Characterization of the dynamic changes in left ventricular morphology and function induced by exercise training and detraining.


Journal

International journal of cardiology
ISSN: 1874-1754
Titre abrégé: Int J Cardiol
Pays: Netherlands
ID NLM: 8200291

Informations de publication

Date de publication:
15 Feb 2019
Historique:
received: 01 02 2018
revised: 06 07 2018
accepted: 26 10 2018
pubmed: 18 11 2018
medline: 4 9 2019
entrez: 17 11 2018
Statut: ppublish

Résumé

Although exercise-induced cardiac hypertrophy has been intensively investigated, its development and regression dynamics have not been comprehensively described. In the current study, we aimed to characterize the effects of regular exercise training and detraining on left ventricular (LV) morphology and function. Rats were divided into exercised (n = 12) and control (n = 12) groups. Exercised rats swam 200 min/day for 12 weeks. After completion of the training protocol, rats remained sedentary for 8 weeks (detraining period). Echocardiographic follow-up was performed regularly to obtain LV long- and short-axis recordings for speckle-tracking echocardiography analysis. Global longitudinal and circumferential strain and systolic strain rate were measured. LV pressure-volume analysis was performed using additional groups of rats to obtain haemodynamic data. Echocardiographic examinations showed the development of LV hypertrophy in the exercised group. These differences disappeared during the detraining period. Strain and strain rate values were all increased after the training period, whereas supernormal values rapidly reversed to the control level after training cessation. Load-independent haemodynamic indices, e.g., preload recruitable stroke work, confirmed the exercise-induced systolic improvement and complete regression after detraining. Our results provide the first comprehensive data to describe the development and regression dynamics of morphological and functional aspects of physiological hypertrophy in detail. Speckle-tracking echocardiography has been proven to be feasible to follow-up changes induced by exercise training and detraining and might provide an early possibility to differentiate between physiological and pathological conditions.

Sections du résumé

BACKGROUND BACKGROUND
Although exercise-induced cardiac hypertrophy has been intensively investigated, its development and regression dynamics have not been comprehensively described. In the current study, we aimed to characterize the effects of regular exercise training and detraining on left ventricular (LV) morphology and function.
METHODS METHODS
Rats were divided into exercised (n = 12) and control (n = 12) groups. Exercised rats swam 200 min/day for 12 weeks. After completion of the training protocol, rats remained sedentary for 8 weeks (detraining period). Echocardiographic follow-up was performed regularly to obtain LV long- and short-axis recordings for speckle-tracking echocardiography analysis. Global longitudinal and circumferential strain and systolic strain rate were measured. LV pressure-volume analysis was performed using additional groups of rats to obtain haemodynamic data.
RESULTS RESULTS
Echocardiographic examinations showed the development of LV hypertrophy in the exercised group. These differences disappeared during the detraining period. Strain and strain rate values were all increased after the training period, whereas supernormal values rapidly reversed to the control level after training cessation. Load-independent haemodynamic indices, e.g., preload recruitable stroke work, confirmed the exercise-induced systolic improvement and complete regression after detraining.
CONCLUSIONS AND TRANSLATIONAL ASPECT UNASSIGNED
Our results provide the first comprehensive data to describe the development and regression dynamics of morphological and functional aspects of physiological hypertrophy in detail. Speckle-tracking echocardiography has been proven to be feasible to follow-up changes induced by exercise training and detraining and might provide an early possibility to differentiate between physiological and pathological conditions.

Identifiants

pubmed: 30442376
pii: S0167-5273(18)30741-1
doi: 10.1016/j.ijcard.2018.10.092
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

178-185

Informations de copyright

Copyright © 2018 The Authors. Published by Elsevier B.V. All rights reserved.

Auteurs

Attila Oláh (A)

Heart and Vascular Center, Semmelweis University, Budapest, Hungary. Electronic address: o.attilio@gmail.com.

Attila Kovács (A)

Heart and Vascular Center, Semmelweis University, Budapest, Hungary.

Árpád Lux (Á)

Heart and Vascular Center, Semmelweis University, Budapest, Hungary.

Márton Tokodi (M)

Heart and Vascular Center, Semmelweis University, Budapest, Hungary.

Szilveszter Braun (S)

Heart and Vascular Center, Semmelweis University, Budapest, Hungary.

Bálint Károly Lakatos (BK)

Heart and Vascular Center, Semmelweis University, Budapest, Hungary.

Csaba Mátyás (C)

Heart and Vascular Center, Semmelweis University, Budapest, Hungary.

Dalma Kellermayer (D)

Heart and Vascular Center, Semmelweis University, Budapest, Hungary.

Mihály Ruppert (M)

Heart and Vascular Center, Semmelweis University, Budapest, Hungary.

Alex Ali Sayour (AA)

Heart and Vascular Center, Semmelweis University, Budapest, Hungary.

Bálint András Barta (BA)

Heart and Vascular Center, Semmelweis University, Budapest, Hungary.

Béla Merkely (B)

Heart and Vascular Center, Semmelweis University, Budapest, Hungary.

Tamás Radovits (T)

Heart and Vascular Center, Semmelweis University, Budapest, Hungary.

Articles similaires

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
1.00
Humans Yoga Low Back Pain Female Male
Humans Meals Time Factors Female Adult

Classifications MeSH