Detailed comparison between the wall thickness and voltages in chronic myocardial infarction.


Journal

Journal of cardiovascular electrophysiology
ISSN: 1540-8167
Titre abrégé: J Cardiovasc Electrophysiol
Pays: United States
ID NLM: 9010756

Informations de publication

Date de publication:
02 2019
Historique:
received: 17 07 2018
revised: 17 09 2018
accepted: 28 09 2018
pubmed: 6 10 2018
medline: 13 3 2020
entrez: 6 10 2018
Statut: ppublish

Résumé

The relationship between the local electrograms (EGMs) and wall thickness (WT) heterogeneity within infarct scars has not been thoroughly described. The relationship between WT and voltages and substrates for ventricular tachycardia (VT) was examined. In 12 consecutive patients with myocardial infarction and VT, WT, defined by a multidetector computed tomography, and voltage were compared. In multicomponent EGMs, amplitudes of both far- and near-field components were manually measured, and the performance of the three-dimensional-mapping system automatic voltage measurement was assessed. Of 15 748 points acquired, 2677 points within 5 mm of the endocardial surface were analyzed. In total, 909 (34.0%) multicomponent EGMs were identified; 785 (86.4%) and 883 (97.1%) were distributed in the WT less than 4 and 5 mm, respectively. Far-field EGM voltages increased linearly from 0.14 mV (0.08-0.28 mV) in the WT: 0 to 1 mm to 0.70 mV (0.43-2.62 mV) in the WT: 4 to 5 mm (ρ = 0.430; P < 0.001), and a significant difference was demonstrated between any two WT-groups (P ≤ 0.001). In contrast, near-field EGM voltages varied from 0.27 mV (0.11-0.44 mV) in the WT: 0 to 1 mm to 0.29 mV (0.17-0.53 mV) in the WT: 4 to 5 mm with a poorer correlation (ρ = 0.062, P = 0.04). The proportion of points where the system automatically measured the voltage on near-field EGMs increased from less than 10% in areas of WT: 4 to 5 mm to 50% in areas less than 2 mm. Of 21 VTs observed, seven hemodynamically stable VTs were mapped and terminated in WT: 1 to 4 mm area. Although far-field voltages gradually increase with the WT, near-field does not. The three-dimensional-mapping system preferentially annotates the near-field components in thinner areas (center of the scar) and the far-field component in thicker areas when building a voltage map. Critical sites of VT are distributed in WT: 1 to 4 mm areas.

Sections du résumé

BACKGROUND
The relationship between the local electrograms (EGMs) and wall thickness (WT) heterogeneity within infarct scars has not been thoroughly described. The relationship between WT and voltages and substrates for ventricular tachycardia (VT) was examined.
METHODS
In 12 consecutive patients with myocardial infarction and VT, WT, defined by a multidetector computed tomography, and voltage were compared. In multicomponent EGMs, amplitudes of both far- and near-field components were manually measured, and the performance of the three-dimensional-mapping system automatic voltage measurement was assessed.
RESULTS
Of 15 748 points acquired, 2677 points within 5 mm of the endocardial surface were analyzed. In total, 909 (34.0%) multicomponent EGMs were identified; 785 (86.4%) and 883 (97.1%) were distributed in the WT less than 4 and 5 mm, respectively. Far-field EGM voltages increased linearly from 0.14 mV (0.08-0.28 mV) in the WT: 0 to 1 mm to 0.70 mV (0.43-2.62 mV) in the WT: 4 to 5 mm (ρ = 0.430; P < 0.001), and a significant difference was demonstrated between any two WT-groups (P ≤ 0.001). In contrast, near-field EGM voltages varied from 0.27 mV (0.11-0.44 mV) in the WT: 0 to 1 mm to 0.29 mV (0.17-0.53 mV) in the WT: 4 to 5 mm with a poorer correlation (ρ = 0.062, P = 0.04). The proportion of points where the system automatically measured the voltage on near-field EGMs increased from less than 10% in areas of WT: 4 to 5 mm to 50% in areas less than 2 mm. Of 21 VTs observed, seven hemodynamically stable VTs were mapped and terminated in WT: 1 to 4 mm area.
CONCLUSIONS
Although far-field voltages gradually increase with the WT, near-field does not. The three-dimensional-mapping system preferentially annotates the near-field components in thinner areas (center of the scar) and the far-field component in thicker areas when building a voltage map. Critical sites of VT are distributed in WT: 1 to 4 mm areas.

Identifiants

pubmed: 30288836
doi: 10.1111/jce.13767
doi:

Types de publication

Comparative Study Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

195-204

Subventions

Organisme : British Heart Foundation
ID : FS/16/71/32487
Pays : United Kingdom

Informations de copyright

© 2018 Wiley Periodicals, Inc.

Auteurs

Masateru Takigawa (M)

Bordeaux University Hospital (CHU), Cardiac Electrophysiology and Cardiac Stimulation Team, IHU Liryc, Electrophysiology and Heart Modeling Institute, fondation Bordeaux Université, Bordeaux, France.

