Epicardial connection between the right-sided pulmonary venous carina and the right atrium in patients with atrial fibrillation: A possible mechanism for preclusion of pulmonary vein isolation without carina ablation.


Journal

Heart rhythm
ISSN: 1556-3871
Titre abrégé: Heart Rhythm
Pays: United States
ID NLM: 101200317

Informations de publication

Date de publication:
05 2019
Historique:
received: 20 09 2018
pubmed: 23 11 2018
medline: 3 10 2020
entrez: 23 11 2018
Statut: ppublish

Résumé

Ablation of the pulmonary venous carina is occasionally required for pulmonary vein isolation (PVI) despite its nonessential role in ipsilateral PVI from the anatomical (endocardial) viewpoint. Although the Bachmann bundle (BB) is a common and main interatrial band, local variations in small tongues of muscular fibers were frequently found in autopsy studies. We sought to clarify the effect of the electrical conduction pattern from the right atrium (RA) to the left atrium (LA) during sinus rhythm on the necessity of performing right-sided pulmonary venous carina ablation to achieve PVI. Study subjects comprised 37 consecutive patients undergoing initial catheter ablation of lone atrial fibrillation. During sinus rhythm, RA and LA activation maps were acquired using an electroanatomical mapping system. LA breakthroughs were classified into 3 sites: BB, fossa ovalis (FO), and right-sided pulmonary venous carina. Patients were divided into the carina-ABL (ablation) or non-carina-ABL group on the basis of the necessity of pulmonary venous carina ablation to achieve PVI. Patients were classified in the non-carina-ABL group (n = 26 [70%]) and carina-ABL group (n = 8 [22%]) after excluding 3 patients (8%) because of their complex ablation lesion sets. Breakthrough occurred in the BB (n = 21 patients [62%]), FO (n = 7 [21%]), carina (n = 1 [3%]), carina and BB (n = 3 [9%]), and carina and FO (n = 2 [6%]). Carina breakthrough occurred in 6 patients (75%) in the carina-ABL group but in no patients in the non-carina-ABL group (P < .0001). PVI was not achievable without carina ablation in one-fifth of patients, probably because of epicardial connections present between the right-sided pulmonary venous carina and the RA.

Sections du résumé

BACKGROUND
Ablation of the pulmonary venous carina is occasionally required for pulmonary vein isolation (PVI) despite its nonessential role in ipsilateral PVI from the anatomical (endocardial) viewpoint. Although the Bachmann bundle (BB) is a common and main interatrial band, local variations in small tongues of muscular fibers were frequently found in autopsy studies.
OBJECTIVE
We sought to clarify the effect of the electrical conduction pattern from the right atrium (RA) to the left atrium (LA) during sinus rhythm on the necessity of performing right-sided pulmonary venous carina ablation to achieve PVI.
METHODS
Study subjects comprised 37 consecutive patients undergoing initial catheter ablation of lone atrial fibrillation. During sinus rhythm, RA and LA activation maps were acquired using an electroanatomical mapping system. LA breakthroughs were classified into 3 sites: BB, fossa ovalis (FO), and right-sided pulmonary venous carina. Patients were divided into the carina-ABL (ablation) or non-carina-ABL group on the basis of the necessity of pulmonary venous carina ablation to achieve PVI.
RESULTS
Patients were classified in the non-carina-ABL group (n = 26 [70%]) and carina-ABL group (n = 8 [22%]) after excluding 3 patients (8%) because of their complex ablation lesion sets. Breakthrough occurred in the BB (n = 21 patients [62%]), FO (n = 7 [21%]), carina (n = 1 [3%]), carina and BB (n = 3 [9%]), and carina and FO (n = 2 [6%]). Carina breakthrough occurred in 6 patients (75%) in the carina-ABL group but in no patients in the non-carina-ABL group (P < .0001).
CONCLUSION
PVI was not achievable without carina ablation in one-fifth of patients, probably because of epicardial connections present between the right-sided pulmonary venous carina and the RA.

Identifiants

pubmed: 30465905
pii: S1547-5271(18)31162-7
doi: 10.1016/j.hrthm.2018.11.017
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

671-678

Informations de copyright

Copyright © 2018 Heart Rhythm Society. Published by Elsevier Inc. All rights reserved.

Auteurs

Kentaro Yoshida (K)

Department of Cardiology, Ibaraki Prefectural Central Hospital, Kasama, Japan; Department of Cardiology, Faculty of Medicine, University of Tsukuba, Tsukuba, Japan. Electronic address: kentaroyo@nifty.com.

Masako Baba (M)

Department of Cardiology, Ibaraki Prefectural Central Hospital, Kasama, Japan.

Yasutoshi Shinoda (Y)

Department of Cardiology, Ibaraki Prefectural Central Hospital, Kasama, Japan; Department of Cardiology, Faculty of Medicine, University of Tsukuba, Tsukuba, Japan.

Tomohiko Harunari (T)

Department of Cardiology, Ibaraki Prefectural Central Hospital, Kasama, Japan; Department of Cardiology, Faculty of Medicine, University of Tsukuba, Tsukuba, Japan.

Yasuaki Tsumagari (Y)

Department of Cardiology, Faculty of Medicine, University of Tsukuba, Tsukuba, Japan.

Naoya Koda (N)

Department of Cardiology, Ibaraki Prefectural Central Hospital, Kasama, Japan.

Kosuke Hayashi (K)

Department of Cardiology, Ibaraki Prefectural Central Hospital, Kasama, Japan.

Takumi Yaguchi (T)

Department of Cardiology, Ibaraki Prefectural Central Hospital, Kasama, Japan.

Hiroaki Watabe (H)

Department of Cardiology, Ibaraki Prefectural Central Hospital, Kasama, Japan.

Hideyuki Hasebe (H)

Department of Cardiology, Faculty of Medicine, University of Tsukuba, Tsukuba, Japan.

Kazutaka Aonuma (K)

Department of Cardiology, Faculty of Medicine, University of Tsukuba, Tsukuba, Japan.

Noriyuki Takeyasu (N)

Department of Cardiology, Ibaraki Prefectural Central Hospital, Kasama, Japan.

Akihiko Nogami (A)

Department of Cardiology, Faculty of Medicine, University of Tsukuba, Tsukuba, Japan.

Masaki Ieda (M)

Department of Cardiology, Faculty of Medicine, University of Tsukuba, Tsukuba, Japan.

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