CXCR6 protects from inflammation and fibrosis in NEMO
Adoptive Transfer
Animals
Apoptosis
Cells, Cultured
Cytochrome P-450 CYP4A
/ metabolism
Fatty Liver
/ metabolism
Hepatocytes
/ metabolism
Inflammation
/ metabolism
Intracellular Signaling Peptides and Proteins
/ deficiency
Liver
/ injuries
Male
Mice, Knockout
Receptors, CXCR6
/ deficiency
Stress, Physiological
Tumor Necrosis Factor-alpha
/ adverse effects
Up-Regulation
Chemokine receptor
Hepatocytes
Inflammation
Liver fibrosis
Journal
Biochimica et biophysica acta. Molecular basis of disease
ISSN: 1879-260X
Titre abrégé: Biochim Biophys Acta Mol Basis Dis
Pays: Netherlands
ID NLM: 101731730
Informations de publication
Date de publication:
01 02 2019
01 02 2019
Historique:
received:
03
07
2018
revised:
16
10
2018
accepted:
22
11
2018
pubmed:
27
11
2018
medline:
19
9
2019
entrez:
27
11
2018
Statut:
ppublish
Résumé
Chronic inflammation in the liver provokes fibrosis and, on long-term, carcinogenesis. This sequence is prototypically recapitulated in mice with hepatocyte-specific knock-out of the NF-κB essential modulator (NEMO), termed NEMO
Identifiants
pubmed: 30476545
pii: S0925-4439(18)30482-4
doi: 10.1016/j.bbadis.2018.11.020
pii:
doi:
Substances chimiques
Intracellular Signaling Peptides and Proteins
0
NEMO protein, mouse
0
Receptors, CXCR6
0
Tumor Necrosis Factor-alpha
0
Cytochrome P-450 CYP4A
EC 1.14.15.3
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
391-402Informations de copyright
Copyright © 2018 Elsevier B.V. All rights reserved.