Regulation of gingival epithelial cytokine response by bacterial cyclic dinucleotides.

Lipopolysaccharide inflammation interleukin keratinocytes periodontitis

Journal

Journal of oral microbiology
ISSN: 2000-2297
Titre abrégé: J Oral Microbiol
Pays: United States
ID NLM: 101551049

Informations de publication

Date de publication:
2019
Historique:
received: 25 09 2018
accepted: 17 10 2018
entrez: 2 1 2019
pubmed: 2 1 2019
medline: 2 1 2019
Statut: epublish

Résumé

Cyclic dinucleotides (cyclic di-guanosine monophosphate (c-di-GMP) and cyclic di-adenosine monophosphate (c-di-AMP)) and lipopolysaccharides (LPS) are pathogen-associated molecular patterns (PAMPs). Individual impacts of PAMPs on immune system have been evaluated, but simultaneous actions of multiple PAMPs have not been studied. Examination the effects of cyclic dinucleotides and Human gingival keratinocytes (HMK) were incubated with 1, 10, and 100 µM concentrations of c-di-GMP and c-di-AMP, either in the presence or absence of LPS decreased extracellular IL-8 levels, while the presence of c-di-AMP inhibited this effect. Incubating HMK cells with c-di-AMP (alone or with LPS) elevated the extracellular level of MCP-1. Extracellular VEGF level increased when cells were incubated with LPS and c-di-GMP together, or with c-di-AMP alone. LPS and c-di-AMP suppressed intracellular IL-1β levels. The c-di-AMP elevated intracellular levels of IL-1Ra. c-di-AMP and, to a lesser extent, c-di-GMP regulate keratinocyte cytokine response, either as an aggregator or as a suppressor of LPS, depending on the cytokine type.

Sections du résumé

BACKGROUND BACKGROUND
Cyclic dinucleotides (cyclic di-guanosine monophosphate (c-di-GMP) and cyclic di-adenosine monophosphate (c-di-AMP)) and lipopolysaccharides (LPS) are pathogen-associated molecular patterns (PAMPs). Individual impacts of PAMPs on immune system have been evaluated, but simultaneous actions of multiple PAMPs have not been studied.
OBJECTIVE OBJECTIVE
Examination the effects of cyclic dinucleotides and
METHODS METHODS
Human gingival keratinocytes (HMK) were incubated with 1, 10, and 100 µM concentrations of c-di-GMP and c-di-AMP, either in the presence or absence of
RESULTS RESULTS
LPS decreased extracellular IL-8 levels, while the presence of c-di-AMP inhibited this effect. Incubating HMK cells with c-di-AMP (alone or with LPS) elevated the extracellular level of MCP-1. Extracellular VEGF level increased when cells were incubated with LPS and c-di-GMP together, or with c-di-AMP alone. LPS and c-di-AMP suppressed intracellular IL-1β levels. The c-di-AMP elevated intracellular levels of IL-1Ra.
CONCLUSION CONCLUSIONS
c-di-AMP and, to a lesser extent, c-di-GMP regulate keratinocyte cytokine response, either as an aggregator or as a suppressor of LPS, depending on the cytokine type.

Identifiants

pubmed: 30598733
doi: 10.1080/20002297.2018.1538927
pii: 1538927
pmc: PMC6263105
doi:

Types de publication

Journal Article

Langues

eng

Pagination

1538927

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Auteurs

Samira Elmanfi (S)

Department of Periodontology, Institute of Dentistry, University of Turku, Turku, Finland.

Jie Zhou (J)

Department of Chemistry and Purdue Institute for Drug Discovery and Purdue Institute of Inflammation, Immunology and Infectious Disease, Purdue University, West Lafayette, IN, USA.

Herman O Sintim (HO)

Department of Chemistry and Purdue Institute for Drug Discovery and Purdue Institute of Inflammation, Immunology and Infectious Disease, Purdue University, West Lafayette, IN, USA.

Eija Könönen (E)

Department of Periodontology, Institute of Dentistry, University of Turku, Turku, Finland.
Oral Health Care, Welfare Division, City of Turku, Turku, Finland.

Mervi Gürsoy (M)

Department of Periodontology, Institute of Dentistry, University of Turku, Turku, Finland.

Ulvi Kahraman Gürsoy (UK)

Department of Periodontology, Institute of Dentistry, University of Turku, Turku, Finland.

Classifications MeSH