Distinct mechanisms govern the phosphorylation of different SR protein splicing factors.

RNA splicing SR protein SRPK SRSF kinase-substrate interaction localization nuclear speckle phosphoryl transfer post-translational mechanism post-translational modification (PTM) processive phosphorylation protein kinase protein phosphorylation regulatory mechanism serine/threonine protein kinase speckles subcellular localization

Journal

The Journal of biological chemistry
ISSN: 1083-351X
Titre abrégé: J Biol Chem
Pays: United States
ID NLM: 2985121R

Informations de publication

Date de publication:
25 01 2019
Historique:
received: 10 04 2018
revised: 17 11 2018
pubmed: 28 11 2018
medline: 9 5 2019
entrez: 28 11 2018
Statut: ppublish

Résumé

Serine-arginine (SR) proteins are essential splicing factors containing a canonical RNA recognition motif (RRM), sometimes followed by a pseudo-RRM, and a C-terminal arginine/serine-rich (RS) domain that undergoes multisite phosphorylation. Phosphorylation regulates the localization and activity of SR proteins, and thus may provide insight into their differential biological roles. The phosphorylation mechanism of the prototypic SRSF1 by serine-arginine protein kinase 1 (SRPK1) has been well-studied, but little is known about the phosphorylation of other SR protein members. In the present study, interaction and kinetic assays unveiled how SRSF1 and the single RRM-containing SRSF3 are phosphorylated by SRPK2, another member of the SRPK family. We showed that a conserved SRPK-specific substrate-docking groove in SRPK2 impacts the binding and phosphorylation of both SR proteins, and the localization of SRSF3. We identified a nonconserved residue within the groove that affects the kinase processivity. We demonstrated that, in contrast to SRSF1, for which SRPK-mediated phosphorylation is confined to the N-terminal region of the RS domain, SRSF3 phosphorylation sites are spread throughout its entire RS domain

Identifiants

pubmed: 30478176
pii: S0021-9258(20)38868-2
doi: 10.1074/jbc.RA118.003392
pmc: PMC6349114
pii:
doi:

Substances chimiques

SRSF1 protein, human 0
SRSF3 protein, human 0
Serine-Arginine Splicing Factors 170974-22-8
Protein Serine-Threonine Kinases EC 2.7.11.1
SRPK2 protein, human EC 2.7.11.1

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1312-1327

Informations de copyright

© 2019 Long et al.

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Auteurs

Yunxin Long (Y)

School of Life Sciences, The Chinese University of Hong Kong, Shatin, N.T., Hong Kong, China.

Weng Hong Sou (WH)

School of Life Sciences, The Chinese University of Hong Kong, Shatin, N.T., Hong Kong, China.

Kristen Wing Yu Yung (KWY)

School of Life Sciences, The Chinese University of Hong Kong, Shatin, N.T., Hong Kong, China.

Haizhen Liu (H)

School of Life Sciences, The Chinese University of Hong Kong, Shatin, N.T., Hong Kong, China.

Stephanie Winn Chee Wan (SWC)

School of Life Sciences, The Chinese University of Hong Kong, Shatin, N.T., Hong Kong, China.

Qingyun Li (Q)

School of Life Sciences, The Chinese University of Hong Kong, Shatin, N.T., Hong Kong, China.

Chuyue Zeng (C)

School of Life Sciences, The Chinese University of Hong Kong, Shatin, N.T., Hong Kong, China.

Carmen Oi Kwan Law (COK)

Department of Biomedical Sciences, City University of Hong Kong, Kowloon, Hong Kong, China.

Gordon Ho Ching Chan (GHC)

School of Life Sciences, The Chinese University of Hong Kong, Shatin, N.T., Hong Kong, China.

Terrence Chi Kong Lau (TCK)

Department of Biomedical Sciences, City University of Hong Kong, Kowloon, Hong Kong, China.

Jacky Chi Ki Ngo (JCK)

School of Life Sciences, The Chinese University of Hong Kong, Shatin, N.T., Hong Kong, China. Electronic address: jackyngo@cuhk.edu.hk.

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Classifications MeSH