Silencing of lncRNA LINC00514 inhibits the malignant behaviors of papillary thyroid cancer through miR-204-3p/CDC23 axis.


Journal

Biochemical and biophysical research communications
ISSN: 1090-2104
Titre abrégé: Biochem Biophys Res Commun
Pays: United States
ID NLM: 0372516

Informations de publication

Date de publication:
22 01 2019
Historique:
received: 26 11 2018
accepted: 06 12 2018
pubmed: 17 12 2018
medline: 11 9 2019
entrez: 17 12 2018
Statut: ppublish

Résumé

Numerous studies have provided that long noncoding RNAs (lncRNAs) possess important roles in regulating tumorigenesis. However, up to data, the role of LINC00514 in cancer, including thyroid cancer, remains unknown. In the present study, we found that LINC00514 expression was significantly upregulated in papillary thyroid cancer (PTC) tissues by bioinformatics analysis. Loss-of-function studies revealed that LINC00514 silencing inhibited the proliferation, migration and invasion of PTC cells while promoting apoptosis in vitro. Moreover, LINC00514 knockdown suppressed PTC growth in vivo. RNA-FISH showed that LINC00514 mainly locates in the nucleus of PTC cells. Through bioinformatics prediction, we identified that LINC00514 served as the sponge for miR-204-3p, and miR-204-3p directly targeted CDC23. Thus, LINC00514 promoted CDC23 expression via restraining miR-204-3p activity, leading to PTC progression. In sum, our findings demonstrated that LINC00514 contributes to PTC progression and might be a potential target for PTC therapy.

Identifiants

pubmed: 30553447
pii: S0006-291X(18)32694-9
doi: 10.1016/j.bbrc.2018.12.051
pii:
doi:

Substances chimiques

Apc8 Subunit, Anaphase-Promoting Complex-Cyclosome 0
CDC23 protein, human 0
MIRN204 microRNA, human 0
MicroRNAs 0
RNA, Long Noncoding 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1145-1148

Informations de copyright

Copyright © 2018 Elsevier Inc. All rights reserved.

Auteurs

Xiangya Li (X)

Department of Endocrinology, Xiangya Hospital of Central South University, Changsha, 410005, China.

Wen Zhong (W)

Department of Endocrinology, Xiangya Hospital of Central South University, Changsha, 410005, China.

Yiling Xu (Y)

Department of Endocrinology, Xiangya Hospital of Central South University, Changsha, 410005, China.

Bo Yu (B)

Department of Endocrinology, Xiangya Hospital of Central South University, Changsha, 410005, China.

Huixia Liu (H)

Department of Endocrinology, Xiangya Hospital of Central South University, Changsha, 410005, China. Electronic address: huixialiu1@163.com.

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Classifications MeSH