Exceptional human longevity is associated with a specific plasma phenotype of ether lipids.

Alkenyl phospholipids Alkyl phospholipids Centenarians Fatty acid unsaturation Mass spectrometry Phosphatidylcholine Phosphatidylethanolamine Plasmalogens

Journal

Redox biology
ISSN: 2213-2317
Titre abrégé: Redox Biol
Pays: Netherlands
ID NLM: 101605639

Informations de publication

Date de publication:
02 2019
Historique:
received: 12 11 2018
revised: 09 01 2019
accepted: 28 01 2019
pubmed: 4 2 2019
medline: 2 4 2019
entrez: 4 2 2019
Statut: ppublish

Résumé

A lipid profile resistant to oxidative damage is an inherent trait associated with animal lifespan. However, there is a lack of lipidomic studies on human longevity. Here we use mass spectrometry based technologies to detect and quantify 137 ether lipids to define a phenotype of healthy humans with exceptional lifespan. Ether lipids were chosen because of their antioxidant properties and ability to modulate oxidative stress. Our results demonstrate that a specific ether lipid signature can be obtained to define the centenarian state. This profile comprises higher level of alkyl forms derived from phosphatidylcholine with shorter number of carbon atoms and double bonds; and decreased content in alkenyl forms from phosphatidylethanolamine with longer chain length and higher double bonds. This compositional pattern suggests that ether lipids from centenarians are more resistant to lipid peroxidation, and that ether lipid signature expresses an optimized feature associated with exceptional human longevity. These results are in keeping with the free radical theory of aging.

Identifiants

pubmed: 30711699
pii: S2213-2317(18)31058-9
doi: 10.1016/j.redox.2019.101127
pmc: PMC6357979
pii:
doi:

Substances chimiques

Lipids 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

101127

Informations de copyright

Copyright © 2019 The Authors. Published by Elsevier B.V. All rights reserved.

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Auteurs

I Pradas (I)

Department of Experimental Medicine, University of Lleida-Institute for Research in Biomedicine of Lleida (UdL-IRBLleida), Lleida 25198, Spain. Electronic address: ipradas@mex.udl.cat.

M Jové (M)

Department of Experimental Medicine, University of Lleida-Institute for Research in Biomedicine of Lleida (UdL-IRBLleida), Lleida 25198, Spain. Electronic address: mariona.jove@udl.cat.

K Huynh (K)

Baker Heart and Diabetes Institute, Melbourne, VIC 3004, Australia. Electronic address: kevin.huynh@baker.edu.au.

J Puig (J)

Girona Biomedical Research Institute (IDIBGI), Hospital Universitari Dr Josep Trueta, Girona 17007, Spain. Electronic address: jpuigmd@gmail.com.

M Ingles (M)

Department of Physiology, University of Valencia, Valencia 46004, Spain. Electronic address: marta.ingles@uv.es.

C Borras (C)

Department of Physiology, University of Valencia, Valencia 46004, Spain. Electronic address: consuelo.borras@uv.es.

J Viña (J)

Department of Physiology, University of Valencia, Valencia 46004, Spain. Electronic address: jose.vina@uv.es.

P J Meikle (PJ)

Baker Heart and Diabetes Institute, Melbourne, VIC 3004, Australia. Electronic address: Peter.Meikle@baker.edu.au.

R Pamplona (R)

Department of Experimental Medicine, University of Lleida-Institute for Research in Biomedicine of Lleida (UdL-IRBLleida), Lleida 25198, Spain. Electronic address: reinald.pamplona@mex.udl.cat.

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