The Current and Evolving Landscape of First-Line Treatments for Advanced Renal Cell Carcinoma.

Immune checkpoint inhibitors Immunotherapy Renal cell carcinoma Targeted therapy Tyrosine kinase inhibitors

Journal

The oncologist
ISSN: 1549-490X
Titre abrégé: Oncologist
Pays: England
ID NLM: 9607837

Informations de publication

Date de publication:
03 2019
Historique:
received: 05 05 2018
accepted: 05 07 2018
pubmed: 31 8 2018
medline: 4 6 2020
entrez: 31 8 2018
Statut: ppublish

Résumé

Agents targeting the vascular endothelial growth factor (VEGF) and its receptors (VEGFRs), as well as the mammalian target of rapamycin (mTOR) and immune checkpoint receptor programmed death 1 (PD-1) signaling pathway have improved clinical outcomes for patients with advanced renal cell carcinoma (RCC). The VEGFR tyrosine kinase inhibitors (TKIs) pazopanib and sunitinib are FDA-approved first-line treatment options for advanced RCC; however, other treatment options in this setting are available, including the recently approved combination of nivolumab (anti-PD-1) and ipilimumab (anti-cytotoxic T-lymphocyte-associated protein-4 [CTLA-4]) for patients with intermediate or poor risk. Unfortunately, treatment guideline recommendations provide little guidance to aid first-line treatment choice. In addition, several ongoing randomized phase III trials of investigational first-line regimens may complicate the RCC treatment paradigm if these agents gain approval. This article reviews clinical trial and real-world evidence for currently approved and investigational first-line treatment regimens for advanced RCC and provides clinical evidence to aid first-line treatment selection. IMPLICATIONS FOR PRACTICE: Vascular endothelial growth factor receptor tyrosine kinase inhibitors are approved by the U.S. Food and Drug Administration as first-line treatment options for advanced renal cell carcinoma; however, the treatment paradigm is rapidly evolving. The combination of nivolumab plus ipilimumab was recently approved for intermediate- and poor-risk patients, and other combination strategies and novel first-line agents will likely be introduced soon.

Identifiants

pubmed: 30158285
pii: theoncologist.2018-0267
doi: 10.1634/theoncologist.2018-0267
pmc: PMC6519762
doi:

Substances chimiques

Protein Kinase Inhibitors 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

338-348

Informations de copyright

© AlphaMed Press 2018.

Déclaration de conflit d'intérêts

Disclosures of potential conflicts of interest may be found at the end of this article.

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Auteurs

Emiliano Calvo (E)

Centro Integral Oncológico Clara Campal and START Madrid, Madrid, Spain emiliano.calvo@startmadrid.com.

Camillio Porta (C)

Medical Oncology, I.R.C.C.S. San Matteo University Hospital Foundation, Pavia, Italy.

Viktor Grünwald (V)

Clinic for Hematology, Hemostaseology, Oncology & Stem Cell Transplantation, Medical School of Hannover, Hannover, Germany.

Bernard Escudier (B)

Department of Medical Oncology, Gustave Roussy, Villejuif, France.

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