The prognostic and predictive impact of inflammatory biomarkers in patients who have advanced-stage cancer treated with immunotherapy.


Journal

Cancer
ISSN: 1097-0142
Titre abrégé: Cancer
Pays: United States
ID NLM: 0374236

Informations de publication

Date de publication:
01 01 2019
Historique:
received: 02 07 2018
revised: 15 08 2018
accepted: 20 08 2018
pubmed: 18 10 2018
medline: 12 10 2019
entrez: 18 10 2018
Statut: ppublish

Résumé

Optimal prognostic and predictive biomarkers for patients with advanced-stage cancer patients who received immunotherapy (IO) are lacking. Inflammatory markers, such as the neutrophil-to-lymphocyte ratio (NLR), the monocyte-to-lymphocyte ratio (MLR), and the platelet-to-lymphocyte ratio (PLR), are readily available. The authors investigated the association between these markers and clinical outcomes of patients with advanced-stage cancer who received IO. A retrospective review was conducted of 90 patients with advanced cancer who received treatment on phase 1 clinical trials of IO-based treatment regimens. NLR, MLR, and PLR values were log-transformed and treated as continuous variables for each patient. Overall survival (OS), progression-free survival (PFS), and clinical benefit were used to measure clinical outcomes. For univariate associations and multivariable analyses, Cox proportional-hazards models or logistic regression models were used. The median patient age was 63 years, and most were men (59%). The most common histologies were melanoma (33%) and gastrointestinal cancers (22%). High baseline NLR, MLR, and PLR values were associated significantly with worse OS and PFS (P < .05) and a lower chance of benefit (NLR and PLR; P < .05). Increased NLR, MLR, and PLR values 6 weeks after baseline were associated with shorter OS and PFS (P ≤ .052). Baseline and early changes in NLR, MLR, and PLR values were strongly associated with clinical outcomes in patients who received IO-based treatment regimens on phase 1 trials. Confirmation in a homogenous patient population treated on late-stage trials or outside of trial settings is warranted. These values may warrant consideration for inclusion when risk stratifying patients enrolled onto phase 1 clinical trials of IO agents.

Sections du résumé

BACKGROUND
Optimal prognostic and predictive biomarkers for patients with advanced-stage cancer patients who received immunotherapy (IO) are lacking. Inflammatory markers, such as the neutrophil-to-lymphocyte ratio (NLR), the monocyte-to-lymphocyte ratio (MLR), and the platelet-to-lymphocyte ratio (PLR), are readily available. The authors investigated the association between these markers and clinical outcomes of patients with advanced-stage cancer who received IO.
METHODS
A retrospective review was conducted of 90 patients with advanced cancer who received treatment on phase 1 clinical trials of IO-based treatment regimens. NLR, MLR, and PLR values were log-transformed and treated as continuous variables for each patient. Overall survival (OS), progression-free survival (PFS), and clinical benefit were used to measure clinical outcomes. For univariate associations and multivariable analyses, Cox proportional-hazards models or logistic regression models were used.
RESULTS
The median patient age was 63 years, and most were men (59%). The most common histologies were melanoma (33%) and gastrointestinal cancers (22%). High baseline NLR, MLR, and PLR values were associated significantly with worse OS and PFS (P < .05) and a lower chance of benefit (NLR and PLR; P < .05). Increased NLR, MLR, and PLR values 6 weeks after baseline were associated with shorter OS and PFS (P ≤ .052).
CONCLUSIONS
Baseline and early changes in NLR, MLR, and PLR values were strongly associated with clinical outcomes in patients who received IO-based treatment regimens on phase 1 trials. Confirmation in a homogenous patient population treated on late-stage trials or outside of trial settings is warranted. These values may warrant consideration for inclusion when risk stratifying patients enrolled onto phase 1 clinical trials of IO agents.

Identifiants

pubmed: 30329148
doi: 10.1002/cncr.31778
doi:

Substances chimiques

Biomarkers, Tumor 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

127-134

Commentaires et corrections

Type : CommentIn
Type : CommentIn

Informations de copyright

© 2018 American Cancer Society.

