MESOPIC AND DARK-ADAPTED TWO-COLOR FUNDUS-CONTROLLED PERIMETRY IN GEOGRAPHIC ATROPHY SECONDARY TO AGE-RELATED MACULAR DEGENERATION.


Journal

Retina (Philadelphia, Pa.)
ISSN: 1539-2864
Titre abrégé: Retina
Pays: United States
ID NLM: 8309919

Informations de publication

Date de publication:
Jan 2020
Historique:
pubmed: 10 10 2018
medline: 7 2 2021
entrez: 10 10 2018
Statut: ppublish

Résumé

To investigate retinal sensitivity in the junctional zone of geographic atrophy (GA) secondary to age-related macular degeneration using patient-tailored perimetry grids for mesopic and dark-adapted two-color fundus-controlled perimetry. Twenty-five eyes with GA of 25 patients (prospective, natural-history Directional Spread in Geographic Atrophy study [DSGA; NCT02051998]) and 40 eyes of 40 normal subjects were included. Patient-tailored perimetry grids were generated using annotated fundus autofluorescence data. Customized software positioned test-points along iso-hulls surrounding the GA boundary at distances of 0.43°, 0.86°, 1.29°, 2.15°, and 3.01°. The grids were used for duplicate mesopic and dark-adapted two-color (cyan and red) fundus-controlled perimetry. Age-adjusted reference-data were obtained through regression analysis of normative data followed by spatial interpolation. The mean sensitivity loss for mesopic testing decreased with the distance to GA (-10.3 dB [0.43°], -8.2 dB [0.86°], -7.1 dB [1.29°], -6.8 dB [2.15°], and -6.6 dB [3.01°]; P < 0.01). Dark-adapted cyan sensitivity loss exceeded dark-adapted red sensitivity loss for all iso-hulls (-14.8 vs. -11.7 dB, -13.5 vs. -10.1 dB, -12.8 vs. -9.1 dB, -11.6 vs. -8.2 dB, -10.7 vs. -8.0 dB; P < 0.01). Patient-tailored fundus-controlled perimetry grids allowed for testing of retinal function in the junctional zone of GA with high spatial resolution. A distinct decrease in mesopic sensitivity loss between 0.43° (125 µm) and 1.29° (375 µm) was observed that leveled off at more distant test-points. In proximity to the GA boundary, the results indicate that rod exceeded cone dysfunction.

Identifiants

pubmed: 30300264
doi: 10.1097/IAE.0000000000002337
pii: 00006982-202001000-00021
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

169-180

Références

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Auteurs

Maximilian Pfau (M)

Department of Ophthalmology, University of Bonn, Bonn, Germany.
GRADE Reading Center, University of Bonn, Bonn, Germany.

Philipp L Müller (PL)

Department of Ophthalmology, University of Bonn, Bonn, Germany.
Center for Rare Diseases, University of Bonn, Bonn, Germany; and.

Leon von der Emde (L)

Department of Ophthalmology, University of Bonn, Bonn, Germany.

Moritz Lindner (M)

Department of Ophthalmology, University of Bonn, Bonn, Germany.
The Nuffield Laboratory of Ophthalmology, Department of Clinical Neurosciences, Sleep and Circadian Neuroscience Institute, Nuffield University of Oxford, Oxford, United Kingdom.

Philipp T Möller (PT)

Department of Ophthalmology, University of Bonn, Bonn, Germany.
GRADE Reading Center, University of Bonn, Bonn, Germany.

Monika Fleckenstein (M)

Department of Ophthalmology, University of Bonn, Bonn, Germany.
GRADE Reading Center, University of Bonn, Bonn, Germany.

Frank G Holz (FG)

Department of Ophthalmology, University of Bonn, Bonn, Germany.
GRADE Reading Center, University of Bonn, Bonn, Germany.

Steffen Schmitz-Valckenberg (S)

Department of Ophthalmology, University of Bonn, Bonn, Germany.
GRADE Reading Center, University of Bonn, Bonn, Germany.

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