Structural and Functional Studies of the RBPJ-SHARP Complex Reveal a Conserved Corepressor Binding Site.


Journal

Cell reports
ISSN: 2211-1247
Titre abrégé: Cell Rep
Pays: United States
ID NLM: 101573691

Informations de publication

Date de publication:
22 01 2019
Historique:
received: 31 05 2018
revised: 05 11 2018
accepted: 21 12 2018
entrez: 24 1 2019
pubmed: 24 1 2019
medline: 7 3 2020
Statut: ppublish

Résumé

Notch is a conserved signaling pathway that is essential for metazoan development and homeostasis; dysregulated signaling underlies the pathophysiology of numerous human diseases. Receptor-ligand interactions result in gene expression changes, which are regulated by the transcription factor RBPJ. RBPJ forms a complex with the intracellular domain of the Notch receptor and the coactivator Mastermind to activate transcription, but it can also function as a repressor by interacting with corepressor proteins. Here, we determine the structure of RBPJ bound to the corepressor SHARP and DNA, revealing its mode of binding to RBPJ. We tested structure-based mutants in biophysical and biochemical-cellular assays to characterize the role of RBPJ as a repressor, clearly demonstrating that RBPJ mutants deficient for SHARP binding are incapable of repressing transcription of genes responsive to Notch signaling in cells. Altogether, our structure-function studies provide significant insights into the repressor function of RBPJ.

Identifiants

pubmed: 30673607
pii: S2211-1247(18)32057-6
doi: 10.1016/j.celrep.2018.12.097
pmc: PMC6352735
mid: NIHMS1519366
pii:
doi:

Substances chimiques

DNA-Binding Proteins 0
Immunoglobulin J Recombination Signal Sequence-Binding Protein 0
Multiprotein Complexes 0
RNA-Binding Proteins 0
Rbpj protein, mouse 0
Receptors, Notch 0
Spen protein, mouse 0
DNA 9007-49-2

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, Non-P.H.S.

Langues

eng

Sous-ensembles de citation

IM

Pagination

845-854.e6

Subventions

Organisme : NCI NIH HHS
ID : R01 CA178974
Pays : United States

Informations de copyright

Copyright © 2018 The Authors. Published by Elsevier Inc. All rights reserved.

Auteurs

Zhenyu Yuan (Z)

Department of Molecular Genetics, Biochemistry and Microbiology, University of Cincinnati College of Medicine, Cincinnati, OH, USA.

Bradley D VanderWielen (BD)

Department of Molecular Genetics, Biochemistry and Microbiology, University of Cincinnati College of Medicine, Cincinnati, OH, USA.

Benedetto Daniele Giaimo (BD)

Institute of Biochemistry, University of Giessen, Giessen, Germany.

Leiling Pan (L)

Department of Internal Medicine I, Center for Internal Medicine, University Medical Center Ulm, 89081 Ulm, Germany.

Courtney E Collins (CE)

Department of Molecular Genetics, Biochemistry and Microbiology, University of Cincinnati College of Medicine, Cincinnati, OH, USA.

Aleksandra Turkiewicz (A)

Institute of Biochemistry, University of Giessen, Giessen, Germany.

Kerstin Hein (K)

Institute of Biochemistry, University of Giessen, Giessen, Germany.

Franz Oswald (F)

Department of Internal Medicine I, Center for Internal Medicine, University Medical Center Ulm, 89081 Ulm, Germany.

Tilman Borggrefe (T)

Institute of Biochemistry, University of Giessen, Giessen, Germany.

Rhett A Kovall (RA)

Department of Molecular Genetics, Biochemistry and Microbiology, University of Cincinnati College of Medicine, Cincinnati, OH, USA. Electronic address: kovallra@ucmail.uc.edu.

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Classifications MeSH