Toward in vivo determination of peripheral nervous system immune activity in amyotrophic lateral sclerosis.


Journal

Muscle & nerve
ISSN: 1097-4598
Titre abrégé: Muscle Nerve
Pays: United States
ID NLM: 7803146

Informations de publication

Date de publication:
05 2019
Historique:
accepted: 03 02 2019
pubmed: 9 2 2019
medline: 23 10 2019
entrez: 9 2 2019
Statut: ppublish

Résumé

We sought to identify patients with amyotrophic lateral sclerosis (ALS) who displayed suspected peripheral nervous system (PNS) inflammation to compare them to those with suspected PNS degeneration. We measured sonographic median and ulnar nerve cross-sectional area (CSA) and cerebrospinal fluid albumin/serum albumin ratio (Q Fifty-seven percent of patients had suspected PNS degeneration, 21% had suspected PNS inflammation, and 21% displayed suspected "normal PNS state." Suspected PNS degeneration was related to classic ALS, shorter disease duration, and a smaller hypoechoic nerve area. Suspected PNS inflammation was associated with men, longer disease duration, and a larger hypoechoic nerve area and was the dominant finding in superoxide dismutase 1 mutation carriers. Our simple approach might aid in the in vivo differentiation of supposed ALS subtypes, those with suspected PNS degeneration vs. inflammation, for stratification in clinical trials. Muscle Nerve 59:567-567, 2019.

Identifiants

pubmed: 30734322
doi: 10.1002/mus.26444
doi:

Substances chimiques

Albumins 0
Serum Albumin 0
Superoxide Dismutase-1 EC 1.15.1.1

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

567-576

Informations de copyright

© 2019 Wiley Periodicals, Inc.

Auteurs

Stefanie Schreiber (S)

Department of Neurology, Otto-von-Guericke University, Leipziger Straße 44, 39120 Magdeburg, Germany.
German Center for Neurodegenerative Diseases within the Helmholtz Association, Magdeburg, Germany.

Frank Schreiber (F)

Department of Neurology, Otto-von-Guericke University, Leipziger Straße 44, 39120 Magdeburg, Germany.
German Center for Neurodegenerative Diseases within the Helmholtz Association, Magdeburg, Germany.

Cornelia Garz (C)

Department of Neurology, Otto-von-Guericke University, Leipziger Straße 44, 39120 Magdeburg, Germany.
German Center for Neurodegenerative Diseases within the Helmholtz Association, Magdeburg, Germany.

Grazyna Debska-Vielhaber (G)

Department of Neurology, Otto-von-Guericke University, Leipziger Straße 44, 39120 Magdeburg, Germany.

Anne Assmann (A)

Department of Neurology, Otto-von-Guericke University, Leipziger Straße 44, 39120 Magdeburg, Germany.
German Center for Neurodegenerative Diseases within the Helmholtz Association, Magdeburg, Germany.

Valentina Perosa (V)

Department of Neurology, Otto-von-Guericke University, Leipziger Straße 44, 39120 Magdeburg, Germany.
German Center for Neurodegenerative Diseases within the Helmholtz Association, Magdeburg, Germany.

Susanne Petri (S)

Department of Neurology, Hannover Medical School, Hannover, Germany.

Reinhard Dengler (R)

Department of Neurology, Hannover Medical School, Hannover, Germany.

Peter Nestor (P)

Queensland Brain Institute, University of Queensland, Brisbane, Queensland, Australia.

Stefan Vielhaber (S)

Department of Neurology, Otto-von-Guericke University, Leipziger Straße 44, 39120 Magdeburg, Germany.
German Center for Neurodegenerative Diseases within the Helmholtz Association, Magdeburg, Germany.

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