Statins and antiplatelet agents are associated with changes in the circulatory markers of endothelial dysfunction in chronic kidney disease.

El uso de estatinas y antiagregantes se asocia con cambios en los marcadores de disfunción endotelial en la enfermedad renal crónica.

Journal

Nefrologia
ISSN: 2013-2514
Titre abrégé: Nefrologia (Engl Ed)
Pays: Spain
ID NLM: 101778581

Informations de publication

Date de publication:
Historique:
received: 01 02 2018
revised: 21 07 2018
accepted: 09 11 2018
pubmed: 9 2 2019
medline: 8 5 2020
entrez: 9 2 2019
Statut: ppublish

Résumé

Patients with chronic kidney disease (CKD) have higher risk of developing cardiovascular disease. In CKD patients the mechanisms involved in, endothelial damage and the role of different drugs used on these patients are not completely understood. The aim of this work is to analyze the effect of statins and platelet antiaggregant (PA) on endothelial microvesicles (EMVs) and other markers of endothelial dysfunction. Cross-sectional study of 41 patients with CKD 3b-4 and 8 healthy volunteers. Circulating levels of EMVs, vascular endothelial growth factor (VEGF), and advance oxidized protein products (AOPPS) were quantified and the correlation with different comorbidity variables and therapeutic strategies were evaluated. EMVs are increased in CKD patients as compared with controls (171.1 vs. 68.3/μl, P<.001). It was observed a negative correlation between age and EMVs. Statins and PA were associated with a reduction in EMVs and VEGF levels, independently of the serum total cholesterol levels (TC). The levels of AOPPS and VEGF were not different in CKD vs. controls. CKD is associated with a change in EMVs, VEGF and AOPP levels. The treatment with statins and PA normalizes these values to almost the observed in controls and this effect is independently of the prevailing TC level. These findings explain the existence of the pleiotropic effects of statins and PA which deserve further studies.

Sections du résumé

BACKGROUNDS AND PURPOSES
Patients with chronic kidney disease (CKD) have higher risk of developing cardiovascular disease. In CKD patients the mechanisms involved in, endothelial damage and the role of different drugs used on these patients are not completely understood. The aim of this work is to analyze the effect of statins and platelet antiaggregant (PA) on endothelial microvesicles (EMVs) and other markers of endothelial dysfunction.
EXPERIMENTAL APPROACH
Cross-sectional study of 41 patients with CKD 3b-4 and 8 healthy volunteers. Circulating levels of EMVs, vascular endothelial growth factor (VEGF), and advance oxidized protein products (AOPPS) were quantified and the correlation with different comorbidity variables and therapeutic strategies were evaluated.
RESULTS
EMVs are increased in CKD patients as compared with controls (171.1 vs. 68.3/μl, P<.001). It was observed a negative correlation between age and EMVs. Statins and PA were associated with a reduction in EMVs and VEGF levels, independently of the serum total cholesterol levels (TC). The levels of AOPPS and VEGF were not different in CKD vs. controls.
CONCLUSION
CKD is associated with a change in EMVs, VEGF and AOPP levels. The treatment with statins and PA normalizes these values to almost the observed in controls and this effect is independently of the prevailing TC level. These findings explain the existence of the pleiotropic effects of statins and PA which deserve further studies.

Identifiants

pubmed: 30732927
pii: S0211-6995(18)30219-4
doi: 10.1016/j.nefro.2018.11.001
pii:
doi:

Substances chimiques

Advanced Oxidation Protein Products 0
Biomarkers 0
Hydroxymethylglutaryl-CoA Reductase Inhibitors 0
Platelet Aggregation Inhibitors 0
Vascular Endothelial Growth Factor A 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng spa

Sous-ensembles de citation

IM

Pagination

287-293

Informations de copyright

Copyright © 2019 Sociedad Española de Nefrología. Published by Elsevier España, S.L.U. All rights reserved.

Auteurs

Estefanya García-Menéndez (E)

Servicio de Nefrología, Hospital Universitario Puerta de Hierro Majadahonda, Majadahonda, Madrid, España. Electronic address: estefanyagarciam@gmail.com.

María Marques Vidas (M)

Servicio de Nefrología, Hospital Universitario Puerta de Hierro Majadahonda, Majadahonda, Madrid, España; REDInREN ISCiii 016/0091009 RETYC, Madrid, España.

Matilde Alique (M)

Facultad de Medicina y Ciencias de la Salud, Universidad de Alcalá, Departmento Biología de Sistemas, Alcalá de Henares, Madrid, España.

Julia Carracedo (J)

Hospital Universitario Reina Sofía, Instituto Maimónides de Investigación Biomédica de Córdoba (IMIBIC), Córdoba, España.

Patricia de Sequera (P)

Servicio de Nefrología, Hospital Universitario Infanta Leonor, Madrid, España.

Elena Corchete (E)

Servicio de Nefrología, Hospital Universitario Infanta Leonor, Madrid, España.

Rafael Pérez García (R)

Servicio de Nefrología, Hospital Universitario Infanta Leonor, Madrid, España.

Rafael Ramírez Chamond (R)

Facultad de Medicina y Ciencias de la Salud, Universidad de Alcalá, Departmento Biología de Sistemas, Alcalá de Henares, Madrid, España.

José M Portolés Pérez (JM)

Servicio de Nefrología, Hospital Universitario Puerta de Hierro Majadahonda, Majadahonda, Madrid, España; REDInREN ISCiii 016/0091009 RETYC, Madrid, España.

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Classifications MeSH