Ruairidh Martin (R)

Bordeaux University Hospital (CHU), Cardiac Electrophysiology and Cardiac Stimulation Team, IHU Liryc, Electrophysiology and Heart Modeling Institute, fondation Bordeaux Université, Bordeaux, France.
Institute of Genetic Medicine, Newcastle University, Newcastle-upon-Tyne, UK.

Ghassen Cheniti (G)

Bordeaux University Hospital (CHU), Cardiac Electrophysiology and Cardiac Stimulation Team, IHU Liryc, Electrophysiology and Heart Modeling Institute, fondation Bordeaux Université, Bordeaux, France.

Takeshi Kitamura (T)

Bordeaux University Hospital (CHU), Cardiac Electrophysiology and Cardiac Stimulation Team, IHU Liryc, Electrophysiology and Heart Modeling Institute, fondation Bordeaux Université, Bordeaux, France.

Konstantinos Vlachos (K)

Bordeaux University Hospital (CHU), Cardiac Electrophysiology and Cardiac Stimulation Team, IHU Liryc, Electrophysiology and Heart Modeling Institute, fondation Bordeaux Université, Bordeaux, France.

Antonio Frontera (A)

Bordeaux University Hospital (CHU), Cardiac Electrophysiology and Cardiac Stimulation Team, IHU Liryc, Electrophysiology and Heart Modeling Institute, fondation Bordeaux Université, Bordeaux, France.

Claire A Martin (CA)

Bordeaux University Hospital (CHU), Cardiac Electrophysiology and Cardiac Stimulation Team, IHU Liryc, Electrophysiology and Heart Modeling Institute, fondation Bordeaux Université, Bordeaux, France.

Felix Bourier (F)

Bordeaux University Hospital (CHU), Cardiac Electrophysiology and Cardiac Stimulation Team, IHU Liryc, Electrophysiology and Heart Modeling Institute, fondation Bordeaux Université, Bordeaux, France.

Anna Lam (A)

Bordeaux University Hospital (CHU), Cardiac Electrophysiology and Cardiac Stimulation Team, IHU Liryc, Electrophysiology and Heart Modeling Institute, fondation Bordeaux Université, Bordeaux, France.

Xavier Pillois (X)

Bordeaux University Hospital (CHU), Cardiac Electrophysiology and Cardiac Stimulation Team, IHU Liryc, Electrophysiology and Heart Modeling Institute, fondation Bordeaux Université, Bordeaux, France.

Josselin Duchateau (J)

Bordeaux University Hospital (CHU), Cardiac Electrophysiology and Cardiac Stimulation Team, IHU Liryc, Electrophysiology and Heart Modeling Institute, fondation Bordeaux Université, Bordeaux, France.

Nicolas Klotz (N)

Bordeaux University Hospital (CHU), Cardiac Electrophysiology and Cardiac Stimulation Team, IHU Liryc, Electrophysiology and Heart Modeling Institute, fondation Bordeaux Université, Bordeaux, France.

Thomas Pambrun (T)

Bordeaux University Hospital (CHU), Cardiac Electrophysiology and Cardiac Stimulation Team, IHU Liryc, Electrophysiology and Heart Modeling Institute, fondation Bordeaux Université, Bordeaux, France.

Arnaud Denis (A)

Bordeaux University Hospital (CHU), Cardiac Electrophysiology and Cardiac Stimulation Team, IHU Liryc, Electrophysiology and Heart Modeling Institute, fondation Bordeaux Université, Bordeaux, France.

Nicolas Derval (N)

Bordeaux University Hospital (CHU), Cardiac Electrophysiology and Cardiac Stimulation Team, IHU Liryc, Electrophysiology and Heart Modeling Institute, fondation Bordeaux Université, Bordeaux, France.

Mélèze Hocini (M)

Bordeaux University Hospital (CHU), Cardiac Electrophysiology and Cardiac Stimulation Team, IHU Liryc, Electrophysiology and Heart Modeling Institute, fondation Bordeaux Université, Bordeaux, France.

Michel Haïssaguerre (M)

Bordeaux University Hospital (CHU), Cardiac Electrophysiology and Cardiac Stimulation Team, IHU Liryc, Electrophysiology and Heart Modeling Institute, fondation Bordeaux Université, Bordeaux, France.

Frédéric Sacher (F)

Bordeaux University Hospital (CHU), Cardiac Electrophysiology and Cardiac Stimulation Team, IHU Liryc, Electrophysiology and Heart Modeling Institute, fondation Bordeaux Université, Bordeaux, France.

Pierre Jaïs (P)

Bordeaux University Hospital (CHU), Cardiac Electrophysiology and Cardiac Stimulation Team, IHU Liryc, Electrophysiology and Heart Modeling Institute, fondation Bordeaux Université, Bordeaux, France.

Hubert Cochet (H)

Bordeaux University Hospital (CHU), Cardiac Electrophysiology and Cardiac Stimulation Team, IHU Liryc, Electrophysiology and Heart Modeling Institute, fondation Bordeaux Université, Bordeaux, France.

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