Effects of a common pharmaceutical, atorvastatin, on energy metabolism and detoxification mechanisms of a marine bivalve Mytilus edulis.
ATP Binding Cassette Transporter, Subfamily B, Member 1
/ metabolism
Animals
Aquatic Organisms
/ drug effects
Atorvastatin
/ chemistry
Biotransformation
/ drug effects
Cell Respiration
/ drug effects
Energy Metabolism
/ drug effects
Gene Expression Regulation
/ drug effects
Hemocytes
/ drug effects
Inactivation, Metabolic
/ drug effects
Metabolome
/ drug effects
Mitochondria
/ drug effects
Mytilus edulis
/ drug effects
Oxygen Consumption
/ drug effects
RNA, Messenger
/ genetics
Stress, Physiological
/ drug effects
Water Pollutants, Chemical
/ toxicity
Bioenergetics
Biotransformation
Carbohydrates
LC–MS analysis
Lipids
Mitochondria
Oxygen consumption
Proteins
Statins
Toxicity
Journal
Aquatic toxicology (Amsterdam, Netherlands)
ISSN: 1879-1514
Titre abrégé: Aquat Toxicol
Pays: Netherlands
ID NLM: 8500246
Informations de publication
Date de publication:
Mar 2019
Mar 2019
Historique:
received:
21
11
2018
revised:
27
12
2018
accepted:
28
12
2018
pubmed:
6
1
2019
medline:
9
3
2019
entrez:
6
1
2019
Statut:
ppublish
Résumé
Biologically active compounds from pharmaceuticals cause concern due to their common occurrence in water and sediments of urbanized coasts and potential threat to marine organisms. Atorvastatin (ATO), a globally prescribed drug, is environmentally stable and bioavailable to marine organisms; however, the physiological and toxic effects of this drug on ecologically important coastal species are yet to be elucidated. We studied the effect of ATO (˜1.2 μg L
Identifiants
pubmed: 30610964
pii: S0166-445X(18)31035-X
doi: 10.1016/j.aquatox.2018.12.022
pii:
doi:
Substances chimiques
ATP Binding Cassette Transporter, Subfamily B, Member 1
0
RNA, Messenger
0
Water Pollutants, Chemical
0
Atorvastatin
A0JWA85V8F
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Pagination
47-61Informations de copyright
Copyright © 2018 Elsevier B.V. All rights reserved.