Clinical and dermoscopic features of cutaneous BAP1-inactivated melanocytic tumors: Results of a multicenter case-control study by the International Dermoscopy Society.


Journal

Journal of the American Academy of Dermatology
ISSN: 1097-6787
Titre abrégé: J Am Acad Dermatol
Pays: United States
ID NLM: 7907132

Informations de publication

Date de publication:
Jun 2019
Historique:
received: 03 07 2018
revised: 23 08 2018
accepted: 06 09 2018
pubmed: 24 9 2018
medline: 29 10 2019
entrez: 24 9 2018
Statut: ppublish

Résumé

Multiple BRCA1-associated protein 1 (BAP1)-inactivated melanocytic tumors (BIMTs) have been associated with a familial cancer syndrome involving germline mutations in BAP1. We sought to describe the clinical and dermoscopic features of BIMTs. This was a retrospective, multicenter, case-control study. Participating centers contributed clinical data, dermoscopic images, and histopathologic data of biopsy-proven BIMTs. We compared the dermoscopic features between BIMTs and control patients. The dataset consisted of 48 BIMTs from 31 patients (22 women; median age 37 years) and 80 control patients. Eleven patients had a BAP1 germline mutation. Clinically, most BIMTs presented as pink, dome-shaped papules (n = 24). Dermoscopically, we identified 5 patterns: structureless pink-to-tan with irregular eccentric dots/globules (n = 14, 29.8%); structureless pink-to-tan with peripheral vessels (n = 10, 21.3%); structureless pink-to-tan (n = 7, 14.9%); a network with raised, structureless, pink-to-tan areas (n = 7, 14.9%); and globular pattern (n = 4, 8.5%). The structureless with eccentric dots/globules pattern and network with raised structureless areas pattern were only identified in BIMT and were more common in patients with BAP1 germline mutations (P < .0001 and P = .001, respectively). Limitations included our small sample size, retrospective design, the absence of germline genetic testing in all patients, and inclusion bias toward more atypical-looking BIMTs. Dome-shaped papules with pink-to-tan structureless areas and peripheral irregular dots/globules or network should raise the clinical suspicion for BIMT.

Sections du résumé

BACKGROUND BACKGROUND
Multiple BRCA1-associated protein 1 (BAP1)-inactivated melanocytic tumors (BIMTs) have been associated with a familial cancer syndrome involving germline mutations in BAP1.
OBJECTIVES OBJECTIVE
We sought to describe the clinical and dermoscopic features of BIMTs.
METHODS METHODS
This was a retrospective, multicenter, case-control study. Participating centers contributed clinical data, dermoscopic images, and histopathologic data of biopsy-proven BIMTs. We compared the dermoscopic features between BIMTs and control patients.
RESULTS RESULTS
The dataset consisted of 48 BIMTs from 31 patients (22 women; median age 37 years) and 80 control patients. Eleven patients had a BAP1 germline mutation. Clinically, most BIMTs presented as pink, dome-shaped papules (n = 24). Dermoscopically, we identified 5 patterns: structureless pink-to-tan with irregular eccentric dots/globules (n = 14, 29.8%); structureless pink-to-tan with peripheral vessels (n = 10, 21.3%); structureless pink-to-tan (n = 7, 14.9%); a network with raised, structureless, pink-to-tan areas (n = 7, 14.9%); and globular pattern (n = 4, 8.5%). The structureless with eccentric dots/globules pattern and network with raised structureless areas pattern were only identified in BIMT and were more common in patients with BAP1 germline mutations (P < .0001 and P = .001, respectively).
LIMITATIONS CONCLUSIONS
Limitations included our small sample size, retrospective design, the absence of germline genetic testing in all patients, and inclusion bias toward more atypical-looking BIMTs.
CONCLUSIONS CONCLUSIONS
Dome-shaped papules with pink-to-tan structureless areas and peripheral irregular dots/globules or network should raise the clinical suspicion for BIMT.

