IgE-Mediated Sensitization to Galactose-α-1,3- Galactose (α-Gal) in Urticaria and Anaphylaxis in Spain: Geographical Variations and Risk Factors.


Journal

Journal of investigational allergology & clinical immunology
ISSN: 1018-9068
Titre abrégé: J Investig Allergol Clin Immunol
Pays: Spain
ID NLM: 9107858

Informations de publication

Date de publication:
2019
Historique:
pubmed: 25 1 2019
medline: 23 5 2020
entrez: 25 1 2019
Statut: ppublish

Résumé

The objectives of this study were to investigate the prevalence of sIgE to galactose-α-1,3-galactose (α-gal) in individuals with acute urticaria or anaphylaxis from different geographical areas of Spain and to evaluate the relevance of demographics and lifestyle as risk factors for this immune response. Participants were recruited from allergy departments at 14 Spanish hospitals. Patients aged 18 years or older presenting with urticaria or anaphylaxis were enrolled into one of 2 arms: cases and controls. An interviewer-administered questionnaire collecting demographic data, lifestyle habits, and the presence of cofactors was obtained from each participant. sIgE to α-gal and total IgE were determined using ImmunoCAP. sIgE levels ≥0.35 kU/L were considered a positive result. The study population comprised 160 cases and 126 controls. The median age was 44 years. The overall prevalence of a positive result of sIgE to α-gal was 15.7%; this was higher in cases (26.3%) than in controls (2.4%). The sIgE anti-α-gal positivity rate ranged from 37.68% (rural) to 15.38% (semiurban), and 7.85% (urban). The rates of positivity were 46.32%, (Northern), 0.72% (Center), and 0% (Mediterranean). A positive result for sIgE to α-gal was associated with a history of tick bites, participation in outdoor activities, pet ownership, and ingestion of mammalian meats or innards before the onset of symptoms. Only alcohol consumption could be implicated as a cofactor. Sensitization to α-gal in patients with urticaria or anaphylaxis differs considerably between the 3 geographical areas studied and is related to tick bites.

Sections du résumé

BACKGROUND BACKGROUND
The objectives of this study were to investigate the prevalence of sIgE to galactose-α-1,3-galactose (α-gal) in individuals with acute urticaria or anaphylaxis from different geographical areas of Spain and to evaluate the relevance of demographics and lifestyle as risk factors for this immune response.
METHODS METHODS
Participants were recruited from allergy departments at 14 Spanish hospitals. Patients aged 18 years or older presenting with urticaria or anaphylaxis were enrolled into one of 2 arms: cases and controls. An interviewer-administered questionnaire collecting demographic data, lifestyle habits, and the presence of cofactors was obtained from each participant. sIgE to α-gal and total IgE were determined using ImmunoCAP. sIgE levels ≥0.35 kU/L were considered a positive result.
RESULTS RESULTS
The study population comprised 160 cases and 126 controls. The median age was 44 years. The overall prevalence of a positive result of sIgE to α-gal was 15.7%; this was higher in cases (26.3%) than in controls (2.4%). The sIgE anti-α-gal positivity rate ranged from 37.68% (rural) to 15.38% (semiurban), and 7.85% (urban). The rates of positivity were 46.32%, (Northern), 0.72% (Center), and 0% (Mediterranean). A positive result for sIgE to α-gal was associated with a history of tick bites, participation in outdoor activities, pet ownership, and ingestion of mammalian meats or innards before the onset of symptoms. Only alcohol consumption could be implicated as a cofactor.
CONCLUSION CONCLUSIONS
Sensitization to α-gal in patients with urticaria or anaphylaxis differs considerably between the 3 geographical areas studied and is related to tick bites.

Identifiants

pubmed: 30676319
doi: 10.18176/jiaci.0373
doi:

Substances chimiques

Allergens 0
Disaccharides 0
galactosyl-(1-3)galactose 13168-24-6
Immunoglobulin E 37341-29-0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

436-443

Auteurs

M B Mateo Borrega (MB)

GAI Guadalajara, Department of Allergy, Hospital Universitario de Guadalajara, Guadalajara, Spain.

B Garcia (B)

Department of Allergy, Complejo Hospitalario de Navarra, Pamplona, Spain.

C H Larramendi (CH)

Allergy Section, Hospital Marina Baixa, Villajoyosa, Spain.

J Azofra (J)

Department of Allergy, Hospital Universitario Central de Asturias, Oviedo, Spain.

E González Mancebo (E)

Department of Allergy, Hospital Universitario de Fuenlabrada, Madrid, Spain.

M I Alvarado (MI)

Department of Allergy, Hospital Virgen del Puerto, Plasencia, Spain.

M D Alonso Díaz de Durana (MD)

Department of Allergy, Hospital Universitario Fundación de Alcorcón, Madrid, Spain.

R Núñez Orjales (R)

Department of Allergy, Hospital Universitario Lucus Augusti, Lugo, Spain.

M C Diéguez (MC)

Department of Allergy, Research Institute Hospital 12 de Octubre (i+12), Madrid, Spain.

M Guilarte (M)

Department of Allergy, Hospital Universitari de la Vall d'Hebron, Barcelona, Spain.

A M Soriano Galarraga (AM)

Department of Allergy, Hospital de Basurto, Bilbao, Spain.

G Sosa (G)

Department of Allergy, Hospital La Zarzuela, Madrid, Spain.

A Ferrer (A)

Department of Allergy, Hospital Vega Baja, Orihuela, Spain.

A García Moral (A)

Department of Allergy, Hospital de la Santa Creu i Sant Pau, Barcelona, Spain.

A M Beristain (AM)

Department of Allergy, Hospital Universitario Central de Asturias, Oviedo, Spain.

J Bartra (J)

Allergy Unit, Pneumology Department, Hospital Clinic, Barcelona, Spain.

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Classifications MeSH