Influence of the circle of Willis on leptomeningeal collateral flow in anterior circulation occlusive stroke: Friend or foe?


Journal

Journal of the neurological sciences
ISSN: 1878-5883
Titre abrégé: J Neurol Sci
Pays: Netherlands
ID NLM: 0375403

Informations de publication

Date de publication:
15 01 2019
Historique:
received: 10 07 2018
revised: 09 10 2018
accepted: 01 11 2018
pubmed: 13 11 2018
medline: 23 7 2019
entrez: 13 11 2018
Statut: ppublish

Résumé

Clinical outcome after large vessel occlusion (LVO) stroke depends on collateral integrity. We aimed to evaluate whether the completeness of the circle of Willis (CoW) and anterior temporal artery (ATA) determines the status of leptomeningeal collaterals (LC) in patients with acute LVO (internal carotid artery (ICA) and middle cerebral artery M1 (MCA) occlusion) treated with endovascular thrombectomy. LC, cross-flow through the anterior communicating artery (ACoA), presence of the ipsilateral posterior communicating artery (IpsiPCoA) and presence of the ATA were evaluated using CT angiography. LC was graded as good when ≥50% collateral filling was noted compared to the unaffected hemisphere. We included 159 patients with a median age of 75 years (IQR 63-82), MCA M1 occlusion in 96 (60%) and good outcome in 68 (45.6%). The LC were good in 129 (81.1%) patients. Complete IpsiPCoA and incomplete ACoA status was inversely associated with good LC in LVO (OR 0.51 (95% CI 0.02-0.07)). A complete CoW was associated with good LC in ICA occlusions, OR 8.4 (p = .025). Good outcome (modified Rankin scale 0-2 at 3 months) was associated with good LC (OR 5.63 (95% CI 1.11-28.4)), small ischemic lesion volume (OR 0.94 (95% CI 0.97-0.98)) and absence of the ACoA and IpsiPCoA (OR 4.47 (95% CI 1.09-18.3)). ATA presence was associated with good leptomeningeal collaterals in LVO (OR 8.13 (95% CI 1.69-39.0)) and in MCA M1 patients (OR 7.9 (95% CI 1.7-36.4)). The effect of ATA was most pronounced in MCA M1 occlusions, and that of ACoA was most pronounced in ICA occlusions.

Sections du résumé

BACKGROUND
Clinical outcome after large vessel occlusion (LVO) stroke depends on collateral integrity. We aimed to evaluate whether the completeness of the circle of Willis (CoW) and anterior temporal artery (ATA) determines the status of leptomeningeal collaterals (LC) in patients with acute LVO (internal carotid artery (ICA) and middle cerebral artery M1 (MCA) occlusion) treated with endovascular thrombectomy.
PATIENTS AND METHODS
LC, cross-flow through the anterior communicating artery (ACoA), presence of the ipsilateral posterior communicating artery (IpsiPCoA) and presence of the ATA were evaluated using CT angiography. LC was graded as good when ≥50% collateral filling was noted compared to the unaffected hemisphere.
RESULTS
We included 159 patients with a median age of 75 years (IQR 63-82), MCA M1 occlusion in 96 (60%) and good outcome in 68 (45.6%). The LC were good in 129 (81.1%) patients. Complete IpsiPCoA and incomplete ACoA status was inversely associated with good LC in LVO (OR 0.51 (95% CI 0.02-0.07)). A complete CoW was associated with good LC in ICA occlusions, OR 8.4 (p = .025). Good outcome (modified Rankin scale 0-2 at 3 months) was associated with good LC (OR 5.63 (95% CI 1.11-28.4)), small ischemic lesion volume (OR 0.94 (95% CI 0.97-0.98)) and absence of the ACoA and IpsiPCoA (OR 4.47 (95% CI 1.09-18.3)).
CONCLUSIONS
ATA presence was associated with good leptomeningeal collaterals in LVO (OR 8.13 (95% CI 1.69-39.0)) and in MCA M1 patients (OR 7.9 (95% CI 1.7-36.4)). The effect of ATA was most pronounced in MCA M1 occlusions, and that of ACoA was most pronounced in ICA occlusions.

Identifiants

pubmed: 30419369
pii: S0022-510X(18)30455-6
doi: 10.1016/j.jns.2018.11.002
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

69-75

Informations de copyright

Copyright © 2018 Elsevier B.V. All rights reserved.

Auteurs

Katharina Millesi (K)

Department of Neurology, Christian Doppler Medical Center, Paracelsus Medical University, Salzburg, Austria.

Johannes Sebastian Mutzenbach (JS)

Department of Neurology, Christian Doppler Medical Center, Paracelsus Medical University, Salzburg, Austria.

Monika Killer-Oberpfalzer (M)

Department of Neurology, Christian Doppler Medical Center, Paracelsus Medical University, Salzburg, Austria; Research Institute for Neurointervention, Christian Doppler Medical Center, Paracelsus Medical University, Salzburg, Austria.

Constantin Hecker (C)

Department of Neurology, Christian Doppler Medical Center, Paracelsus Medical University, Salzburg, Austria.

Lukas Machegger (L)

Division of neuroradiology, Christian Doppler Medical Center, Paracelsus Medical University, Salzburg, Austria.

Nele Bubel (N)

Department of Neurology, Christian Doppler Medical Center, Paracelsus Medical University, Salzburg, Austria.

Christian Ramesmayer (C)

Department of Neurology, Christian Doppler Medical Center, Paracelsus Medical University, Salzburg, Austria.

Slaven Pikija (S)

Department of Neurology, Christian Doppler Medical Center, Paracelsus Medical University, Salzburg, Austria. Electronic address: s.pikija@salk.at.

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