Of Aging Mice and Men: Gait Speed Decline Is a Translatable Trait, With Species-Specific Underlying Properties.


Journal

The journals of gerontology. Series A, Biological sciences and medical sciences
ISSN: 1758-535X
Titre abrégé: J Gerontol A Biol Sci Med Sci
Pays: United States
ID NLM: 9502837

Informations de publication

Date de publication:
16 08 2019
Historique:
received: 11 12 2018
accepted: 11 01 2019
pubmed: 17 1 2019
medline: 12 6 2020
entrez: 17 1 2019
Statut: ppublish

Résumé

In the last two decades, great strides were made in our ability to extend the life span of model organisms through dietary and other manipulations. Survival curves provide evidence of altered aging processes but are uninformative on what lead to that increase in life span. Longitudinal assessments of health and function during intervention studies could help in the identification of predictive biomarkers for health and survival. Comparable biomarkers of healthspan are necessary to effectively translate interventions into human clinical trials. Gait speed is a well-established predictive biomarker of healthspan in humans for risk of disability, health outcomes and mortality, and is relatively simple to assess noninvasively in rodents. In this study, we assessed and compared gait speed in males from two species (mice and humans), from young adulthood to advanced old age. Although gait speed decreases nonlinearly with age in both species, the underlying drivers of this change in gait speed were different, with humans exhibiting a shortened step length, and mice displaying a decrease in cadence. Future longitudinal and interventional studies in mice should examine the predictive value of longitudinal declines in gait speed for health and survival.

Identifiants

pubmed: 30649206
pii: 5289189
doi: 10.1093/gerona/glz015
pmc: PMC6696716
doi:

Types de publication

Comparative Study Journal Article Research Support, N.I.H., Intramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

1413-1416

Informations de copyright

Published by Oxford University Press on behalf of The Gerontological Society of America 2019.

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Auteurs

Woei-Nan Bair (WN)

Translational Gerontology Branch, National Institute on Aging, Intramural Research Program, National Institute of Health, Baltimore, Maryland.

Michael Petr (M)

Translational Gerontology Branch, National Institute on Aging, Intramural Research Program, National Institute of Health, Baltimore, Maryland.
Center for Healthy Aging, ICMM, University of Copenhagen, Denmark.

Irene Alfaras (I)

Aging Institute, University of Pittsburgh School of Medicine, Pennsylvania.

Sarah J Mitchell (SJ)

Genetics and Complex Diseases, Harvard T.H. Chan School of Public Health, Boston, Massachusetts.

Michel Bernier (M)

Translational Gerontology Branch, National Institute on Aging, Intramural Research Program, National Institute of Health, Baltimore, Maryland.

Luigi Ferrucci (L)

Translational Gerontology Branch, National Institute on Aging, Intramural Research Program, National Institute of Health, Baltimore, Maryland.

Stephanie A Studenski (SA)

Translational Gerontology Branch, National Institute on Aging, Intramural Research Program, National Institute of Health, Baltimore, Maryland.

Rafael De Cabo (R)

Translational Gerontology Branch, National Institute on Aging, Intramural Research Program, National Institute of Health, Baltimore, Maryland.

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