Traction bronchiectasis on high-resolution computed tomography may predict fatal acute eosinophilic pneumonia.


Journal

Respiratory investigation
ISSN: 2212-5353
Titre abrégé: Respir Investig
Pays: Netherlands
ID NLM: 101581124

Informations de publication

Date de publication:
Jan 2019
Historique:
received: 17 06 2018
revised: 12 09 2018
accepted: 18 09 2018
pubmed: 12 12 2018
medline: 21 3 2019
entrez: 12 12 2018
Statut: ppublish

Résumé

Most patients with acute eosinophilic pneumonia (AEP) show rapid improvement. However, some cases of AEP prove fatal. The aims of this study were to determine the clinical, radiographic, and pathologic characteristics of patients in whom AEP has a fatal outcome and to identify predictors of a poor prognosis. We retrospectively identified the medical records of all patients diagnosed with AEP at our institution in Japan from July 2005 to July 2013. There were four deaths among 41 patients diagnosed to have AEP during the study period. All the patients who died were male; three cases were idiopathic and one was medication-related. The median bronchoalveolar lavage eosinophil differential count was 59%. An autopsy was performed on the patient with medication-related AEP who died and the pathologic finding was diffuse alveolar damage with eosinophilic infiltration. Diffuse ground-glass attenuation and traction bronchiectasis (TBE) were identified on high-resolution computed tomography in the four patients with fatal AEP. TBE was observed in six patients (five with idiopathic AEP, one with medication-related AEP), and 67% of these patients died. None of the patients with smoking-related AEP had TBE; all these patients had better responses to treatment and survived. We observed the characteristics of patients with fatal AEP who did not respond to treatment. TBE was observed in all fatal cases and may be associated with a poor prognosis.

Sections du résumé

BACKGROUND BACKGROUND
Most patients with acute eosinophilic pneumonia (AEP) show rapid improvement. However, some cases of AEP prove fatal. The aims of this study were to determine the clinical, radiographic, and pathologic characteristics of patients in whom AEP has a fatal outcome and to identify predictors of a poor prognosis.
METHODS METHODS
We retrospectively identified the medical records of all patients diagnosed with AEP at our institution in Japan from July 2005 to July 2013.
RESULTS RESULTS
There were four deaths among 41 patients diagnosed to have AEP during the study period. All the patients who died were male; three cases were idiopathic and one was medication-related. The median bronchoalveolar lavage eosinophil differential count was 59%. An autopsy was performed on the patient with medication-related AEP who died and the pathologic finding was diffuse alveolar damage with eosinophilic infiltration. Diffuse ground-glass attenuation and traction bronchiectasis (TBE) were identified on high-resolution computed tomography in the four patients with fatal AEP. TBE was observed in six patients (five with idiopathic AEP, one with medication-related AEP), and 67% of these patients died. None of the patients with smoking-related AEP had TBE; all these patients had better responses to treatment and survived.
CONCLUSIONS CONCLUSIONS
We observed the characteristics of patients with fatal AEP who did not respond to treatment. TBE was observed in all fatal cases and may be associated with a poor prognosis.

Identifiants

pubmed: 30528687
pii: S2212-5345(18)30116-3
doi: 10.1016/j.resinv.2018.09.005
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

67-72

Informations de copyright

Copyright © 2018 The Japanese Respiratory Society. Published by Elsevier B.V. All rights reserved.

Auteurs

Reoto Takei (R)

Department of Respiratory Medicine, Kurashiki Central Hospital, 1-1-1 Miwa, Kurashiki, Okayama 710-8602, Japan. Electronic address: rtakei415@gmail.com.

Machiko Arita (M)

Department of Respiratory Medicine, Kurashiki Central Hospital, 1-1-1 Miwa, Kurashiki, Okayama 710-8602, Japan.

Shogo Kumagai (S)

Division of Cancer Immunology, Exploratory Oncology Research and Clinical Trial Center, National Cancer Center, Kashiwa, Japan.

Yuhei Ito (Y)

Department of Respiratory Medicine, Ise Red Cross Hospital, Ise, Japan.

Takuya Takaiwa (T)

Department of Respiratory Medicine, Sakai City Medical Center, Sakai, Japan.

Fumiaki Tokioka (F)

Department of Respiratory Medicine, Kurashiki Central Hospital, 1-1-1 Miwa, Kurashiki, Okayama 710-8602, Japan.

Junya Itakura (J)

Department of Anatomic Pathology, Kurashiki Central Hospital, Kurashiki, Japan.

Tadashi Ishida (T)

Department of Respiratory Medicine, Kurashiki Central Hospital, 1-1-1 Miwa, Kurashiki, Okayama 710-8602, Japan.

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