The Role of Xenobiotic Receptors on Hepatic Glycolipid Metabolism.


Journal

Current drug metabolism
ISSN: 1875-5453
Titre abrégé: Curr Drug Metab
Pays: Netherlands
ID NLM: 100960533

Informations de publication

Date de publication:
2019
Historique:
received: 05 01 2018
revised: 13 03 2018
accepted: 20 08 2018
pubmed: 20 9 2018
medline: 25 7 2019
entrez: 20 9 2018
Statut: ppublish

Résumé

PXR (Pregnane X Receptor) and CAR (Constitutive Androstane Receptor) are termed as xenobiotic receptors, which are known as core factors in regulation of the transcription of metabolic enzymes and drug transporters. However, accumulating evidence has shown that PXR and CAR exert their effects on energy metabolism through the regulation of gluconeogenesis, lipogenesis and β-oxidation. Therefore, in this review, we are trying to summary recent advances to show how xenobiotic receptors regulate energy metabolism. A structured search of databases has been performed by using focused review topics. According to conceptual framework, the main idea of research literature was summarized and presented. For introduction of each receptor, the general introduction and the critical functions in hepatic glucose and lipid metabolism have been included. Recent important studies have shown that CAR acts as a negative regulator of lipogenesis, gluconeogenesis and β -oxidation. PXR activation induces lipogenesis, inhibits gluconeogenesis and inhabits β-oxidation. In this review, the importance of xenobiotic receptors in hepatic glucose and lipid metabolism has been confirmed. Therefore, PXR and CAR may become new therapeutic targets for metabolic syndrome, including obesity and diabetes. However, further research is required to promote the clinical application of this new energy metabolism function of xenobiotic receptors.

Sections du résumé

BACKGROUND BACKGROUND
PXR (Pregnane X Receptor) and CAR (Constitutive Androstane Receptor) are termed as xenobiotic receptors, which are known as core factors in regulation of the transcription of metabolic enzymes and drug transporters. However, accumulating evidence has shown that PXR and CAR exert their effects on energy metabolism through the regulation of gluconeogenesis, lipogenesis and β-oxidation. Therefore, in this review, we are trying to summary recent advances to show how xenobiotic receptors regulate energy metabolism.
METHODS METHODS
A structured search of databases has been performed by using focused review topics. According to conceptual framework, the main idea of research literature was summarized and presented.
RESULTS RESULTS
For introduction of each receptor, the general introduction and the critical functions in hepatic glucose and lipid metabolism have been included. Recent important studies have shown that CAR acts as a negative regulator of lipogenesis, gluconeogenesis and β -oxidation. PXR activation induces lipogenesis, inhibits gluconeogenesis and inhabits β-oxidation.
CONCLUSION CONCLUSIONS
In this review, the importance of xenobiotic receptors in hepatic glucose and lipid metabolism has been confirmed. Therefore, PXR and CAR may become new therapeutic targets for metabolic syndrome, including obesity and diabetes. However, further research is required to promote the clinical application of this new energy metabolism function of xenobiotic receptors.

Identifiants

pubmed: 30227815
pii: CDM-EPUB-93130
doi: 10.2174/1389200219666180918152241
doi:

Substances chimiques

Constitutive Androstane Receptor 0
Glycolipids 0
Pregnane X Receptor 0
Receptors, Cytoplasmic and Nuclear 0
Glucose IY9XDZ35W2

Types de publication

Journal Article Review

Langues

eng

Pagination

29-35

Informations de copyright

Copyright© Bentham Science Publishers; For any queries, please email at epub@benthamscience.net.

Auteurs

Ke Chen (K)

Department of Hepatobiliary Surgery, The Second Affiliated Hospital and Yuying Children's Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, China.

Jinwei Zhong (J)

Department of Gastroenterology, The Second Affiliated Hospital and Yuying Children's Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, China.

Lin Hu (L)

Pi-wei Institute, Guangzhou University of Chinese Medicine, Guangzhou, China.

Ruliu Li (R)

Pi-wei Institute, Guangzhou University of Chinese Medicine, Guangzhou, China.

Qun Du (Q)

Pi-wei Institute, Guangzhou University of Chinese Medicine, Guangzhou, China.

Jiazhong Cai (J)

Pi-wei Institute, Guangzhou University of Chinese Medicine, Guangzhou, China.

Yanwu Li (Y)

Pi-wei Institute, Guangzhou University of Chinese Medicine, Guangzhou, China.

Yong Gao (Y)

Pi-wei Institute, Guangzhou University of Chinese Medicine, Guangzhou, China.

Xiaona Cui (X)

Department of Pathogenic Biology and Immunology, Jiangsu Key Laboratory of Immunity and Metabolism, Xuzhou Medical University, Xuzhou, Jiangsu, China.

Xiaoying Yang (X)

Department of Pathogenic Biology and Immunology, Jiangsu Key Laboratory of Immunity and Metabolism, Xuzhou Medical University, Xuzhou, Jiangsu, China.

Xiaojie Wu (X)

Department of Immunology, Binzhou Medical University, Yantai, Shangdong, China.

Lu Yao (L)

Jilin Medical University, Jilin, China.

Juji Dai (J)

School of Basic Medical Sciences, Wenzhou Medical University, Wenzhou, Zhejiang, China.

Yan Wang (Y)

Department of Otolaryngology, The Second Affiliated Hospital & Yuying Children's Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, China.

Haiyong Jin (H)

Department of Otolaryngology, The Second Affiliated Hospital & Yuying Children's Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, China.

Articles similaires

[Redispensing of expensive oral anticancer medicines: a practical application].

Lisanne N van Merendonk, Kübra Akgöl, Bastiaan Nuijen
1.00
Humans Antineoplastic Agents Administration, Oral Drug Costs Counterfeit Drugs

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
1.00
Humans Yoga Low Back Pain Female Male

Classifications MeSH