Mechanisms of Myocardial Infarction in Patients With Nonobstructive Coronary Artery Disease: Results From the Optical Coherence Tomography Study.


Journal

JACC. Cardiovascular imaging
ISSN: 1876-7591
Titre abrégé: JACC Cardiovasc Imaging
Pays: United States
ID NLM: 101467978

Informations de publication

Date de publication:
11 2019
Historique:
received: 11 03 2018
revised: 19 07 2018
accepted: 08 08 2018
pubmed: 22 10 2018
medline: 26 5 2020
entrez: 22 10 2018
Statut: ppublish

Résumé

This study sought to assess the presence and morphological features of coronary plaques on optical coherence tomography (OCT) as the causes of myocardial infarction with nonobstructive coronary arteries (MINOCA). Although coronary atherosclerosis has been postulated as a potential mechanism of MINOCA, the interaction between disrupted coronary plaques and myocardial injury remains unknown. In a prospective study, consecutive patients with MI but without significant coronary stenosis (≥50%) at angiography underwent OCT and cardiac magnetic resonance (CMR) with late gadolinium-enhancement (LGE). The infarct-related artery (IRA) was identified by localization of ischemic-type LGE. Thirty-eight MINOCA patients (mean age 62 ± 13 years, 55% female, 39% with ST-segment elevation) were enrolled. Maximal diameter stenosis was 35% by angiography, and 5 patients (13%) had normal angiogram results. Plaque disruption and coronary thrombus were observed in 9 patients (24%) and 7 patients (18%), respectively. Sixteen of 31 patients (52%) undergoing CMR showed LGE. Ischemic-type LGE was present in 7 patients (23%) and was more common in patients with than without plaque disruption (50% vs. 13%, respectively; p = 0.053) and coronary thrombus (67% vs. 12%, respectively; p = 0.014). In the per-lesion analysis, the IRA showed significantly more plaque disruption (40% vs. 6%; p = 0.02), thrombus (50% vs. 4%; p = 0.014), and thin-cap fibroatheroma (70% vs. 30%; p = 0.03) than the non-IRA. Plaque disruption and thrombus are not uncommon in MI without obstructive coronary stenoses at angiography and may be associated with the presence and location of ischemic-type myocardial injury on CMR. OCT may be valuable in identifying atherosclerotic etiology in individuals with MINOCA. (Optical Coherence Tomography in Patients With Acute Myocardial Infarction and Nonobstructive Coronary Artery Disease [SOFT-MI]; NCT02783963).

Sections du résumé

OBJECTIVES
This study sought to assess the presence and morphological features of coronary plaques on optical coherence tomography (OCT) as the causes of myocardial infarction with nonobstructive coronary arteries (MINOCA).
BACKGROUND
Although coronary atherosclerosis has been postulated as a potential mechanism of MINOCA, the interaction between disrupted coronary plaques and myocardial injury remains unknown.
METHODS
In a prospective study, consecutive patients with MI but without significant coronary stenosis (≥50%) at angiography underwent OCT and cardiac magnetic resonance (CMR) with late gadolinium-enhancement (LGE). The infarct-related artery (IRA) was identified by localization of ischemic-type LGE.
RESULTS
Thirty-eight MINOCA patients (mean age 62 ± 13 years, 55% female, 39% with ST-segment elevation) were enrolled. Maximal diameter stenosis was 35% by angiography, and 5 patients (13%) had normal angiogram results. Plaque disruption and coronary thrombus were observed in 9 patients (24%) and 7 patients (18%), respectively. Sixteen of 31 patients (52%) undergoing CMR showed LGE. Ischemic-type LGE was present in 7 patients (23%) and was more common in patients with than without plaque disruption (50% vs. 13%, respectively; p = 0.053) and coronary thrombus (67% vs. 12%, respectively; p = 0.014). In the per-lesion analysis, the IRA showed significantly more plaque disruption (40% vs. 6%; p = 0.02), thrombus (50% vs. 4%; p = 0.014), and thin-cap fibroatheroma (70% vs. 30%; p = 0.03) than the non-IRA.
CONCLUSIONS
Plaque disruption and thrombus are not uncommon in MI without obstructive coronary stenoses at angiography and may be associated with the presence and location of ischemic-type myocardial injury on CMR. OCT may be valuable in identifying atherosclerotic etiology in individuals with MINOCA. (Optical Coherence Tomography in Patients With Acute Myocardial Infarction and Nonobstructive Coronary Artery Disease [SOFT-MI]; NCT02783963).

Identifiants

pubmed: 30343070
pii: S1936-878X(18)30750-2
doi: 10.1016/j.jcmg.2018.08.022
pii:
doi:

Banques de données

ClinicalTrials.gov
['NCT02783963']

Types de publication

Comparative Study Journal Article Observational Study Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2210-2221

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2019 American College of Cardiology Foundation. Published by Elsevier Inc. All rights reserved.

Auteurs

Maksymilian P Opolski (MP)

Department of Interventional Cardiology and Angiology, Institute of Cardiology, Warsaw, Poland. Electronic address: opolski.mp@gmail.com.

Mateusz Spiewak (M)

Magnetic Resonance Unit, Department of Radiology, Institute of Cardiology, Warsaw, Poland.

Magdalena Marczak (M)

Magnetic Resonance Unit, Department of Radiology, Institute of Cardiology, Warsaw, Poland.

Artur Debski (A)

Department of Interventional Cardiology and Angiology, Institute of Cardiology, Warsaw, Poland.

Paul Knaapen (P)

Department of Cardiology, VU University Medical Center, Amsterdam, the Netherlands.

Stefan P Schumacher (SP)

Department of Cardiology, VU University Medical Center, Amsterdam, the Netherlands.

Adam D Staruch (AD)

Department of Interventional Cardiology and Angiology, Institute of Cardiology, Warsaw, Poland.

Kajetan Grodecki (K)

First Faculty of Medicine, Medical University of Warsaw, Warsaw, Poland.

Zbigniew Chmielak (Z)

Department of Interventional Cardiology and Angiology, Institute of Cardiology, Warsaw, Poland.

Hubert Lazarczyk (H)

Department of Information Technology, Institute of Cardiology, Warsaw, Poland.

Krzysztof Kukula (K)

Department of Interventional Cardiology and Angiology, Institute of Cardiology, Warsaw, Poland.

Pawel Tyczynski (P)

Department of Interventional Cardiology and Angiology, Institute of Cardiology, Warsaw, Poland.

Jerzy Pregowski (J)

Department of Interventional Cardiology and Angiology, Institute of Cardiology, Warsaw, Poland.

Maciej Dabrowski (M)

Department of Interventional Cardiology and Angiology, Institute of Cardiology, Warsaw, Poland.

Jacek Kadziela (J)

Department of Interventional Cardiology and Angiology, Institute of Cardiology, Warsaw, Poland.

Elzbieta Florczak (E)

Department of Hypertension, Institute of Cardiology, Warsaw, Poland.

Anna Skrobisz (A)

Department of Intensive Cardiac Therapy, Institute of Cardiology, Warsaw, Poland.

Adam Witkowski (A)

Department of Interventional Cardiology and Angiology, Institute of Cardiology, Warsaw, Poland.

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