Maternal suppplementation with conjugated linoleic acid reduce anxiety and lipid peroxidation in the offspring brain.


Journal

Journal of affective disorders
ISSN: 1573-2517
Titre abrégé: J Affect Disord
Pays: Netherlands
ID NLM: 7906073

Informations de publication

Date de publication:
15 01 2019
Historique:
received: 10 11 2017
revised: 09 08 2018
accepted: 10 09 2018
pubmed: 22 9 2018
medline: 26 2 2019
entrez: 22 9 2018
Statut: ppublish

Résumé

Maternal consumption of fatty acids can alter neuronal membrane function, synaptic connections, and protect the brain from alterations caused by disturbances such as lipid peroxidation and anxiety in the offspring. We aimed to investigate how the maternal consumption of conjugated linoleic acid (CLA) interferes in anxiety behavior of the offspring and cerebral lipid peroxidation. Three groups were formed: control (CG) - diet without CLA; CLA1 - diet containing 1% of CLA; and CLA3 - diet containing 3% of CLA. These diets were offered to the mothers from the 7th day of gestation until the end of lactation. The following behavioral tests were used: Elevated plus maze (EPM), Open Field (OF) and Light-dark Box (LDB). Levels of malondialdehyde (MDA) and glutathione were measured in the offspring's brains. Data were analyzed by ANOVA followed by the Holm-Sidak post-test or the Kruskal-Wallis test (p < 0.05). CLA1 and CLA3 showed higher number of entries in the open arms and time spent in the central area in EPM, they translocated and ambulated more in the clear area of the LDB and presented more rearing in the OF compared to CG (p < 0.05); moreover, they presented higher concentration of glutathione and lower MDA in brain tissue (p < 0.05). We evaluated the effect of maternal consumption of CLA on anxiety and lipid peroxidation in rats' offspring, but a similar study should be performed in humans. Maternal intake of CLA induced a decrease in the parameters of anxiety and cerebral lipid peroxidation in the offspring.

Sections du résumé

BACKGROUND
Maternal consumption of fatty acids can alter neuronal membrane function, synaptic connections, and protect the brain from alterations caused by disturbances such as lipid peroxidation and anxiety in the offspring. We aimed to investigate how the maternal consumption of conjugated linoleic acid (CLA) interferes in anxiety behavior of the offspring and cerebral lipid peroxidation.
METHODS
Three groups were formed: control (CG) - diet without CLA; CLA1 - diet containing 1% of CLA; and CLA3 - diet containing 3% of CLA. These diets were offered to the mothers from the 7th day of gestation until the end of lactation. The following behavioral tests were used: Elevated plus maze (EPM), Open Field (OF) and Light-dark Box (LDB). Levels of malondialdehyde (MDA) and glutathione were measured in the offspring's brains. Data were analyzed by ANOVA followed by the Holm-Sidak post-test or the Kruskal-Wallis test (p < 0.05).
RESULTS
CLA1 and CLA3 showed higher number of entries in the open arms and time spent in the central area in EPM, they translocated and ambulated more in the clear area of the LDB and presented more rearing in the OF compared to CG (p < 0.05); moreover, they presented higher concentration of glutathione and lower MDA in brain tissue (p < 0.05).
LIMITATIONS
We evaluated the effect of maternal consumption of CLA on anxiety and lipid peroxidation in rats' offspring, but a similar study should be performed in humans.
CONCLUSIONS
Maternal intake of CLA induced a decrease in the parameters of anxiety and cerebral lipid peroxidation in the offspring.

Identifiants

pubmed: 30236761
pii: S0165-0327(17)32321-2
doi: 10.1016/j.jad.2018.09.020
pii:
doi:

Substances chimiques

Linoleic Acids, Conjugated 0
Malondialdehyde 4Y8F71G49Q
Glutathione GAN16C9B8O

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

75-82

Informations de copyright

Copyright © 2018 Elsevier B.V. All rights reserved.

Auteurs

Michelly Pires Queiroz (MP)

Program of Food Science and Tecnology, Federal University of Paraiba, Cidade Universitária, s/n - Castelo Branco III, João Pessoa, PB 58051-085, Brazil. Electronic address: queiroz_m.p@hotmail.com.

Martiniano da Silva Lima (MDS)

Laboratory of Experimental Nutrition, Department of Nutrition, Federal University of Campina Grande, Cuité, CG, Brazil.

Marília Ferreira Frazão Tavares de Melo (MFFT)

Laboratory of Experimental Nutrition, Department of Nutrition, Federal University of Campina Grande, Cuité, CG, Brazil.

Camila Carolina de Menezes Santos Bertozzo (CCMS)

Laboratory of Experimental Nutrition, Department of Nutrition, Federal University of Campina Grande, Cuité, CG, Brazil.

Daline Fernandes de Araújo (DF)

Department of Biofísica and Pharmacology, Federal University of Rio Grande do Norte, Natal, RN, Brazil.

Gerlane Coelho Bernardo Guerra (GCB)

Department of Biofísica and Pharmacology, Federal University of Rio Grande do Norte, Natal, RN, Brazil.

Rita de Cassia Ramos do Egypto Queiroga (RCRDE)

Program of Food Science and Tecnology, Federal University of Paraiba, Cidade Universitária, s/n - Castelo Branco III, João Pessoa, PB 58051-085, Brazil; Laboratory of Bromatology, Department of Nutrition, Federal University of Paraiba, João Pessoa, PB, Brazil. Electronic address: rcqueiroga@uol.com.br.

Juliana Késsia Barbosa Soares (JKB)

Program of Food Science and Tecnology, Federal University of Paraiba, Cidade Universitária, s/n - Castelo Branco III, João Pessoa, PB 58051-085, Brazil; Laboratory of Experimental Nutrition, Department of Nutrition, Federal University of Campina Grande, Cuité, CG, Brazil.

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Classifications MeSH