Carotid Artery Stiffness and Incident Depressive Symptoms: The Paris Prospective Study III.


Journal

Biological psychiatry
ISSN: 1873-2402
Titre abrégé: Biol Psychiatry
Pays: United States
ID NLM: 0213264

Informations de publication

Date de publication:
15 03 2019
Historique:
received: 21 05 2018
revised: 27 08 2018
accepted: 11 09 2018
pubmed: 10 11 2018
medline: 6 2 2020
entrez: 10 11 2018
Statut: ppublish

Résumé

Arterial stiffness may contribute to late-life depression via cerebral microvascular damage, but evidence is scarce. No longitudinal study has evaluated the association between arterial stiffness and risk of depressive symptoms. Therefore, we investigated the association between carotid artery stiffness and incident depressive symptoms in a large community-based cohort study. This longitudinal study included 7013 participants (mean age 59.7 ± 6.3 years; 35.8% women) free of depressive symptoms at baseline. Carotid artery stiffness (high-resolution echo tracking) was determined at baseline. Presence of depressive symptoms was determined at baseline and at 4 and 6 years of follow-up, and was defined as a score ≥7 on the validated Questionnaire of Depression, Second Version, Abridged and/or new use of antidepressant medication. Logistic regression and generalized estimating equations were used. In total, 6.9% (n = 484) of the participants had incident depressive symptoms. Individuals in the lowest tertile of carotid distensibility coefficient (indicating greater carotid artery stiffness) compared with those in the highest tertile had a higher risk of incident depressive symptoms (odds ratio: 1.43; 95% confidence interval: 1.10-1.87), after adjustment for age, sex, living alone, education, lifestyle, cardiovascular risk factors, and baseline Questionnaire of Depression, Second Version, Abridged scores. Results were qualitatively similar when we used carotid Young's elastic modulus as a measure of carotid stiffness instead of carotid distensibility coefficient, and when we used generalized estimating equations instead of logistic regression. Greater carotid stiffness is associated with a higher incidence of depressive symptoms. This supports the hypothesis that carotid stiffness may contribute to the development of late-life depression.

Sections du résumé

BACKGROUND
Arterial stiffness may contribute to late-life depression via cerebral microvascular damage, but evidence is scarce. No longitudinal study has evaluated the association between arterial stiffness and risk of depressive symptoms. Therefore, we investigated the association between carotid artery stiffness and incident depressive symptoms in a large community-based cohort study.
METHODS
This longitudinal study included 7013 participants (mean age 59.7 ± 6.3 years; 35.8% women) free of depressive symptoms at baseline. Carotid artery stiffness (high-resolution echo tracking) was determined at baseline. Presence of depressive symptoms was determined at baseline and at 4 and 6 years of follow-up, and was defined as a score ≥7 on the validated Questionnaire of Depression, Second Version, Abridged and/or new use of antidepressant medication. Logistic regression and generalized estimating equations were used.
RESULTS
In total, 6.9% (n = 484) of the participants had incident depressive symptoms. Individuals in the lowest tertile of carotid distensibility coefficient (indicating greater carotid artery stiffness) compared with those in the highest tertile had a higher risk of incident depressive symptoms (odds ratio: 1.43; 95% confidence interval: 1.10-1.87), after adjustment for age, sex, living alone, education, lifestyle, cardiovascular risk factors, and baseline Questionnaire of Depression, Second Version, Abridged scores. Results were qualitatively similar when we used carotid Young's elastic modulus as a measure of carotid stiffness instead of carotid distensibility coefficient, and when we used generalized estimating equations instead of logistic regression.
CONCLUSIONS
Greater carotid stiffness is associated with a higher incidence of depressive symptoms. This supports the hypothesis that carotid stiffness may contribute to the development of late-life depression.

Identifiants

pubmed: 30409381
pii: S0006-3223(18)31889-4
doi: 10.1016/j.biopsych.2018.09.018
pii:
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

498-505

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2018 Society of Biological Psychiatry. Published by Elsevier Inc. All rights reserved.

Auteurs

Thomas T van Sloten (TT)

Faculté de Médecine, Sorbonne Paris Cité, Université Paris Descartes, Paris, France; Department of Epidemiology, Paris Cardiovascular Research Center, UMR-S970, Paris, France; Department of Arterial Mechanics, Paris Cardiovascular Research Center, UMR-S970, Paris, France; Cardiovascular Research Institute Maastricht and Department of Internal Medicine, Maastricht University Medical Centre, Maastricht, the Netherlands. Electronic address: t.van.sloten@mumc.nl.

Pierre Boutouyrie (P)

Faculté de Médecine, Sorbonne Paris Cité, Université Paris Descartes, Paris, France; Department of Arterial Mechanics, Paris Cardiovascular Research Center, UMR-S970, Paris, France; Department of Pharmacology, Georges Pompidou European Hospital, Public Assistance Hospitals of Paris, Paris, France.

Muriel Tafflet (M)

Faculté de Médecine, Sorbonne Paris Cité, Université Paris Descartes, Paris, France; Department of Epidemiology, Paris Cardiovascular Research Center, UMR-S970, Paris, France.

Lucile Offredo (L)

Faculté de Médecine, Sorbonne Paris Cité, Université Paris Descartes, Paris, France; Department of Epidemiology, Paris Cardiovascular Research Center, UMR-S970, Paris, France.

Frédérique Thomas (F)

Preventive and Clinical Investigation Center, Paris, France.

Catherine Guibout (C)

Faculté de Médecine, Sorbonne Paris Cité, Université Paris Descartes, Paris, France; Department of Epidemiology, Paris Cardiovascular Research Center, UMR-S970, Paris, France.

Rachel E Climie (RE)

Faculté de Médecine, Sorbonne Paris Cité, Université Paris Descartes, Paris, France; Department of Epidemiology, Paris Cardiovascular Research Center, UMR-S970, Paris, France; Department of Arterial Mechanics, Paris Cardiovascular Research Center, UMR-S970, Paris, France; Menzies Institute for Medical Research, University of Tasmania, Hobert, Australia.

Cédric Lemogne (C)

Faculté de Médecine, Sorbonne Paris Cité, Université Paris Descartes, Paris, France; Psychiatry and Neuroscience Center, U894, French Institute of Health and Medical Research, Paris, France; Department of Psychiatry, Georges Pompidou European Hospital, Public Assistance Hospitals of Paris, Paris, France.

Bruno Pannier (B)

Preventive and Clinical Investigation Center, Paris, France.

Stéphane Laurent (S)

Faculté de Médecine, Sorbonne Paris Cité, Université Paris Descartes, Paris, France; Department of Arterial Mechanics, Paris Cardiovascular Research Center, UMR-S970, Paris, France; Department of Pharmacology, Georges Pompidou European Hospital, Public Assistance Hospitals of Paris, Paris, France.

Xavier Jouven (X)

Faculté de Médecine, Sorbonne Paris Cité, Université Paris Descartes, Paris, France; Department of Epidemiology, Paris Cardiovascular Research Center, UMR-S970, Paris, France.

Jean-Philippe Empana (JP)

Faculté de Médecine, Sorbonne Paris Cité, Université Paris Descartes, Paris, France; Department of Epidemiology, Paris Cardiovascular Research Center, UMR-S970, Paris, France.

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