.

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