Infection and Functional Modulation of Human Monocytes and Macrophages by Varicella-Zoster Virus.


Journal

Journal of virology
ISSN: 1098-5514
Titre abrégé: J Virol
Pays: United States
ID NLM: 0113724

Informations de publication

Date de publication:
01 02 2019
Historique:
received: 29 10 2018
accepted: 31 10 2018
pubmed: 9 11 2018
medline: 9 11 2019
entrez: 9 11 2018
Statut: epublish

Résumé

Varicella-zoster virus (VZV) is associated with viremia during primary infection that is presumed to stem from infection of circulating immune cells. While VZV has been shown to be capable of infecting a number of different subsets of circulating immune cells, such as T cells, dendritic cells, and NK cells, less is known about the interaction between VZV and monocytes. Here, we demonstrate that blood-derived human monocytes are permissive to VZV replication

Identifiants

pubmed: 30404793
pii: JVI.01887-18
doi: 10.1128/JVI.01887-18
pmc: PMC6340020
pii:
doi:

Substances chimiques

Antigens, Viral 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

Copyright © 2019 American Society for Microbiology.

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Auteurs

Jarrod J Kennedy (JJ)

Discipline of Infectious Diseases and Immunology, The University of Sydney, Sydney, New South Wales, Australia.

Megan Steain (M)

Discipline of Infectious Diseases and Immunology, The University of Sydney, Sydney, New South Wales, Australia.

Barry Slobedman (B)

Discipline of Infectious Diseases and Immunology, The University of Sydney, Sydney, New South Wales, Australia.

Allison Abendroth (A)

Discipline of Infectious Diseases and Immunology, The University of Sydney, Sydney, New South Wales, Australia allison.abendroth@sydney.edu.au.

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Classifications MeSH