Mitochondrial dysfunction and oxidative stress in induced pluripotent stem cell models of Parkinson's disease.


Journal

The European journal of neuroscience
ISSN: 1460-9568
Titre abrégé: Eur J Neurosci
Pays: France
ID NLM: 8918110

Informations de publication

Date de publication:
02 2019
Historique:
received: 03 07 2018
revised: 11 10 2018
accepted: 22 10 2018
pubmed: 9 11 2018
medline: 23 6 2020
entrez: 9 11 2018
Statut: ppublish

Résumé

Parkinson's disease (PD) is the second most common neurodegenerative disease. Two percent of the population above the age of 60 is affected by the disease. The pathological hallmarks of PD include loss of dopaminergic neurons and the presence of Lewy bodies. Mitochondrial dysfunction and oxidative stress are thought to play a pivotal role in both sporadic and familial forms of the disease. In this review we focus on the role of mitochondrial dysfunction and oxidative stress in induced pluripotent stem cell (IPSC) models of PD.We also provide an overview of therapeutics that have been tested and some possible new therapeutics that can be tested in IPSC models of PD.

Identifiants

pubmed: 30408242
doi: 10.1111/ejn.14264
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

525-532

Subventions

Organisme : The Cure Parkinson's Trust
Pays : International

Informations de copyright

© 2018 Federation of European Neuroscience Societies and John Wiley & Sons Ltd.

Auteurs

Anindita Bose (A)

Helen and Robert Appel Alzheimer's Disease Research Institute, Feil Family Brain and Mind Research Institute, Weill Cornell Medicine, New York, New York.

M Flint Beal (MF)

Feil Family Brain and Mind Research Institute, Weill Cornell Medicine, New York, New York.

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Classifications MeSH