Maternal HPA-1a antibody level and its role in predicting the severity of Fetal/Neonatal Alloimmune Thrombocytopenia: a systematic review.


Journal

Vox sanguinis
ISSN: 1423-0410
Titre abrégé: Vox Sang
Pays: England
ID NLM: 0413606

Informations de publication

Date de publication:
Jan 2019
Historique:
received: 19 04 2018
revised: 05 09 2018
accepted: 19 10 2018
pubmed: 20 12 2018
medline: 22 3 2019
entrez: 20 12 2018
Statut: ppublish

Résumé

In Caucasians, fetal/neonatal alloimmune thrombocytopenia (FNAIT) is most commonly due to maternal HPA-1a antibodies. HPA-1a typing followed by screening for anti-HPA-1a antibodies in HPA-1bb women may identify first pregnancies at risk. Our goal was to review results from previous published studies to examine whether the maternal antibody level to HPA-1a could be used to identify high-risk pregnancies. The studies included were categorized by recruitment strategies: screening of unselected pregnancies or samples analyzed from known or suspected FNAIT patients. Three prospective studies reported results from screening programmes, and 10 retrospective studies focused on suspected cases of FNAIT. In 8 studies samples for antibody measurement, performed by the monoclonal antibody immobilization of platelet antigen (MAIPA) assay, and samples for determining fetal/neonatal platelet count were collected simultaneously. In these 8 studies, the maternal antibody level correlated with the risk of severe thrombocytopenia. The prospective studies reported high negative predictive values (88-95%), which would allow for the use of maternal anti-HPA-1a antibody level as a predictive tool in a screening setting, in order to identify cases at low risk for FNAIT. However, due to low positive predictive values reported in prospective as well as retrospective studies (54-97%), the maternal antibody level is less suited for the final diagnosis and for guiding antenatal treatment. HPA-1a antibody level has the potential to predict the severity of FNAIT.

Sections du résumé

BACKGROUND AND OBJECTIVES OBJECTIVE
In Caucasians, fetal/neonatal alloimmune thrombocytopenia (FNAIT) is most commonly due to maternal HPA-1a antibodies. HPA-1a typing followed by screening for anti-HPA-1a antibodies in HPA-1bb women may identify first pregnancies at risk. Our goal was to review results from previous published studies to examine whether the maternal antibody level to HPA-1a could be used to identify high-risk pregnancies.
MATERIALS AND METHODS METHODS
The studies included were categorized by recruitment strategies: screening of unselected pregnancies or samples analyzed from known or suspected FNAIT patients.
RESULTS RESULTS
Three prospective studies reported results from screening programmes, and 10 retrospective studies focused on suspected cases of FNAIT. In 8 studies samples for antibody measurement, performed by the monoclonal antibody immobilization of platelet antigen (MAIPA) assay, and samples for determining fetal/neonatal platelet count were collected simultaneously. In these 8 studies, the maternal antibody level correlated with the risk of severe thrombocytopenia. The prospective studies reported high negative predictive values (88-95%), which would allow for the use of maternal anti-HPA-1a antibody level as a predictive tool in a screening setting, in order to identify cases at low risk for FNAIT. However, due to low positive predictive values reported in prospective as well as retrospective studies (54-97%), the maternal antibody level is less suited for the final diagnosis and for guiding antenatal treatment.
CONCLUSION CONCLUSIONS
HPA-1a antibody level has the potential to predict the severity of FNAIT.

Identifiants

pubmed: 30565711
doi: 10.1111/vox.12725
doi:

Substances chimiques

Antigens, Human Platelet 0
Biomarkers 0
ITGB3 protein, human 0
Integrin beta3 0

Types de publication

Journal Article Systematic Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

79-94

Subventions

Organisme : Canadian Blood Services
Organisme : European Union Seventh Framework Program PROFNAIT project

Investigateurs

Shubha Allard (S)
Celso Bianco (C)
Jeannie Callum (J)
Veerle Compernolle (V)
Dean Fergusson (D)
Susan Nahirniak (S)
Katerina Pavenski (K)
Joanne Pink (J)
Cynthia So-Osman (C)
Simon J Stanworth (SJ)
Zbigniew M Szczepiorkowski (ZM)
Erica Wood (E)

Informations de copyright

© 2018 International Society of Blood Transfusion.

Auteurs

Mette Kjaer (M)

Department of Laboratory Medicine, Diagnostic Clinic, University Hospital of North Norway, Tromsø, Norway.
Finnmark Hospital Trust, Finnmark, Norway.

Gerald Bertrand (G)

Platelet Immunology Department, French Blood Services of Brittany, Rennes, France.

Tamam Bakchoul (T)

Center for Clinical Transfusion Medicine, University of Tuebingen, Tuebingen, Germany.
Institute of Immunology and Transfusion Medicine, University Hospital Greifswald, Greifswald, Germany.

Edwin Massey (E)

NHS Blood and Transplant, Bristol, UK.

Jillian M Baker (JM)

Hospital for Sick Children, St. Michael's Hospital, Toronto, ON, Canada.

Lani Lieberman (L)

University Health Network, University of Toronto, Toronto, ON, Canada.

Susano Tanael (S)

Center for Innovation, Canadian Blood Services, Toronto, ON, Canada.

Andreas Greinacher (A)

Institute of Immunology and Transfusion Medicine, University Hospital Greifswald, Greifswald, Germany.

Michael F Murphy (MF)

NHS Blood and Transplant, Oxford University Hospitals and University of Oxford, Oxford, UK.

Donald M Arnold (DM)

Division of Hematology and Thromboembolism, McMaster University, Hamilton, ON, Canada.

Shoma Baidya (S)

Australian Red Cross Blood Service, Brisbane, QLD, Australia.

James Bussel (J)

Weill Cornell Medicine, New York, NY, USA.

Heather Hume (H)

Division of Hematology-Oncology, Centre Hospitalier Universitaire Sainte-Justine, University of Montreal, Montreal, QC, Canada.

Cécile Kaplan (C)

Institut National de la Transfusion Sanguine, Paris, France.

Dick Oepkes (D)

Department of Obstetrics, Leiden University Medical Center, Leiden, The Netherlands.

Greg Ryan (G)

Fetal Medicine Unit, Mount Sinai Hospital, Toronto, ON, Canada.

Helen Savoia (H)

Royal Children's Hospital, Melbourne, VIC, Australia.

Nadine Shehata (N)

Center for Innovation, Canadian Blood Services, Toronto, ON, Canada.
Department of Medicine, Mount Sinai Hospital, Toronto, ON, Canada.
Department of Obstetric Medicine, Mount Sinai Hospital, Toronto, ON, Canada.

Jens Kjeldsen-Kragh (J)

Department of Laboratory Medicine, Diagnostic Clinic, University Hospital of North Norway, Tromsø, Norway.
Department of Clinical Immunology and Transfusion Medicine, Regional and University Laboratories Region Skåne, Lund, Sweden.

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Classifications MeSH