Urinary lead in relation to combustion-derived air pollution in urban environments. A longitudinal study of an international panel.


Journal

Environment international
ISSN: 1873-6750
Titre abrégé: Environ Int
Pays: Netherlands
ID NLM: 7807270

Informations de publication

Date de publication:
04 2019
Historique:
received: 21 08 2018
revised: 16 01 2019
accepted: 16 01 2019
pubmed: 3 2 2019
medline: 29 10 2019
entrez: 3 2 2019
Statut: ppublish

Résumé

Urinary lead (Pb) is generally considered to have limited use in biomonitoring environmental exposure to lead. Carbon load in airway macrophages (AM BC) is an internal marker to assess long-term exposure to combustion-derived aerosol particles. In urban environments, atmospheric Pb and black carbon may have common sources. We aimed to study the temporal change of urinary Pb (U-Pb) when exposure to outdoor air pollution changes, and the relationship between U-Pb and AM BC. A panel of 50 young healthy adults [mean (SD) 26.7 (5.2) years], including 17 long-term (>1 year) residents in Leuven, Belgium (BE), 15 and 18 newcomers (arrived <3 weeks) from low- and middle-income countries (LMIC) and high-income countries (HIC), respectively, underwent 8 repeated measurements at 6 weeks intervals. In urine spot samples obtained at 5 time points (T1, T2, T4, T6, T8), 24 trace elements were quantified by inductively coupled plasma-mass spectrometry. At each time point, AM BC was quantified as the median surface of black inclusions (in μm Only U-Pb differed between groups and exhibited significant time trends. Participants from the LMIC group had significantly higher initial U-Pb (1.18 μg/g creat) than the HIC group (0.44 μg/g creat) and BE group (0.45 μg/g creat). In the LMIC group, U-Pb decreased significantly with time by 0.061 μg/g creatinine per 30 days [95% confidence interval (CI): 0.034, 0.088]. U-Pb remained unchanged in the other two groups. An increase in AM BC of 1 μm This panel study demonstrates that U-Pb may be a valid alternative to blood Pb for biomonitoring changes in exposure to lead, at least at group level. In addition, we identified a positive association between U-Pb and AM BC, a biomarker of exposure to traffic-related air pollution, suggesting the existence of common sources of Pb and black carbon in urban environments.

Sections du résumé

BACKGROUND
Urinary lead (Pb) is generally considered to have limited use in biomonitoring environmental exposure to lead. Carbon load in airway macrophages (AM BC) is an internal marker to assess long-term exposure to combustion-derived aerosol particles. In urban environments, atmospheric Pb and black carbon may have common sources. We aimed to study the temporal change of urinary Pb (U-Pb) when exposure to outdoor air pollution changes, and the relationship between U-Pb and AM BC.
METHODS
A panel of 50 young healthy adults [mean (SD) 26.7 (5.2) years], including 17 long-term (>1 year) residents in Leuven, Belgium (BE), 15 and 18 newcomers (arrived <3 weeks) from low- and middle-income countries (LMIC) and high-income countries (HIC), respectively, underwent 8 repeated measurements at 6 weeks intervals. In urine spot samples obtained at 5 time points (T1, T2, T4, T6, T8), 24 trace elements were quantified by inductively coupled plasma-mass spectrometry. At each time point, AM BC was quantified as the median surface of black inclusions (in μm
RESULTS
Only U-Pb differed between groups and exhibited significant time trends. Participants from the LMIC group had significantly higher initial U-Pb (1.18 μg/g creat) than the HIC group (0.44 μg/g creat) and BE group (0.45 μg/g creat). In the LMIC group, U-Pb decreased significantly with time by 0.061 μg/g creatinine per 30 days [95% confidence interval (CI): 0.034, 0.088]. U-Pb remained unchanged in the other two groups. An increase in AM BC of 1 μm
CONCLUSION
This panel study demonstrates that U-Pb may be a valid alternative to blood Pb for biomonitoring changes in exposure to lead, at least at group level. In addition, we identified a positive association between U-Pb and AM BC, a biomarker of exposure to traffic-related air pollution, suggesting the existence of common sources of Pb and black carbon in urban environments.

Identifiants

pubmed: 30710802
pii: S0160-4120(18)31892-0
doi: 10.1016/j.envint.2019.01.044
pii:
doi:

Substances chimiques

Air Pollutants 0
Soot 0
Vehicle Emissions 0
Lead 2P299V784P

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

75-81

Informations de copyright

Copyright © 2019 Elsevier Ltd. All rights reserved.

Auteurs

Yang Bai (Y)

Centre for Environment and Health, KU Leuven, Herestraat 49, O&N 1 box 706, 3000 Leuven, Belgium. Electronic address: yang.bai@kuleuven.be.

Annouschka Laenen (A)

Leuven Biostatistics and Statistical Bioinformatics Centre (L-BioStat), Kapucijnenvoer 35 blok d, box 7001, 3000 Leuven, Belgium. Electronic address: annouschka.laenen@kuleuven.be.

Vincent Haufroid (V)

Louvain Centre for Toxicology and Applied Pharmacology (LTAP), Université Catholique de Louvain, Avenue Mounier 52/B1.52.12, 1200 Woluwe-Saint-Lambert, Belgium. Electronic address: vincent.haufroid@uclouvain.be.

Tim S Nawrot (TS)

Centre for Environment and Health, KU Leuven, Herestraat 49, O&N 1 box 706, 3000 Leuven, Belgium; Centre for Environmental Sciences, Hasselt University, Agoralaan Building D, 3590 Diepenbeek, Belgium. Electronic address: tim.nawrot@uhasselt.be.

Benoit Nemery (B)

Centre for Environment and Health, KU Leuven, Herestraat 49, O&N 1 box 706, 3000 Leuven, Belgium. Electronic address: ben.nemery@kuleuven.be.

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Classifications MeSH