Role of autonomic nervous system in atrial fibrillation.


Journal

International journal of cardiology
ISSN: 1874-1754
Titre abrégé: Int J Cardiol
Pays: Netherlands
ID NLM: 8200291

Informations de publication

Date de publication:
15 07 2019
Historique:
received: 12 07 2018
revised: 01 11 2018
accepted: 14 11 2018
pubmed: 1 12 2018
medline: 17 3 2020
entrez: 1 12 2018
Statut: ppublish

Résumé

Atrial fibrillation is the most common sustained arrhythmia and is associated with significant morbidity and mortality. The autonomic nervous system has a significant role in the milieu predisposing to the triggers, perpetuators and substrate for atrial fibrillation. It has direct electrophysiological effects and causes alterations in atrial structure. In a significant portion of patients with atrial fibrillation, the autonomic nervous system activity is likely a composite of reflex excitation due to atrial fibrillation itself and contribution of concomitant risk factors such as hypertension, obesity and sleep-disordered breathing. We review the role of autonomic nervous system activation, with focus on changes in reflex control during atrial fibrillation and the role of combined sympatho-vagal activation for atrial fibrillation initiation, maintenance and progression. Finally, we discuss the potential impact of combined aggressive risk factor management as a strategy to modify the autonomic nervous system in patients with atrial fibrillation and to reverse the arrhythmogenic substrate.

Identifiants

pubmed: 30497894
pii: S0167-5273(18)31862-X
doi: 10.1016/j.ijcard.2018.11.091
pii:
doi:

Types de publication

Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

181-188

Informations de copyright

Copyright © 2018 Elsevier B.V. All rights reserved.

Auteurs

Dominik Linz (D)

Centre for Heart Rhythm Disorders (CHRD), South Australian Health and Medical Research Institute (SAHMRI), University of Adelaide and Royal Adelaide Hospital, Adelaide, Australia. Electronic address: Dominik.Linz@adelaide.edu.au.

Adrian D Elliott (AD)

Centre for Heart Rhythm Disorders (CHRD), South Australian Health and Medical Research Institute (SAHMRI), University of Adelaide and Royal Adelaide Hospital, Adelaide, Australia.

Mathias Hohl (M)

Saarland University, Klinik für Innere Medizin III (Cardiology, Angiology and Intensive Care Medicine), Homburg, Saar, Germany.

Varun Malik (V)

Centre for Heart Rhythm Disorders (CHRD), South Australian Health and Medical Research Institute (SAHMRI), University of Adelaide and Royal Adelaide Hospital, Adelaide, Australia.

Ulrich Schotten (U)

University Maastricht, Dept. of Physiology, Maastricht, the Netherlands.

Dobromir Dobrev (D)

Institute of Pharmacology, West German Heart and Vascular Center, University Duisburg-Essen, Essen, Germany.

Stanley Nattel (S)

Institute of Pharmacology, West German Heart and Vascular Center, University Duisburg-Essen, Essen, Germany; Department of Medicine, Montreal Heart Institute, Université de Montréal, Montréal, Quebec, Canada; Department of Pharmacology and Therapeutics, McGill University, Montreal, Quebec, Canada.

Michael Böhm (M)

Saarland University, Klinik für Innere Medizin III (Cardiology, Angiology and Intensive Care Medicine), Homburg, Saar, Germany.

John Floras (J)

University Health Network and Sinai Health System Division of Cardiology, University of Toronto, Toronto, Ontario, Canada.

Dennis H Lau (DH)

Centre for Heart Rhythm Disorders (CHRD), South Australian Health and Medical Research Institute (SAHMRI), University of Adelaide and Royal Adelaide Hospital, Adelaide, Australia.

Prashanthan Sanders (P)

Centre for Heart Rhythm Disorders (CHRD), South Australian Health and Medical Research Institute (SAHMRI), University of Adelaide and Royal Adelaide Hospital, Adelaide, Australia.

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Classifications MeSH