Age-Related Changes in HAPLN1 Increase Lymphatic Permeability and Affect Routes of Melanoma Metastasis.


Journal

Cancer discovery
ISSN: 2159-8290
Titre abrégé: Cancer Discov
Pays: United States
ID NLM: 101561693

Informations de publication

Date de publication:
01 2019
Historique:
received: 26 02 2018
revised: 31 07 2018
accepted: 19 09 2018
pubmed: 4 10 2018
medline: 7 3 2020
entrez: 4 10 2018
Statut: ppublish

Résumé

Older patients with melanoma have lower rates of sentinel lymph node (LN) metastases yet paradoxically have inferior survival. Patient age correlated with an inability to retain Technetium radiotracer during sentinel LN biopsy in more than 1,000 patients, and high Technetium counts correlated to better survival. We hypothesized that loss of integrity in the lymphatic vasculature due to extracellular matrix (ECM) degradation might play a role. We have implicated HAPLN1 in age-dependent ECM degradation in the dermis. Here, we queried whether HAPLN1 could be altered in the lymphatic ECM. Lymphatic HAPLN1 expression was prognostic of long-term patient survival. Adding recombinant HAPLN1 to aged fibroblast ECMs

Identifiants

pubmed: 30279172
pii: 2159-8290.CD-18-0168
doi: 10.1158/2159-8290.CD-18-0168
pmc: PMC6328344
mid: NIHMS1508175
doi:

Substances chimiques

Extracellular Matrix Proteins 0
Proteoglycans 0
link protein 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

82-95

Subventions

Organisme : NCI NIH HHS
ID : K99 CA208012
Pays : United States
Organisme : NCI NIH HHS
ID : P30 CA010815
Pays : United States
Organisme : NCI NIH HHS
ID : P01 CA114046
Pays : United States
Organisme : NCI NIH HHS
ID : R01 CA174746
Pays : United States
Organisme : NCI NIH HHS
ID : P50 CA174523
Pays : United States
Organisme : NCI NIH HHS
ID : R01 CA207935
Pays : United States

Commentaires et corrections

Type : CommentIn

Informations de copyright

©2018 American Association for Cancer Research.

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Auteurs

Brett L Ecker (BL)

Department of Surgery, University of Pennsylvania, Philadelphia, Pennsylvania.
The Wistar Institute, Philadelphia, Pennsylvania.

Amanpreet Kaur (A)

The Wistar Institute, Philadelphia, Pennsylvania.
University of the Sciences, Philadelphia, Pennsylvania.

Stephen M Douglass (SM)

The Wistar Institute, Philadelphia, Pennsylvania.

Marie R Webster (MR)

The Wistar Institute, Philadelphia, Pennsylvania.

Filipe V Almeida (FV)

The Wistar Institute, Philadelphia, Pennsylvania.

Gloria E Marino (GE)

The Wistar Institute, Philadelphia, Pennsylvania.
Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania.

Andrew J Sinnamon (AJ)

Department of Surgery, University of Pennsylvania, Philadelphia, Pennsylvania.

Madalyn G Neuwirth (MG)

Department of Surgery, University of Pennsylvania, Philadelphia, Pennsylvania.

Gretchen M Alicea (GM)

The Wistar Institute, Philadelphia, Pennsylvania.
University of the Sciences, Philadelphia, Pennsylvania.

Abibatou Ndoye (A)

The Wistar Institute, Philadelphia, Pennsylvania.
University of the Sciences, Philadelphia, Pennsylvania.

Mitchell Fane (M)

The Wistar Institute, Philadelphia, Pennsylvania.

Xiaowei Xu (X)

Department of Pathology, University of Pennsylvania, Philadelphia, Pennsylvania.

Myung Shin Sim (MS)

The University of California, Los Angeles, California.

Gary B Deutsch (GB)

Northwell Health Institute, Los Angeles, California.

Mark B Faries (MB)

The Angeles Clinic and Research Institute, Cedars Sinai, Los Angeles, California.

Giorgos C Karakousis (GC)

Department of Surgery, University of Pennsylvania, Philadelphia, Pennsylvania.

Ashani T Weeraratna (AT)

The Wistar Institute, Philadelphia, Pennsylvania. aweeraratna@wistar.org.

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