Reverse left ventricular structural remodeling after catheter ablation of atrial fibrillation in patients with preserved left ventricular function: Insights from cardiovascular magnetic resonance native T1 mapping.


Journal

Heart rhythm
ISSN: 1556-3871
Titre abrégé: Heart Rhythm
Pays: United States
ID NLM: 101200317

Informations de publication

Date de publication:
03 2019
Historique:
received: 23 07 2018
pubmed: 25 9 2018
medline: 10 10 2020
entrez: 25 9 2018
Statut: ppublish

Résumé

Catheter ablation of atrial fibrillation (AF) improves left ventricular (LV) function in patients with LV systolic dysfunction, suggestive of underlying arrhythmia-induced adverse remodeling. The objectives of this study were to evaluate whether arrhythmia-induced LV remodeling occurs in patients with AF and preserved LV systolic function and to assess whether this remodeling is reversible after restoration of sinus rhythm by catheter ablation. Forty-three patients with AF and preserved LV systolic function (LV ejection fraction 62% ± 7%) underwent cardiovascular magnetic resonance (CMR) imaging before catheter ablation including native T1 mapping using a modified Look-Locker inversion recovery sequence. Twenty-five patients underwent follow-up CMR 3 months after catheter ablation. Twenty-two matched controls without AF underwent the same CMR protocol. Patients with AF had higher baseline LV native T1 values than did controls (1296 ± 55 ms vs 1243 ± 55 ms; P < .01). During a median follow-up of 9 months (interquartile range 4-14 months), 17 patients (40%) experienced AF recurrence. No differences in baseline T1 values were observed between patients with and without AF recurrence. There was a significant decrease in native T1 values in patients with successful restoration of sinus rhythm after catheter ablation at 3 months of follow-up CMR (1300 ± 45 ms vs 1270 ± 55 ms; P < .01), while they remain unchanged in patients with AF recurrence (1303 ± 51 ms vs 1309 ± 31 ms; P = .64). These preliminary results suggest that subclinical arrhythmia-induced LV structural remodeling occurs in patients with AF and preserved LV systolic function. This remodeling might be reversible after catheter ablation with successful restoration of sinus rhythm as quantified noninvasively and gadolinium-free by CMR native T1 mapping.

Sections du résumé

BACKGROUND
Catheter ablation of atrial fibrillation (AF) improves left ventricular (LV) function in patients with LV systolic dysfunction, suggestive of underlying arrhythmia-induced adverse remodeling.
OBJECTIVES
The objectives of this study were to evaluate whether arrhythmia-induced LV remodeling occurs in patients with AF and preserved LV systolic function and to assess whether this remodeling is reversible after restoration of sinus rhythm by catheter ablation.
METHODS
Forty-three patients with AF and preserved LV systolic function (LV ejection fraction 62% ± 7%) underwent cardiovascular magnetic resonance (CMR) imaging before catheter ablation including native T1 mapping using a modified Look-Locker inversion recovery sequence. Twenty-five patients underwent follow-up CMR 3 months after catheter ablation. Twenty-two matched controls without AF underwent the same CMR protocol.
RESULTS
Patients with AF had higher baseline LV native T1 values than did controls (1296 ± 55 ms vs 1243 ± 55 ms; P < .01). During a median follow-up of 9 months (interquartile range 4-14 months), 17 patients (40%) experienced AF recurrence. No differences in baseline T1 values were observed between patients with and without AF recurrence. There was a significant decrease in native T1 values in patients with successful restoration of sinus rhythm after catheter ablation at 3 months of follow-up CMR (1300 ± 45 ms vs 1270 ± 55 ms; P < .01), while they remain unchanged in patients with AF recurrence (1303 ± 51 ms vs 1309 ± 31 ms; P = .64).
CONCLUSION
These preliminary results suggest that subclinical arrhythmia-induced LV structural remodeling occurs in patients with AF and preserved LV systolic function. This remodeling might be reversible after catheter ablation with successful restoration of sinus rhythm as quantified noninvasively and gadolinium-free by CMR native T1 mapping.

Identifiants

pubmed: 30248459
pii: S1547-5271(18)30939-1
doi: 10.1016/j.hrthm.2018.09.016
pii:
doi:

Types de publication

Journal Article Observational Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

424-432

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2018 Heart Rhythm Society. Published by Elsevier Inc. All rights reserved.

Auteurs

Johannes T Kowallick (JT)

Institute for Diagnostic and Interventional Radiology, University Medical Centre Göttingen, Georg-August-University, Göttingen, Germany; DZHK (German Centre for Cardiovascular Research), partner site Göttingen, Göttingen, Germany. Electronic address: johannes.kowallick@med.uni-goettingen.de.

Wieland Staab (W)

Institute for Diagnostic and Interventional Radiology, University Medical Centre Göttingen, Georg-August-University, Göttingen, Germany; DZHK (German Centre for Cardiovascular Research), partner site Göttingen, Göttingen, Germany.

Andreas Schuster (A)

DZHK (German Centre for Cardiovascular Research), partner site Göttingen, Göttingen, Germany; Department of Cardiology and Pneumology, University Medical Centre Göttingen, Georg-August-University, Göttingen, Germany; Department of Cardiology, Royal North Shore Hospital, The Kolling Institute, Northern Clinical School, University of Sydney, Sydney, Australia.

Sören J Backhaus (SJ)

DZHK (German Centre for Cardiovascular Research), partner site Göttingen, Göttingen, Germany; Department of Cardiology and Pneumology, University Medical Centre Göttingen, Georg-August-University, Göttingen, Germany.

Mark Weber-Krüger (M)

DZHK (German Centre for Cardiovascular Research), partner site Göttingen, Göttingen, Germany; Department of Cardiology and Pneumology, University Medical Centre Göttingen, Georg-August-University, Göttingen, Germany.

Lukas Bauer (L)

Institute for Diagnostic and Interventional Radiology, University Medical Centre Göttingen, Georg-August-University, Göttingen, Germany; DZHK (German Centre for Cardiovascular Research), partner site Göttingen, Göttingen, Germany.

Christian Sohns (C)

Electrophysiology Bremen, Heart Center Bremen, Bremen, Germany.

Joachim Lotz (J)

Institute for Diagnostic and Interventional Radiology, University Medical Centre Göttingen, Georg-August-University, Göttingen, Germany; DZHK (German Centre for Cardiovascular Research), partner site Göttingen, Göttingen, Germany.

Gerd Hasenfuß (G)

DZHK (German Centre for Cardiovascular Research), partner site Göttingen, Göttingen, Germany; Department of Cardiology and Pneumology, University Medical Centre Göttingen, Georg-August-University, Göttingen, Germany.

Lars Lüthje (L)

DZHK (German Centre for Cardiovascular Research), partner site Göttingen, Göttingen, Germany; Department of Cardiology and Pneumology, University Medical Centre Göttingen, Georg-August-University, Göttingen, Germany.

Markus Zabel (M)

DZHK (German Centre for Cardiovascular Research), partner site Göttingen, Göttingen, Germany; Department of Cardiology and Pneumology, University Medical Centre Göttingen, Georg-August-University, Göttingen, Germany.

Leonard Bergau (L)

DZHK (German Centre for Cardiovascular Research), partner site Göttingen, Göttingen, Germany; Department of Cardiology and Pneumology, University Medical Centre Göttingen, Georg-August-University, Göttingen, Germany.

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