Intracranial Aneurysms in Sickle Cell Disease.


Journal

Current neurovascular research
ISSN: 1875-5739
Titre abrégé: Curr Neurovasc Res
Pays: United Arab Emirates
ID NLM: 101208439

Informations de publication

Date de publication:
2019
Historique:
received: 01 01 2019
revised: 16 01 2019
accepted: 21 01 2019
pubmed: 2 2 2019
medline: 31 3 2020
entrez: 2 2 2019
Statut: ppublish

Résumé

The exact causes of intracranial aneurysms (IAs) are still unknown. However, certain diseases are known to be associated with IAs. To analyze the differences in IA characteristics in the general population and in individuals with sickle-cell disease (SCD). We systematically searched PubMed, Scopus, Web of Science, and Cochrane Library for Data on SCD patients with IAs. We compared IA characteristics of SCD patients with those from 2451 healthy IA carriers from our observational cohort. 129 SCD patients with IAs were identified in 42 studies. The SCD patient cohort was characterized by younger age (mean 27.1 vs 54.9 years, p<0.0001) and lower female prevalence (57.7% vs 68.4%, p=0.0177). The prevalence (47% vs 34.5%, p=0.004) and the number (3.02 vs 2.56 IAs/patient, p=0.004) of multiple IAs were also higher in the SCD cohort. Unruptured IAs (3.27 vs 6.16 mm, p<0.0001), but not ruptured IAs (7.8 vs 7.34 mm, p=0.9086) were significantly smaller in the SCD cohort. In addition, IAs were more frequently located in the internal carotid artery (45% vs 29%, p<0.0001) or posterior circulation (43% vs 20%, p<0.0001). Higher age (≥30 years, p=0.007), IA size ≥7 mm (p=0.008), and location in posterior circulation (p=0.01) were independently associated with subarachnoid hemorrhage in SCD. There is a distinct demographic and radiographic pattern of IA in SCD. Risk factors for IA rupture in SCD are mostly congruent with those in healthy individuals.

Sections du résumé

BACKGROUND
The exact causes of intracranial aneurysms (IAs) are still unknown. However, certain diseases are known to be associated with IAs.
OBJECTIVE
To analyze the differences in IA characteristics in the general population and in individuals with sickle-cell disease (SCD).
METHODS
We systematically searched PubMed, Scopus, Web of Science, and Cochrane Library for Data on SCD patients with IAs. We compared IA characteristics of SCD patients with those from 2451 healthy IA carriers from our observational cohort.
RESULTS
129 SCD patients with IAs were identified in 42 studies. The SCD patient cohort was characterized by younger age (mean 27.1 vs 54.9 years, p<0.0001) and lower female prevalence (57.7% vs 68.4%, p=0.0177). The prevalence (47% vs 34.5%, p=0.004) and the number (3.02 vs 2.56 IAs/patient, p=0.004) of multiple IAs were also higher in the SCD cohort. Unruptured IAs (3.27 vs 6.16 mm, p<0.0001), but not ruptured IAs (7.8 vs 7.34 mm, p=0.9086) were significantly smaller in the SCD cohort. In addition, IAs were more frequently located in the internal carotid artery (45% vs 29%, p<0.0001) or posterior circulation (43% vs 20%, p<0.0001). Higher age (≥30 years, p=0.007), IA size ≥7 mm (p=0.008), and location in posterior circulation (p=0.01) were independently associated with subarachnoid hemorrhage in SCD.
CONCLUSION
There is a distinct demographic and radiographic pattern of IA in SCD. Risk factors for IA rupture in SCD are mostly congruent with those in healthy individuals.

Identifiants

pubmed: 30706782
pii: CNR-EPUB-96271
doi: 10.2174/1567202616666190131160847
doi:

Types de publication

Journal Article Observational Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

63-76

Informations de copyright

Copyright© Bentham Science Publishers; For any queries, please email at epub@benthamscience.net.

Auteurs

Ramazan Jabbarli (R)

Department of Neurosurgery, University Hospital of Essen, Essen, Germany.

Thiemo F Dinger (TF)

Department of Neurosurgery, University Hospital of Essen, Essen, Germany.

Daniela Pierscianek (D)

Department of Neurosurgery, University Hospital of Essen, Essen, Germany.

Marvin D Oppong (MD)

Department of Neurosurgery, University Hospital of Essen, Essen, Germany.

Bixia Chen (B)

Department of Neurosurgery, University Hospital of Essen, Essen, Germany.

Philipp Dammann (P)

Department of Neurosurgery, University Hospital of Essen, Essen, Germany.

Karsten H Wrede (KH)

Department of Neurosurgery, University Hospital of Essen, Essen, Germany.

Klaus Kaier (K)

Institute for Medical Biometry and Statistics, University of Freiburg, Freiburg Breisgau, Germany.

Martin Köhrmann (M)

Clinic for Neurology, University Hospital of Essen, Essen, Germany.

Michael Forsting (M)

Institute for Diagnostic and Interventional Radiology, University Hospital of Essen, Essen, Germany.

Christoph Kleinschnitz (C)

Clinic for Neurology, University Hospital of Essen, Essen, Germany.

Ulrich Sure (U)

Department of Neurosurgery, University Hospital of Essen, Essen, Germany.

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