Echocardiographic diastolic function evolution in patients with an anterior Q-wave myocardial infarction: insights from the REVE-2 study.


Journal

ESC heart failure
ISSN: 2055-5822
Titre abrégé: ESC Heart Fail
Pays: England
ID NLM: 101669191

Informations de publication

Date de publication:
02 2019
Historique:
received: 17 01 2018
revised: 21 05 2018
accepted: 30 08 2018
pubmed: 22 11 2018
medline: 16 3 2019
entrez: 22 11 2018
Statut: ppublish

Résumé

Myocardial fibrosis plays a key role in the development of adverse left ventricular remodelling after myocardial infarction (MI). This study aimed to determine whether the circulating levels of BNP, collagen peptides, and galectin-3 are associated with diastolic function evolution (both deterioration and improvement) at 1 year after an anterior MI. The REVE-2 is a prospective multicentre study including 246 patients with a first anterior Q-wave MI. Echocardiographic assessment was performed at hospital discharge and ±1 year after MI. BNP, galectin-3, and collagen peptides were measured ±1 month after MI. Left ventricular diastolic dysfunction (DD) was defined according to the presence of at least two criteria of echocardiographic parameters: septal e' < 8 cm/s, lateral e' < 10 cm/s, and left atrial volume ≥ 34 mL/m The present study suggests that circulating levels of amino-terminal propeptide of type III procollagen, galectin-3, and BNP may be independently associated with new-onset DD in post-MI patients.

Identifiants

pubmed: 30460754
doi: 10.1002/ehf2.12359
pmc: PMC6351891
doi:

Substances chimiques

Blood Proteins 0
Galectin 3 0
Galectins 0
LGALS3 protein, human 0
Natriuretic Peptide, Brain 114471-18-0

Types de publication

Journal Article Multicenter Study Observational Study Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

70-79

Informations de copyright

© 2018 The Authors. ESC Heart Failure published by John Wiley & Sons Ltd on behalf of the European Society of Cardiology.

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Auteurs

João Pedro Ferreira (JP)

Centre d'Investigation Clinique CIC-P 1433, INSERM, Nancy, France.
F-CRIN INI-CRCT, Nancy, France.
Department of Physiology, University of Porto, Porto, Portugal.

Christophe Bauters (C)

CHU Lille, Lille, France.
INSERM U1167, Institut Pasteur de Lille, Université de Lille Nord de France, FHU-REMOD-VHF, Lille, France.
Faculté de Médecine de Lille, Lille, France.

Romain Eschalier (R)

Centre d'Investigation Clinique CIC-P 1433, INSERM, Nancy, France.
F-CRIN INI-CRCT, Nancy, France.
Department of Cardiology, Université Clermont Auvergne, TGI/ISIT/CaViti, Institut Pascal and CHU Clermont-Ferrand, Clermont-Ferrand, France.

Zohra Lamiral (Z)

Centre d'Investigation Clinique CIC-P 1433, INSERM, Nancy, France.

Renaud Fay (R)

Centre d'Investigation Clinique CIC-P 1433, INSERM, Nancy, France.

Olivier Huttin (O)

Centre d'Investigation Clinique CIC-P 1433, INSERM, Nancy, France.

Nicolas Girerd (N)

Centre d'Investigation Clinique CIC-P 1433, INSERM, Nancy, France.
F-CRIN INI-CRCT, Nancy, France.
Department of Cardiology, CHRU Nancy, Nancy, France.
Université de Lorraine, Nancy, France.

Faiez Zannad (F)

Centre d'Investigation Clinique CIC-P 1433, INSERM, Nancy, France.
F-CRIN INI-CRCT, Nancy, France.
Université de Lorraine, Nancy, France.
INSERM U1116, Nancy, France.

Florence Pinet (F)

F-CRIN INI-CRCT, Nancy, France.
CHU Lille, Lille, France.
INSERM U1167, Institut Pasteur de Lille, Université de Lille Nord de France, FHU-REMOD-VHF, Lille, France.

Patrick Rossignol (P)

Centre d'Investigation Clinique CIC-P 1433, INSERM, Nancy, France.
F-CRIN INI-CRCT, Nancy, France.
Université de Lorraine, Nancy, France.
INSERM U1116, Nancy, France.

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