Fetal Surgery Decreases Anesthesia-Induced Neuroapoptosis in the Mid-Gestational Fetal Ovine Brain.


Journal

Fetal diagnosis and therapy
ISSN: 1421-9964
Titre abrégé: Fetal Diagn Ther
Pays: Switzerland
ID NLM: 9107463

Informations de publication

Date de publication:
2019
Historique:
received: 29 09 2017
accepted: 09 07 2018
pubmed: 15 10 2018
medline: 2 7 2020
entrez: 15 10 2018
Statut: ppublish

Résumé

Studies demonstrating an association between anesthesia and brain cell death (neuroapoptosis) in young animals were performed without accompanying surgery. This study tests the hypothesis that fetal surgery decreases anesthesia-induced neuroapoptosis. Seventy-day-pregnant ewes received 2% isoflurane for 1 h (low dose [LD]) or 4% for 3 h (high dose [HD]) with or without fetal surgery (S). Unexposed fetuses served as controls (C). Fetal brains were processed for neuroapoptosis using anti-caspase-3 antibodies. Data were analyzed using ANOVA. Twenty-eight fetal sheep were evaluated. Dentate gyrus neuroapoptosis was lower in the HD+S group (13.1 ± 3.76 × 105/mm3) than in the HD (19.1 ± 1.40 × 105/mm3, p = 0.012) and C groups (18.3 ± 3.55 × 105/mm3, p = 0.035). In the pyramidal layer of the hippocampus, neuroapoptosis was lower in the HD+S group (8.11 ± 4.88 × 105/mm3) than in the HD (14.8 ± 2.82 × 105/mm3, p = 0.006) and C groups (14.1 ± 4.54 × 105/mm3, p = 0.019). The LD+S group showed a trend towards a significant decrease in neuroapoptosis in the pyramidal layer (LD+S 7.51 ± 1.48 vs. LD 13.5 ± 1.87 vs. C 14.1 ± 4.54 × 105/mm3, p = 0.07) but not in the dentate gyrus. Fetal surgery did not affect neuroapoptosis in the frontal cortex or endplate. Fetal surgery decreases isoflurane-induced neuroapoptosis in the dentate gyrus and the pyramidal layer of mid-gestational fetal sheep. Long-term effects of these observations on memory and learning deserve further exploration.

Sections du résumé

BACKGROUND BACKGROUND
Studies demonstrating an association between anesthesia and brain cell death (neuroapoptosis) in young animals were performed without accompanying surgery. This study tests the hypothesis that fetal surgery decreases anesthesia-induced neuroapoptosis.
MATERIALS AND METHODS METHODS
Seventy-day-pregnant ewes received 2% isoflurane for 1 h (low dose [LD]) or 4% for 3 h (high dose [HD]) with or without fetal surgery (S). Unexposed fetuses served as controls (C). Fetal brains were processed for neuroapoptosis using anti-caspase-3 antibodies. Data were analyzed using ANOVA.
RESULTS RESULTS
Twenty-eight fetal sheep were evaluated. Dentate gyrus neuroapoptosis was lower in the HD+S group (13.1 ± 3.76 × 105/mm3) than in the HD (19.1 ± 1.40 × 105/mm3, p = 0.012) and C groups (18.3 ± 3.55 × 105/mm3, p = 0.035). In the pyramidal layer of the hippocampus, neuroapoptosis was lower in the HD+S group (8.11 ± 4.88 × 105/mm3) than in the HD (14.8 ± 2.82 × 105/mm3, p = 0.006) and C groups (14.1 ± 4.54 × 105/mm3, p = 0.019). The LD+S group showed a trend towards a significant decrease in neuroapoptosis in the pyramidal layer (LD+S 7.51 ± 1.48 vs. LD 13.5 ± 1.87 vs. C 14.1 ± 4.54 × 105/mm3, p = 0.07) but not in the dentate gyrus. Fetal surgery did not affect neuroapoptosis in the frontal cortex or endplate.
CONCLUSIONS CONCLUSIONS
Fetal surgery decreases isoflurane-induced neuroapoptosis in the dentate gyrus and the pyramidal layer of mid-gestational fetal sheep. Long-term effects of these observations on memory and learning deserve further exploration.

Identifiants

pubmed: 30317244
pii: 000491925
doi: 10.1159/000491925
doi:

Substances chimiques

Isoflurane CYS9AKD70P
Caspase 3 EC 3.4.22.-

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

111-118

Informations de copyright

© 2018 S. Karger AG, Basel.

Auteurs

Olutoyin A Olutoye (OA)

Department of Anesthesiology, Baylor College of Medicine, Department of Anesthesiology, Peri-Operative and Pain Medicine, Texas Children's Fetal Center, Texas Children's Hospital, Houston, Texas, USA, oao@bcm.edu.

Stephanie M Cruz (SM)

Michael E. DeBakey Department of Surgery, Baylor College of Medicine, Texas Children's Fetal Center, Texas Children's Hospital, Houston, Texas, USA.

Adesola C Akinkuotu (AC)

Michael E. DeBakey Department of Surgery, Baylor College of Medicine, Texas Children's Fetal Center, Texas Children's Hospital, Houston, Texas, USA.

Fariha Sheikh (F)

Michael E. DeBakey Department of Surgery, Baylor College of Medicine, Texas Children's Fetal Center, Texas Children's Hospital, Houston, Texas, USA.

Irving J Zamora (IJ)

Michael E. DeBakey Department of Surgery, Baylor College of Medicine, Texas Children's Fetal Center, Texas Children's Hospital, Houston, Texas, USA.

Ling Yu (L)

Michael E. DeBakey Department of Surgery, Baylor College of Medicine, Texas Children's Fetal Center, Texas Children's Hospital, Houston, Texas, USA.

Adekunle M Adesina (AM)

Section of Pediatric Neuropathology, Department of Pathology, Baylor College of Medicine, Texas Children's Hospital, Houston, Texas, USA.

Oluyinka O Olutoye (OO)

Michael E. DeBakey Department of Surgery, Baylor College of Medicine, Texas Children's Fetal Center, Texas Children's Hospital, Houston, Texas, USA.

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Classifications MeSH