Activation of the hypothalamic paraventricular nucleus by acute intermittent hypoxia: Implications for sympathetic long-term facilitation neuroplasticity.
Animals
Arginine Vasopressin
/ metabolism
Arterial Pressure
Cell Count
Corticotropin-Releasing Hormone
/ metabolism
Heart Rate
Hypoxia
/ physiopathology
Long-Term Potentiation
Male
Neuronal Plasticity
Neuropeptides
/ metabolism
Paraventricular Hypothalamic Nucleus
/ physiopathology
Proto-Oncogene Proteins c-fos
/ metabolism
Rats
Rats, Sprague-Dawley
Sympathetic Nervous System
/ physiopathology
Anxiety
Depression
Hypertension
Neural plasticity
Sympathetic nerve activity
Journal
Experimental neurology
ISSN: 1090-2430
Titre abrégé: Exp Neurol
Pays: United States
ID NLM: 0370712
Informations de publication
Date de publication:
04 2019
04 2019
Historique:
received:
15
09
2018
revised:
03
12
2018
accepted:
30
12
2018
pubmed:
4
1
2019
medline:
23
2
2020
entrez:
4
1
2019
Statut:
ppublish
Résumé
Exposure to acute intermittent hypoxia (AIH) induces a progressive increase of sympathetic nerve activity (SNA) that reflects a form of neuroplasticity known as sympathetic long-term facilitation (sLTF). Our recent findings indicate that activity of neurons in the hypothalamic paraventricular nucleus (PVN) contributes to AIH-induced sLTF, but neither the intra-PVN distribution nor the neurochemical identity of AIH responsive neurons has been determined. Here, awake rats were exposed to 10 cycles of AIH and c-Fos immunohistochemistry was performed to identify transcriptionally activated neurons in rostral, middle and caudal planes of the PVN. Effects of graded intensities of AIH were investigated in separate groups of rats (n = 6/group) in which inspired oxygen (O
Identifiants
pubmed: 30605624
pii: S0014-4886(18)30481-3
doi: 10.1016/j.expneurol.2018.12.011
pmc: PMC6378125
mid: NIHMS1518209
pii:
doi:
Substances chimiques
Neuropeptides
0
Proto-Oncogene Proteins c-fos
0
Arginine Vasopressin
113-79-1
Corticotropin-Releasing Hormone
9015-71-8
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
1-8Subventions
Organisme : NHLBI NIH HHS
ID : P01 HL088052
Pays : United States
Organisme : NHLBI NIH HHS
ID : T32 HL007446
Pays : United States
Informations de copyright
Copyright © 2019 Elsevier Inc. All rights reserved.
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