The protective effect of epigallocatechin 3-gallate on mouse pancreatic islets via the Nrf2 pathway.


Journal

Surgery today
ISSN: 1436-2813
Titre abrégé: Surg Today
Pays: Japan
ID NLM: 9204360

Informations de publication

Date de publication:
Jun 2019
Historique:
received: 26 09 2018
accepted: 20 12 2018
pubmed: 8 2 2019
medline: 14 6 2019
entrez: 8 2 2019
Statut: ppublish

Résumé

Epigallocatechin 3-gallate (EGCG), a green tea polyphenol, has been shown to have anti-oxidant and anti-inflammatory effects in vitro and in vivo. The aim of this study was to investigate the effects and mechanism of EGCG on isolated pancreatic islets as pre-conditioning for pancreatic islet transplantation. The pancreatic islets were divided into two groups: an islet culture medium group (control) and an islet culture medium with EGCG (100 µM) group. We investigated the islet viability, Nrf2 expression, reactive oxygen species (ROS) production, and heme oxygenase-1 (HO-1) mRNA. Five hundred islet equivalents after 12 h of culture for the EGCG 100 µM and control group were transplanted under the kidney capsule of streptozotocin-induced diabetic ICR mice. The cell viability and insulin secretion ability in the EGCG group were preserved, and the nuclear translocation of Nrf2 was increased in the EGCG group (p < 0.01). While the HO-1 mRNA levels were also higher in the EGCG group than in the control group (p < 0.05), the ROS production was lower (p < 0.01). An in vivo functional assessment showed that the blood glucose level had decreased in the EGCG group after transplantation (p < 0.01). EGCG protects the viability and function of islets by suppressing ROS production via the Nrf2 pathway.

Identifiants

pubmed: 30730004
doi: 10.1007/s00595-019-1761-0
pii: 10.1007/s00595-019-1761-0
doi:

Substances chimiques

Blood Glucose 0
NF-E2-Related Factor 2 0
Nfe2l2 protein, mouse 0
RNA, Messenger 0
Reactive Oxygen Species 0
Catechin 8R1V1STN48
epigallocatechin gallate BQM438CTEL
Heme Oxygenase-1 EC 1.14.14.18

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

536-545

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Auteurs

Yuma Wada (Y)

Department of Surgery, Graduate School of Biomedical Sciences, Tokushima University, 3-18-15 Kuramoto-cho, 770-8503, Tokushima, Japan.

Atsushi Takata (A)

Department of Surgery, Graduate School of Biomedical Sciences, Tokushima University, 3-18-15 Kuramoto-cho, 770-8503, Tokushima, Japan.

Tetsuya Ikemoto (T)

Department of Surgery, Graduate School of Biomedical Sciences, Tokushima University, 3-18-15 Kuramoto-cho, 770-8503, Tokushima, Japan. ikemoto.tetsuya@tokushima-u.ac.jp.

Yuji Morine (Y)

Department of Surgery, Graduate School of Biomedical Sciences, Tokushima University, 3-18-15 Kuramoto-cho, 770-8503, Tokushima, Japan.

Satoru Imura (S)

Department of Surgery, Graduate School of Biomedical Sciences, Tokushima University, 3-18-15 Kuramoto-cho, 770-8503, Tokushima, Japan.

Shuichi Iwahashi (S)

Department of Surgery, Graduate School of Biomedical Sciences, Tokushima University, 3-18-15 Kuramoto-cho, 770-8503, Tokushima, Japan.

Yu Saito (Y)

Department of Surgery, Graduate School of Biomedical Sciences, Tokushima University, 3-18-15 Kuramoto-cho, 770-8503, Tokushima, Japan.

Mitsuo Shimada (M)

Department of Surgery, Graduate School of Biomedical Sciences, Tokushima University, 3-18-15 Kuramoto-cho, 770-8503, Tokushima, Japan.

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Classifications MeSH