Auteurs

Mehmet A Bilen (MA)

Department of Hematology and Medical Oncology, Emory University School of Medicine, Atlanta, Georgia.
Winship Cancer Institute of Emory University, Atlanta, Georgia.

Dylan J Martini (DJ)

Department of Hematology and Medical Oncology, Emory University School of Medicine, Atlanta, Georgia.
Winship Cancer Institute of Emory University, Atlanta, Georgia.

Yuan Liu (Y)

Departments of Biostatistics and Bioinformatics, Emory University, Atlanta, Georgia.

Colleen Lewis (C)

Winship Cancer Institute of Emory University, Atlanta, Georgia.

Hannah H Collins (HH)

Winship Cancer Institute of Emory University, Atlanta, Georgia.

Julie M Shabto (JM)

Department of Hematology and Medical Oncology, Emory University School of Medicine, Atlanta, Georgia.
Winship Cancer Institute of Emory University, Atlanta, Georgia.

Mehmet Akce (M)

Department of Hematology and Medical Oncology, Emory University School of Medicine, Atlanta, Georgia.
Winship Cancer Institute of Emory University, Atlanta, Georgia.

Haydn T Kissick (HT)

Winship Cancer Institute of Emory University, Atlanta, Georgia.
Department of Urology, Emory University School of Medicine, Atlanta, Georgia.

Bradley C Carthon (BC)

Department of Hematology and Medical Oncology, Emory University School of Medicine, Atlanta, Georgia.
Winship Cancer Institute of Emory University, Atlanta, Georgia.

Walid L Shaib (WL)

Department of Hematology and Medical Oncology, Emory University School of Medicine, Atlanta, Georgia.
Winship Cancer Institute of Emory University, Atlanta, Georgia.

Olatunji B Alese (OB)

Department of Hematology and Medical Oncology, Emory University School of Medicine, Atlanta, Georgia.
Winship Cancer Institute of Emory University, Atlanta, Georgia.

Rathi N Pillai (RN)

Department of Hematology and Medical Oncology, Emory University School of Medicine, Atlanta, Georgia.
Winship Cancer Institute of Emory University, Atlanta, Georgia.

Conor E Steuer (CE)

Department of Hematology and Medical Oncology, Emory University School of Medicine, Atlanta, Georgia.
Winship Cancer Institute of Emory University, Atlanta, Georgia.

Christina S Wu (CS)

Department of Hematology and Medical Oncology, Emory University School of Medicine, Atlanta, Georgia.
Winship Cancer Institute of Emory University, Atlanta, Georgia.

David H Lawson (DH)

Department of Hematology and Medical Oncology, Emory University School of Medicine, Atlanta, Georgia.
Winship Cancer Institute of Emory University, Atlanta, Georgia.

Ragini R Kudchadkar (RR)

Department of Hematology and Medical Oncology, Emory University School of Medicine, Atlanta, Georgia.
Winship Cancer Institute of Emory University, Atlanta, Georgia.

Bassel F El-Rayes (BF)

Department of Hematology and Medical Oncology, Emory University School of Medicine, Atlanta, Georgia.
Winship Cancer Institute of Emory University, Atlanta, Georgia.

Viraj A Master (VA)

Department of Urology, Emory University School of Medicine, Atlanta, Georgia.

Suresh S Ramalingam (SS)

Department of Hematology and Medical Oncology, Emory University School of Medicine, Atlanta, Georgia.
Winship Cancer Institute of Emory University, Atlanta, Georgia.

Taofeek K Owonikoko (TK)

Department of Hematology and Medical Oncology, Emory University School of Medicine, Atlanta, Georgia.
Winship Cancer Institute of Emory University, Atlanta, Georgia.

R Donald Harvey (RD)

Department of Hematology and Medical Oncology, Emory University School of Medicine, Atlanta, Georgia.
Winship Cancer Institute of Emory University, Atlanta, Georgia.
Department of Pharmacology, Emory University School of Medicine, Atlanta, Georgia.

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