Methylglyoxal evokes acute Ca

Animals Calcium / metabolism Calcium Release Activated Calcium Channels / metabolism Cells, Cultured Endothelial Cells / drug effects Mice Pyruvaldehyde / pharmacology
Calcium entry Calcium release activated calcium (CRAC) channels Endothelial cells Methylglyoxal

Journal

Cell calcium
ISSN: 1532-1991
Titre abrégé: Cell Calcium
Pays: Netherlands
ID NLM: 8006226

Informations de publication

Date de publication:
03 2019
Historique:
received: 20 09 2018
revised: 08 01 2019
accepted: 08 01 2019
pubmed: 19 1 2019
medline: 14 5 2020
entrez: 19 1 2019
Statut: ppublish

Résumé

Methylglyoxal (MG) is a by-product of glucose metabolism and its accumulation has been linked to the development of diabetic complications such as retinopathy and nephropathy by affecting multiple signalling pathways. However, its influence on the intracellular Ca

Identifiants

DOI: 10.1016/j.ceca.2019.01.002 PMID: 30658323
pubmed: 30658323
pii: S0143-4160(18)30178-7
doi: 10.1016/j.ceca.2019.01.002
pii:
doi:

Substances chimiques

Calcium Release Activated Calcium Channels 0
Pyruvaldehyde 722KLD7415
Calcium SY7Q814VUP

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

66-75

Informations de copyright

Copyright © 2019 Elsevier Ltd. All rights reserved.

Auteurs

Robin Sachdeva (R)

Institute of Pharmacology, Heidelberg University, Im Neuenheimer Feld 366, 69120 Heidelberg, Germany.

Thomas Fleming (T)

Department of Medicine I and Clinical Chemistry, Heidelberg University Hospital, Germany; German Center for Diabetes Research (DZD), Germany.

Dagmar Schumacher (D)

Institute of Pharmacology, Heidelberg University, Im Neuenheimer Feld 366, 69120 Heidelberg, Germany.

Sarah Homberg (S)

Institute of Pharmacology, Heidelberg University, Im Neuenheimer Feld 366, 69120 Heidelberg, Germany.

Kathrin Stilz (K)

Institute of Pharmacology, Heidelberg University, Im Neuenheimer Feld 366, 69120 Heidelberg, Germany.

Franziska Mohr (F)

Institute of Physiology and Pathophysiology, Division of Cardiovascular Physiology, Im Neuenheimer Feld 326, 69120 Heidelberg, Germany.

Andreas H Wagner (AH)

Institute of Physiology and Pathophysiology, Division of Cardiovascular Physiology, Im Neuenheimer Feld 326, 69120 Heidelberg, Germany.

Volodymyr Tsvilovskyy (V)

Institute of Pharmacology, Heidelberg University, Im Neuenheimer Feld 366, 69120 Heidelberg, Germany.

Ilka Mathar (I)

Institute of Pharmacology, Heidelberg University, Im Neuenheimer Feld 366, 69120 Heidelberg, Germany.

Marc Freichel (M)

Institute of Pharmacology, Heidelberg University, Im Neuenheimer Feld 366, 69120 Heidelberg, Germany. Electronic address: marc.freichel@pharma.uni-heidelberg.de.

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Classifications MeSH

questionsmedicales.fr › Eucaryotes › Animaux › Methylglyoxal evokes acute Ca
questionsmedicales.fr › Facteurs biologiques › Facteurs de la coagulation sanguine › Calcium › Methylglyoxal evokes acute Ca
questionsmedicales.fr › Acides aminés, peptides et protéines › Protéines › Protéines membranaires › Protéines de transport membranaire › Canaux ioniques › Canaux calciques › Canaux calciques activés par la libération de calcium › Methylglyoxal evokes acute Ca
questionsmedicales.fr › Cellules › Cellules cultivées › Methylglyoxal evokes acute Ca
questionsmedicales.fr › Cellules › Cellules épithéliales › Cellules endothéliales › Methylglyoxal evokes acute Ca
questionsmedicales.fr › Eucaryotes › Animaux › Chordés › Vertébrés › Mammifères › Eutheria › Rodentia › Muridae › Murinae › Souris › Methylglyoxal evokes acute Ca
questionsmedicales.fr › Composés chimiques organiques › Aldéhydes › Glyoxal › Méthylglyoxal › Methylglyoxal evokes acute Ca