Identifiants

pubmed: 30244062
pii: S0190-9622(18)32591-X
doi: 10.1016/j.jaad.2018.09.014
pmc: PMC6426687
mid: NIHMS992287
pii:
doi:

Substances chimiques

BAP1 protein, human 0
Tumor Suppressor Proteins 0
Ubiquitin Thiolesterase EC 3.4.19.12

Types de publication

Comparative Study Journal Article Multicenter Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

1585-1593

Subventions

Organisme : NCI NIH HHS
ID : P30 CA008748
Pays : United States

Informations de copyright

Copyright © 2018 American Academy of Dermatology, Inc. Published by Elsevier Inc. All rights reserved.

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Auteurs

Oriol Yélamos (O)

Dermatology Service, Department of Medicine, Memorial Sloan Kettering Cancer Center, New York, New York; Dermatology Department, Hospital Clínic, Institut d'Investigacions Biomediques August Pi i Sunyer, Universitat de Barcelona, and CIBER de Enfermedades Raras, Instituto de Salud Carlos III, Barcelona, Spain. Electronic address: oyelamos@gmail.com.

Cristián Navarrete-Dechent (C)

Dermatology Service, Department of Medicine, Memorial Sloan Kettering Cancer Center, New York, New York; Department of Dermatology, Facultad de Medicina, Pontificia Universidad Católica de Chile, Santiago, Chile.

Michael A Marchetti (MA)

Dermatology Service, Department of Medicine, Memorial Sloan Kettering Cancer Center, New York, New York.

Tova Rogers (T)

Dermatology Service, Department of Medicine, Memorial Sloan Kettering Cancer Center, New York, New York.

Zoe Apalla (Z)

First Department of Dermatology, Aristotle University, Thessaloniki, Greece.

Philippe Bahadoran (P)

Dermatology Department, Centre Hospitalier Universitaire de Nice, Nice, France.

Nuria Blázquez-Sánchez (N)

Dermatology Department, Hospital Costa del Sol, Marbella, Spain.

Klaus Busam (K)

Pathology Department, Memorial Sloan Kettering Cancer Center, New York, New York.

Cristina Carrera (C)

Dermatology Department, Hospital Clínic, Institut d'Investigacions Biomediques August Pi i Sunyer, Universitat de Barcelona, and CIBER de Enfermedades Raras, Instituto de Salud Carlos III, Barcelona, Spain.

Stephen W Dusza (SW)

Dermatology Service, Department of Medicine, Memorial Sloan Kettering Cancer Center, New York, New York.

Arnaud de la Fouchardière (A)

Département de Biopathologie, Centre Léon Bérard, Lyon, France.

Gerardo Ferrara (G)

Anatomic Pathology Unit, Hospital of Macerata, Macerata, Italy.

Pedram Gerami (P)

Dermatology Department, Feinberg School of Medicine, Northwestern University, Chicago, Illinois.

Harald Kittler (H)

Department of Dermatology, Medical University of Vienna, Vienna, Austria.

Aimilios Lallas (A)

First Department of Dermatology, Aristotle University, Thessaloniki, Greece.

Josep Malvehy (J)

Dermatology Department, Hospital Clínic, Institut d'Investigacions Biomediques August Pi i Sunyer, Universitat de Barcelona, and CIBER de Enfermedades Raras, Instituto de Salud Carlos III, Barcelona, Spain.

José F Millán-Cayetano (JF)

Dermatology Department, Hospital Costa del Sol, Marbella, Spain.

Kelly C Nelson (KC)

Dermatology Department, The University of Texas MD Anderson Cancer Center, Houston, Texas.

Victor Li Quan (VL)

Dermatology Department, Feinberg School of Medicine, Northwestern University, Chicago, Illinois.

Susana Puig (S)

Dermatology Department, Hospital Clínic, Institut d'Investigacions Biomediques August Pi i Sunyer, Universitat de Barcelona, and CIBER de Enfermedades Raras, Instituto de Salud Carlos III, Barcelona, Spain.

Howard Stevens (H)

Skin Care Network, Barnet, London, United Kingdom.

Luc Thomas (L)

Department of Dermatology, Lyon 1 University, Centre Hospitalier Lyon Sud and Lyon's Cancer Research Center INSERM U1052 - CNRS UMR5286, Lyon, France.

Ashfaq A Marghoob (AA)

Dermatology Service, Department of Medicine, Memorial Sloan Kettering Cancer Center, New York, New York.

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Classifications MeSH