Histologic subtype of treatment failures after noninvasive therapy for superficial basal cell carcinoma: An observational study.


Journal

Journal of the American Academy of Dermatology
ISSN: 1097-6787
Titre abrégé: J Am Acad Dermatol
Pays: United States
ID NLM: 7907132

Informations de publication

Date de publication:
Apr 2019
Historique:
received: 18 06 2018
revised: 24 10 2018
accepted: 13 12 2018
pubmed: 26 12 2018
medline: 13 4 2019
entrez: 25 12 2018
Statut: ppublish

Résumé

There have been concerns that recurrences after noninvasive therapy for basal cell carcinoma (BCC) transform into a "more aggressive" histologic subtype. We sought to evaluate the proportion of patients with a nonsuperficial treatment failure after noninvasive therapy for superficial BCC. An observational study was performed using data from a single blind, noninferiority, randomized controlled trial (March 2008-August 2010) with 5-year follow-up in patients with primary superficial BCC treated with methylaminolevulinate-photodynamic therapy, 5-fluorouracil, or imiquimod. Data were used from 166 adults with a histologically confirmed treatment failure. A nonsuperficial subtype was found in 64 of 166 treatment failures (38.6%). Proportions with a more aggressive subtype than the primary tumor were 51.3% (38/74) for early and 28.3% (26/92) for later treatment failures (P = .003). The proportion of more aggressive early failures was significantly lower after imiquimod (26.3%) compared with methylaminolevulinate-photodynamic therapy (54.8%, P = .086) and 5-fluorouracil (66.7%, P = .011). There was limited information on the exact time of occurrence of treatment failures. More aggressive treatment failure recurrences after noninvasive therapy for superficial BCC occur most often within the first 3 months posttreatment, probably indicating underdiagnosis of more aggressive components in the primary tumor rather than transformation.

Sections du résumé

BACKGROUND BACKGROUND
There have been concerns that recurrences after noninvasive therapy for basal cell carcinoma (BCC) transform into a "more aggressive" histologic subtype.
OBJECTIVE OBJECTIVE
We sought to evaluate the proportion of patients with a nonsuperficial treatment failure after noninvasive therapy for superficial BCC.
METHODS METHODS
An observational study was performed using data from a single blind, noninferiority, randomized controlled trial (March 2008-August 2010) with 5-year follow-up in patients with primary superficial BCC treated with methylaminolevulinate-photodynamic therapy, 5-fluorouracil, or imiquimod. Data were used from 166 adults with a histologically confirmed treatment failure.
RESULTS RESULTS
A nonsuperficial subtype was found in 64 of 166 treatment failures (38.6%). Proportions with a more aggressive subtype than the primary tumor were 51.3% (38/74) for early and 28.3% (26/92) for later treatment failures (P = .003). The proportion of more aggressive early failures was significantly lower after imiquimod (26.3%) compared with methylaminolevulinate-photodynamic therapy (54.8%, P = .086) and 5-fluorouracil (66.7%, P = .011).
LIMITATIONS CONCLUSIONS
There was limited information on the exact time of occurrence of treatment failures.
CONCLUSION CONCLUSIONS
More aggressive treatment failure recurrences after noninvasive therapy for superficial BCC occur most often within the first 3 months posttreatment, probably indicating underdiagnosis of more aggressive components in the primary tumor rather than transformation.

Identifiants

pubmed: 30582993
pii: S0190-9622(18)33100-1
doi: 10.1016/j.jaad.2018.12.028
pii:
doi:

Substances chimiques

Antimetabolites, Antineoplastic 0
Antineoplastic Agents 0
Photosensitizing Agents 0
methyl 5-aminolevulinate 585NM85KYM
Aminolevulinic Acid 88755TAZ87
Imiquimod P1QW714R7M
Fluorouracil U3P01618RT

Types de publication

Journal Article Observational Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

1022-1028

Informations de copyright

Copyright © 2018 American Academy of Dermatology, Inc. Published by Elsevier Inc. All rights reserved.

Auteurs

Lieke C J van Delft (LCJ)

Department of Dermatology, Maastricht University Medical Center+, Maastricht, the Netherlands; GROW-School for Oncology and Developmental Biology, Maastricht University Medical Center+, Maastricht, the Netherlands. Electronic address: lieke.van.delft@mumc.nl.

Patty J Nelemans (PJ)

Department of Epidemiology, Maastricht University, Maastricht, the Netherlands.

Maud H E Jansen (MHE)

Department of Dermatology, Maastricht University Medical Center+, Maastricht, the Netherlands; GROW-School for Oncology and Developmental Biology, Maastricht University Medical Center+, Maastricht, the Netherlands.

Aimee H M M Arits (AHMM)

Department of Dermatology, Maastricht University Medical Center+, Maastricht, the Netherlands; GROW-School for Oncology and Developmental Biology, Maastricht University Medical Center+, Maastricht, the Netherlands; Department of Dermatology, Catharina Hospital, Eindhoven, the Netherlands.

Marieke H Roozeboom (MH)

Department of Dermatology, Elkerliek Hospital, Helmond, the Netherlands.

Myrurgia A Hamid (MA)

Department of Pathology, Maastricht University Medical Center+, Maastricht, the Netherlands.

Klara Mosterd (K)

Department of Dermatology, Maastricht University Medical Center+, Maastricht, the Netherlands; GROW-School for Oncology and Developmental Biology, Maastricht University Medical Center+, Maastricht, the Netherlands.

Nicole W J Kelleners-Smeets (NWJ)

Department of Dermatology, Maastricht University Medical Center+, Maastricht, the Netherlands; GROW-School for Oncology and Developmental Biology, Maastricht University Medical Center+, Maastricht, the Netherlands.

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