Molecular targeting for treatment of human T-lymphotropic virus type 1 infection.


Journal

Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie
ISSN: 1950-6007
Titre abrégé: Biomed Pharmacother
Pays: France
ID NLM: 8213295

Informations de publication

Date de publication:
Jan 2019
Historique:
received: 13 08 2018
revised: 22 10 2018
accepted: 24 10 2018
entrez: 16 12 2018
pubmed: 16 12 2018
medline: 2 4 2019
Statut: ppublish

Résumé

Human T-cell lymphotropic virus type 1 (HTLV-1) infection is linked to adult T-cell leukemia-lymphoma (ATLL) and HTLV-I-associated myelopathy/tropical spastic paraparesis (HAM/TSP) and several other disorders. ATLL occurs in approximately 5% of the 15-20 million people infected by HTLV-1 in the world. In general, ATLL is resistant to chemotherapy, which underlines the need for new and effective therapeutic strategies. Previous studies highlighted the role of viral enzymes, responsible for viral replication, and regulatory proteins such as Tax and HBZ in the progression of HTLV-1-associated diseases. There are conflicting reports on the efficacy of current enzyme inhibitors, mainly developed against human immunodeficiency virus (HIV), for treatment of HTLV-1 infection. New treatment approaches including monoclonal antibodies show promising results and exert significant cytotoxic effects on ATLL cells. This manuscript reviews the recent developments in molecular targeting for treatment of HTLV-1 infection.

Identifiants

pubmed: 30551530
pii: S0753-3322(18)35481-7
doi: 10.1016/j.biopha.2018.10.139
pii:
doi:

Substances chimiques

Antibodies, Monoclonal 0
Enzyme Inhibitors 0

Types de publication

Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

770-778

Informations de copyright

Copyright © 2018 Elsevier Masson SAS. All rights reserved.

Auteurs

Arash Soltani (A)

Department of Clinical Biochemistry, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran; Student Research Committee, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Seyed Isaac Hashemy (SI)

Surgical Oncology Research Center, Mashhad University of Medical Sciences, Mashhad, Iran.

Farnaz Zahedi Avval (F)

Department of Clinical Biochemistry, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Anvar Soleimani (A)

Department of Clinical Biochemistry, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Houshang Rafatpanah (H)

Immunology Research Center, Division of Inflammation and Inflammatory Diseases, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Seyed Abdorahim Rezaee (SA)

Immunology Research Center, Division of Inflammation and Inflammatory Diseases, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Renate Griffith (R)

School of Medical Sciences/Pharmacology, UNSW Sydney, Kensington, NSW 2052, Australia. Electronic address: r.griffith@unsw.edu.au.

Baratali Mashkani (B)

Department of Clinical Biochemistry, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran; Surgical Oncology Research Center, Mashhad University of Medical Sciences, Mashhad, Iran. Electronic address: mashkaniba@mums.ac.ir.

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Classifications MeSH