β-1 adrenoceptors and AT1 receptors may not be involved in catecholamine-induced lethal arrhythmias
Animals
Arrhythmias, Cardiac
/ chemically induced
Catecholamines
/ pharmacology
Dose-Response Relationship, Drug
Electrocardiography
/ drug effects
Male
Rats
Rats, Sprague-Dawley
Receptor, Angiotensin, Type 1
/ metabolism
Receptors, Adrenergic, beta-1
/ metabolism
Sympathetic Nervous System
/ drug effects
AT1 receptors
arythmies provoquées par les catécholamines
atenolol
blocage des récepteurs
catecholamine-induced arrhythmias
catécholamines plasmatiques
losartan
plasma catecholamines
récepteurs AT1
récepteurs β adrénergiques
β-adrenoceptors
Journal
Canadian journal of physiology and pharmacology
ISSN: 1205-7541
Titre abrégé: Can J Physiol Pharmacol
Pays: Canada
ID NLM: 0372712
Informations de publication
Date de publication:
Jun 2019
Jun 2019
Historique:
pubmed:
18
12
2018
medline:
5
11
2019
entrez:
18
12
2018
Statut:
ppublish
Résumé
An excessive amount of catecholamines produce arrhythmias, but the exact mechanisms of this action are not fully understood. For this purpose, Sprague-Dawley rats were treated with or without atenolol, a β
Identifiants
pubmed: 30557041
doi: 10.1139/cjpp-2018-0531
doi:
Substances chimiques
Catecholamines
0
Receptor, Angiotensin, Type 1
0
Receptors, Adrenergic, beta-1
0
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM