Spinal involvement with calcium pyrophosphate deposition disease in an academic rheumatology center: A series of 37 patients.


Journal

Seminars in arthritis and rheumatism
ISSN: 1532-866X
Titre abrégé: Semin Arthritis Rheum
Pays: United States
ID NLM: 1306053

Informations de publication

Date de publication:
06 2019
Historique:
received: 09 07 2018
revised: 17 09 2018
accepted: 09 10 2018
pubmed: 13 11 2018
medline: 21 4 2020
entrez: 13 11 2018
Statut: ppublish

Résumé

Calcium pyrophosphate dihydrate deposition disease (CPPD) has been reported to involve the spine, but few systematic studies have been published. To further characterize the spinal involvement with CPPD by a review of CPPD patients hospitalized in a rheumatology department. We retrospectively reviewed data for patients consecutively admitted with a diagnosis of CPPD in the rheumatology department of Lariboisière hospital in Paris, France over 5 years by using a standardized protocol and electronic case report forms. Imaging studies were also reviewed. Spinal CPPD was diagnosed in 37/152 (24.3%) CPPD patients. Patient with spinal involvement had more widespread peripheral CC. The cervical (n = 21) and lumbar (n = 19) segments were most involved. CT-scan was more sensitive than plain radiographs for detecting spinal calcifications. Crown dens syndrome was a prominent feature of cervical involvement. Inflammatory sterile spondylo-discitis was observed in 6 patients. Lesions were frequently multiple and were classified into 4 types. Ruling out septic discitis required image-guided biopsies in 3 patients. Sacroiliac involvement included calcification in 5 patients and severe sterile destructive arthropathy and joint fusion in one patient each. Degenerative changes were common, and CPPD could not be implicated because of the patients 'age and lack of a control population. In 12 patients, severe clinical features requiring hospitalization were related to such degenerative changes. Symptomatic involvement of the spine was observed in 24% of this series of hospitalized CPPD patients. Specific entities were the cause of hospitalisation in 25 of the 37 patients with spinal calcification and included inflammatory pain related to crystal deposits and destructive arthropathy of the spine and sacroiliac joints. Discitis exhibited a wide range of MRI features and biopsies were needed to rule out infection in 3 of the 6 discitis.

Sections du résumé

BACKGROUND
Calcium pyrophosphate dihydrate deposition disease (CPPD) has been reported to involve the spine, but few systematic studies have been published.
OBJECTIVE
To further characterize the spinal involvement with CPPD by a review of CPPD patients hospitalized in a rheumatology department.
METHODS
We retrospectively reviewed data for patients consecutively admitted with a diagnosis of CPPD in the rheumatology department of Lariboisière hospital in Paris, France over 5 years by using a standardized protocol and electronic case report forms. Imaging studies were also reviewed.
RESULTS
Spinal CPPD was diagnosed in 37/152 (24.3%) CPPD patients. Patient with spinal involvement had more widespread peripheral CC. The cervical (n = 21) and lumbar (n = 19) segments were most involved. CT-scan was more sensitive than plain radiographs for detecting spinal calcifications. Crown dens syndrome was a prominent feature of cervical involvement. Inflammatory sterile spondylo-discitis was observed in 6 patients. Lesions were frequently multiple and were classified into 4 types. Ruling out septic discitis required image-guided biopsies in 3 patients. Sacroiliac involvement included calcification in 5 patients and severe sterile destructive arthropathy and joint fusion in one patient each. Degenerative changes were common, and CPPD could not be implicated because of the patients 'age and lack of a control population. In 12 patients, severe clinical features requiring hospitalization were related to such degenerative changes.
CONCLUSION
Symptomatic involvement of the spine was observed in 24% of this series of hospitalized CPPD patients. Specific entities were the cause of hospitalisation in 25 of the 37 patients with spinal calcification and included inflammatory pain related to crystal deposits and destructive arthropathy of the spine and sacroiliac joints. Discitis exhibited a wide range of MRI features and biopsies were needed to rule out infection in 3 of the 6 discitis.

Identifiants

pubmed: 30415946
pii: S0049-0172(18)30437-2
doi: 10.1016/j.semarthrit.2018.10.009
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1113-1126

Informations de copyright

Copyright © 2018 Elsevier Inc. All rights reserved.

Auteurs

Abdelhafeez Moshrif (A)

Department of Rheumatology, Al Azhar University, Egypt.

Jean Denis Laredo (JD)

Department of Radiology, Hôpital Lariboisière, Assistance Publique des Hôpitaux de Paris, France; Université Paris Diderot France, France; UMR-CNRS 7052, Laboratoire de Recherches Orthopédiques B20A,Paris, France.

Hassan Bassiouni (H)

Department of Rheumatology, Al Azhar University, Egypt.

Mohamed Abdelkareem (M)

Department of Rheumatology, Al Azhar University, Egypt.

Pascal Richette (P)

Université Paris Diderot France, France; Department of Rheumatology, Hôpital Lariboisière, Assistance Publique des Hôpitaux de Paris, 75010 Paris, France; INSERM U1132, Hôpital Lariboisière, Université Paris Diderot, France.

Matthieu Reshe Rigon (MR)

Université Paris Diderot France, France; Department of Biostatistics and Information, Hôpital Saint Louis, Assistance Publique des Hôpitaux de Paris, France; INSERM U1153, Université Paris Diderot, France.

Thomas Bardin (T)

Université Paris Diderot France, France; Department of Rheumatology, Hôpital Lariboisière, Assistance Publique des Hôpitaux de Paris, 75010 Paris, France; INSERM U1132, Hôpital Lariboisière, Université Paris Diderot, France. Electronic address: thomas.bardin@aphp.fr.

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