MNK1/NODAL Signaling Promotes Invasive Progression of Breast Ductal Carcinoma
Animals
Breast Carcinoma In Situ
/ metabolism
Breast Neoplasms
/ metabolism
CRISPR-Cas Systems
Carcinoma, Ductal, Breast
/ metabolism
Cell Line, Tumor
Cell Proliferation
Disease Progression
Female
Heterografts
Humans
Intracellular Signaling Peptides and Proteins
/ genetics
Mice
Mice, Nude
Nodal Protein
/ metabolism
Protein Serine-Threonine Kinases
/ genetics
Signal Transduction
Journal
Cancer research
ISSN: 1538-7445
Titre abrégé: Cancer Res
Pays: United States
ID NLM: 2984705R
Informations de publication
Date de publication:
01 04 2019
01 04 2019
Historique:
received:
24
05
2018
revised:
02
11
2018
accepted:
10
01
2019
pubmed:
20
1
2019
medline:
1
1
2020
entrez:
20
1
2019
Statut:
ppublish
Résumé
The mechanisms by which breast cancers progress from relatively indolent ductal carcinoma
Identifiants
pubmed: 30659022
pii: 0008-5472.CAN-18-1602
doi: 10.1158/0008-5472.CAN-18-1602
pmc: PMC6513674
mid: NIHMS1022476
doi:
Substances chimiques
Intracellular Signaling Peptides and Proteins
0
NODAL protein, human
0
Nodal Protein
0
MKNK1 protein, human
EC 2.7.1.-
Protein Serine-Threonine Kinases
EC 2.7.11.1
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
1646-1657Subventions
Organisme : NCI NIH HHS
ID : R01 CA207445
Pays : United States
Organisme : NCI NIH HHS
ID : R21 CA226567
Pays : United States
Organisme : CIHR
ID : PJT-156269
Pays : Canada
Organisme : CIHR
ID : MOP-142281
Pays : Canada
Informations de copyright
©2019 American Association for Cancer Research.
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